Friday, October 28, 2011

CWD Herd Monitoring Program to be Enforced Jan. 2012 TEXAS

Texas Animal Health Commission (TAHC) Announcement October 27, 2011

Attention: Elk Producers

CWD Herd Monitoring Program to be Enforced Jan. 2012


AUSTIN - Elk producers wishing to sell or move elk must enroll in the Texas Animal Health Commission (TAHC) Chronic Wasting Disease (CWD) herd monitoring program or have elk tested as described below. During 2011, TAHC rules for elk movement have been held in abeyance to encourage producers to enroll in the program.

After January 1, 2012, however, elk will only be allowed to move after all surveillance requirements have been met. Surveillance requirements can be met one of two ways:

By enrolling in the CWD status program, testing all mortalities and achieving "status," or By having a valid "not detected" CWD test on file prior to movement. The number of valid CWD tests required are based on the number of elk being moved and whether they are captive or free ranging. The TAHC implemented new elk herd requirements on January 1, 2010, to ensure a stronger surveillance system for CWD in elk. "The program is intended to help protect the exotic and native wildlife and the cervid industry of Texas from the possible introduction of CWD, by developing an effective surveillance system", Dr. Dee Ellis, TAHC State Veterinarian, said." "Enrollment and ultimately achieving status will allow producers to move elk without requiring additional mortality testing," Dr. Ellis added. "I strongly encourage all elk producers to sign up today."

CWD is a transmissible spongiform encephalopathy known to affect elk, moose, white-tailed deer, black-tailed deer and mule deer. It is a fatal, degenerative brain wasting disease. The typical clinical signs of CWD are emaciation, behavioral changes and excessive salivation.

CWD is not known to affect people, and has not been detected in Texas to date. Since it was first detected in Colorado in 1967 however, it has subsequently been diagnosed in 17 other states and continues to threaten the cervid industry in other parts of the US.

The TAHC enforces interstate movement requirements for elk and other cervids entering Texas. Out of state animals must originate from a herd which has participated for at least five years in a state-approved CWD herd certification program, and with no clinical signs of CWD in the herd. In today's environment, the mobility and transportation of agricultural animals throughout the state and country has greatly increased the potential exposure to diseases.

"Adequate and timely surveillance testing is critical to detect a newly introduced or emerging disease as quickly as possible, so that it can be eliminated before potential spread to other animals", explained Dr. Terry Hensley, Assistant State Veterinarian.

CWD has not been detected in captive or free-ranging deer or elk in Texas, but elk producers must continue to be mindful of the disease, and take necessary precautions to ensure the safety of exotic livestock.

"Maintaining surveillance for CWD in Texas is critical for effective animal disease response," Dr. Hensley added.

By keeping appropriate records and sufficient sampling of animals as required by the program, a herd can achieve a recognized "herd status" for CWD. Under the TAHC's elk enrollment program, elk would be test- eligible at 16 months of age or older, and tests conducted in a herd would be valid for one year.

In order to be eligible for moving elk, participation in the program is mandatory. Elk owners can enroll their herds in the CWD monitoring program today by contacting their local TAHC regional office.

Region 1 (Amarillo, TX) Phone: 806-354-9335 Region Director: Dr. Brad Williams Supervising Inspector: Bob Young

Region 5 (Beeville, TX)

Phone: 361-358-3234

Regional Director: Dr. David Finch

Supervising Inspector: Howard Helmers

Region 2 (Hempstead, TX)

Phone: 979-921-9481

Regional Director: Dr. Mark Michalke

Supervising Inspector: Dwayne Easley Region 6 (Lampasas, TX)

Phone: 512-556-6277

Region Director: Dr. Pete Fincher

Supervising Inspector: David Martin

Region 3 (Fort Worth, TX)

Phone: 817-244-2597 Region Director: Dr. Max Dow Supervising Inspector: Bobby Crozier

Region 7 (Rockdale, TX)

Phone: 512-446-2507

Regional Director: Dr. Tommy Barton

Supervising Inspector: Russell Iselt

Region 4 (Mt. Pleasant, TX)

Phone: 903-572-1966

Regional Director: Dr. Greg Hawkins

Supervising Inspector: Chip Nicholson

For more information about CWD, visit

Founded in 1893, the Texas Animal Health Commission works to protect the health of all Texas livestock, including: cattle, swine, poultry, sheep, goats, equine animals, and exotic livestock.


"CWD is not known to affect people, and has not been detected in Texas to date." ???

key word is 'to date'.

please see ;

EFSA Journal 2011 The European Response to BSE: A Success Story

This is an interesting editorial about the Mad Cow Disease debacle, and it's ramifications that will continue to play out for decades to come ;

Monday, October 10, 2011

EFSA Journal 2011 The European Response to BSE: A Success Story


EFSA and the European Centre for Disease Prevention and Control (ECDC) recently delivered a scientific opinion on any possible epidemiological or molecular association between TSEs in animals and humans (EFSA Panel on Biological Hazards (BIOHAZ) and ECDC, 2011). This opinion confirmed Classical BSE prions as the only TSE agents demonstrated to be zoonotic so far but the possibility that a small proportion of human cases so far classified as "sporadic" CJD are of zoonotic origin could not be excluded. Moreover, transmission experiments to non-human primates suggest that some TSE agents in addition to Classical BSE prions in cattle (namely L-type Atypical BSE, Classical BSE in sheep, transmissible mink encephalopathy (TME) and ***chronic wasting disease (CWD) agents) might have zoonotic potential.


see follow-up here about North America BSE Mad Cow TSE prion risk factors, and the ever emerging strains of Transmissible Spongiform Encephalopathy in many species here in the USA, including humans ;

Monday, June 27, 2011

Zoonotic Potential of CWD: Experimental Transmissions to Non-Human Primates

Thursday, May 26, 2011

Travel History, Hunting, and Venison Consumption Related to Prion Disease Exposure, 2006-2007 FoodNet Population Survey

Journal of the American Dietetic Association Volume 111, Issue 6 , Pages 858-863, June 2011.


Wednesday, September 08, 2010


Wednesday, January 5, 2011



David W. Colby1,* and Stanley B. Prusiner1,2

Thursday, April 03, 2008

A prion disease of cervids: Chronic wasting disease

2008 1: Vet Res. 2008 Apr 3;39(4):41

A prion disease of cervids: Chronic wasting disease

Sigurdson CJ.


*** twenty-seven CJD patients who regularly consumed venison were reported to the Surveillance Center***,


full text ;


October 1994

Mr R.N. Elmhirst Chairman British Deer Farmers Association Holly Lodge Spencers Lane BerksWell Coventry CV7 7BZ

Dear Mr Elmhirst,


Thank you for your recent letter concerning the publication of the third annual report from the CJD Surveillance Unit. I am sorry that you are dissatisfied with the way in which this report was published.

The Surveillance Unit is a completely independant outside body and the Department of Health is committed to publishing their reports as soon as they become available. In the circumstances it is not the practice to circulate the report for comment since the findings of the report would not be amended. In future we can ensure that the British Deer Farmers Association receives a copy of the report in advance of publication.

The Chief Medical Officer has undertaken to keep the public fully informed of the results of any research in respect of CJD. This report was entirely the work of the unit and was produced completely independantly of the the Department.

The statistical results reqarding the consumption of venison was put into perspective in the body of the report and was not mentioned at all in the press release. Media attention regarding this report was low key but gave a realistic presentation of the statistical findings of the Unit. This approach to publication was successful in that consumption of venison was highlighted only once by the media ie. in the News at one television proqramme.

I believe that a further statement about the report, or indeed statistical links between CJD and consumption of venison, would increase, and quite possibly give damaging credence, to the whole issue. From the low key media reports of which I am aware it seems unlikely that venison consumption will suffer adversely, if at all.


Thursday, May 26, 2011

Travel History, Hunting, and Venison Consumption Related to Prion Disease Exposure, 2006-2007 FoodNet Population Survey

Journal of the American Dietetic Association Volume 111, Issue 6 , Pages 858-863, June 2011.

NOR IS THE FDA recalling this CWD positive elk meat for the well being of the dead elk ;

Wednesday, March 18, 2009

Noah's Ark Holding, LLC, Dawson, MN RECALL Elk products contain meat derived from an elk confirmed to have CWD NV, CA, TX, CO, NY, UT, FL, OK RECALLS AND FIELD CORRECTIONS: FOODS CLASS II

Sunday, July 27, 2008

DOCKET-- 03D-0186 -- FDA Issues Draft Guidance on Use of Material From Deer and Elk in Animal Feed; Availability

-------- Original Message --------

Subject: DOCKET-- 03D-0186 -- FDA Issues Draft Guidance on Use of Material From Deer and Elk in Animal Feed; Availability

Date: Fri, 16 May 2003 11:47:37 -0500

From: "Terry S. Singeltary Sr."


Greetings FDA,

i would kindly like to comment on;

Docket 03D-0186

FDA Issues Draft Guidance on Use of Material From Deer and Elk in Animal Feed; Availability

Several factors on this apparent voluntary proposal disturbs me greatly, please allow me to point them out;

1. MY first point is the failure of the partial ruminant-to-ruminant feed ban of 8/4/97. this partial and voluntary feed ban of some ruminant materials being fed back to cattle is terribly flawed. without the _total_ and _mandatory_ ban of all ruminant materials being fed back to ruminants including cattle, sheep, goat, deer, elk and mink, chickens, fish (all farmed animals for human/animal consumption), this half ass measure will fail terribly, as in the past decades...

2. WHAT about sub-clinical TSE in deer and elk? with the recent findings of deer fawns being infected with CWD, how many could possibly be sub-clinically infected. until we have a rapid TSE test to assure us that all deer/elk are free of disease (clinical and sub-clinical), we must ban not only documented CWD infected deer/elk, but healthy ones as well. it this is not done, they system will fail...

3. WE must ban not only CNS (SRMs specified risk materials), but ALL tissues. recent new and old findings support infectivity in the rump or ass muscle. wether it be low or high, accumulation will play a crucial role in TSEs.

4. THERE are and have been for some time many TSEs in the USA. TME in mink, Scrapie in Sheep and Goats, and unidentified TSE in USA cattle. all this has been proven, but the TSE in USA cattle has been totally ignored for decades. i will document this data below in my references.

5. UNTIL we ban all ruminant by-products from being fed back to ALL ruminants, until we rapid TSE test (not only deer/elk) but cattle in sufficient numbers to find (1 million rapid TSE test in USA cattle annually for 5 years), any partial measures such as the ones proposed while ignoring sub-clinical TSEs and not rapid TSE testing cattle, not closing down feed mills that continue to violate the FDA's BSE feed regulation (21 CFR 589.2000) and not making freely available those violations, will only continue to spread these TSE mad cow agents in the USA. I am curious what we will call a phenotype in a species that is mixed with who knows how many strains of scrapie, who knows what strain or how many strains of TSE in USA cattle, and the CWD in deer and elk (no telling how many strains there), but all of this has been rendered for animal feeds in the USA for decades. it will get interesting once someone starts looking in all species, including humans here in the USA, but this has yet to happen...

6. IT is paramount that CJD be made reportable in every state (especially ''sporadic'' cjd), and that a CJD Questionnaire must be issued to every family of a victim of TSE. only checking death certificates will not be sufficient. this has been proven as well (see below HISTORY OF CJD -- CJD QUESTIONNAIRE)

7. WE must learn from our past mistakes, not continue to make the same mistakes...


snip...see full text ;

Wednesday, October 12, 2011

White-tailed deer are susceptible to the agent of sheep scrapie by intracerebral inoculation

Wednesday, September 21, 2011

Evidence for distinct CWD strains in experimental CWD in ferrets

Wednesday, July 06, 2011

Swine Are Susceptible to Chronic Wasting Disease by Intracerebral Inoculation

Tuesday, October 25, 2011

CWD Update 102 October 20, 2011

Thursday, June 09, 2011

Detection of CWD prions in salivary, urinary, and intestinal tissues of deer: potential mechanisms of prion shedding and transmission

Wednesday, June 01, 2011

Management of CWD in Canada: Past Practices, Current Conditions, Current Science, Future Risks and Options


Sunday, October 04, 2009


Thursday, February 17, 2011

Environmental Sources of Scrapie Prions

Saturday, May 16, 2009

Chronic Wasting Disease Herd Certification Program Document ID APHIS-2006-0118-0096

Thursday, August 4, 2011

Terry Singeltary Sr. on the Creutzfeldt-Jakob Disease Public Health Crisis, Date aired: 27 Jun 2011 (see video)




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