Thursday, March 29, 2012

Missouri officials seek states' advice on chronic wasting disease in deer

From: Terry S. Singeltary Sr.

Sent: Thursday, March 29, 2012 6:26 PM


Cc: ; ; ; ; ; ; ; ; ;

Subject: re-Missouri officials seek states' advice on chronic wasting disease in deer

re-Missouri officials seek states' advice on chronic wasting disease in deer

Thursday, March 29, 2012 | 5:20 p.m. CDT BY John McLaughlin Missouri conservation officials are hoping for a different outcome than Wisconsin had after an outbreak of chronic wasting disease in the wild population of white-tailed deer.

The outbreak in Wisconsin, which began with three deer in 2002, continues to spread despite aggressive strategies to keep it in check, said Travis Anderson, wildlife biologist for the Missouri Natural Resources Department.


Greetings Missourian and Missouri hunters et al,

A kind and warm greetings from Texas.

My name is Terry S. Singeltary Sr., I live in Bacliff, Texas, and I have been following the mad cow/deer/elk/sheep/goat/cat/dog/mink saga since my mother died from the Heidenhain Variant of Creutzfeldt Jakob Disease. I urge you to take heed to what I am about to present as evidence, that GAME FARMERS/RANCHERS, STRAW BRED BUCK BREEDERS, are a big problem and cause of CWD. NOT all of it is their fault, bbbut I am going to show you where some of these game farms have been nothing more than a petri dish for CWD. I hope that you take the time to read. I tend to overwhelm folks with TSE prion science, but please take the time to study this, we have all been exposed, one way or the other via friendly fire, I assure you. but that’s another story. ...

layperson, meat eater, fisherman, hunter, retired medically

kindest regards,


From: Terry S. Singeltary Sr.

Sent: Thursday, March 29, 2012 10:25 AM




Deer czar: herd estimates are inaccurate

Thursday, March 29, 2012 8:15 a.m. CDT


UNDATED (WSAU) Wisconsin’s “deer czar” says the D-N-R spends too much time miscalculating the size of the deer herd, and not enough time dealing with habitat and people. James Kroll issued his preliminary report yesterday, after Governor Scott Walker hired him last fall to review the D-N-R’s deer management efforts. Kroll, a deer researcher from Texas, said landowners and hunters have a quote, “intense dissatisfaction” with the agency – and a lot of it stems from what he called a failed effort to control chronic wasting disease in the last decade.

Kroll said the program regarded deer in the disease zones as pests rather than a major big game animal. He also said the D-N-R’s deer herd estimates are indefensible, and they’re based on procedures that are decades old. And he said officials have not done adequate studies on wolves, and how they fit into the state’s eco-system.

D-N-R big game ecologist Kevin Wallenfang said his agency has known about some of the issues in the report for a long time – and they’re already addressing them. Two years ago, officials started a $2-million effort to improve the deer population estimates, and determine the impact by predators. It has also expanded a computer list of hunter observations, and improved communications with the public. The D-N-R also ended the Earn-a-Buck program that required hunters to kill a doe before taking a buck.

Town hall meetings are being set throughout Wisconsin next month to discuss Kroll’s preliminary report. A final report is expected in June.

Wisconsin hired this guy from Texas to evaluate their CWD problem. I don’t know how this guy from Texas can help them, CWD has been waltzing across Texas for 10 years. seems Dr. Kroll should go back home and take care of CWD in Texas coming in from New Mexico. ...just my opinion of course. ...tss



MARCH, 2012

Drs. James C. Kroll (Trustee), David C. Guynn, Jr. and Gary L Alt

Presented to,

Wisconsin Department of Administration

Madison, Wisconsin

Original press release issued by Wisconsin DNR on March 28th, 2012

A Hillsboro man kicked off the Twelve Days of Christmas with two days of poaching.

From the night before Christmas to the holiday night, the 20-year-old roamed the roads shining and shooting bucks and does as they stood blindly in the light. “This guy wasn’t hunting. He was poaching,” Conservation Warden Mike Nice of Richland Center said of the suspect who is alleged to have illegally taken approximately 100 deer since 2011.

“In my 22 years as a conservation warden, I have never seen another individual come close to that total. The suspect didn’t care,” Warden Nice said of the multi-county case he investigated with Deputy Warden Mike Williams and Warden Cody Adams of Prairie du Chien.

The suspect, along with two other young adult males and one juvenile male, face criminal charges stemming from the eight poaching cases — four in Richland County and four in Vernon County — that were documented by the investigating wardens. Twenty one years revocation of hunting rights is being sought along with restitution and jail time for the Hillsboro man. Confiscation of three rifles used to shoot the deer is also being requested.

Criminal charges were filed in the counties in March. Court proceedings are expected this spring. Nice said the wardens were helped in their investigation by Richland County District Attorney Jennifer Harper, who, by sheer coincidence, herself received information from a tipster. “District Attorney Harper relayed the tip, but, just as importantly, she has done an outstanding job in prosecuting criminal DNR violations in her capacity as the district attorney,” Nice said.

Assistant District Attorney Stacy Smith will be prosecuting the charges in Vernon County. A total of four individuals are being charged in the case.

Thursday, February 09, 2012


Friday, February 03, 2012

Wisconsin Farm-Raised Deer Farms and CWD there from 2012 report Singeltary et al

Saturday, February 04, 2012

Wisconsin 16 age limit on testing dead deer Game Farm CWD Testing Protocol Needs To Be Revised

Tuesday, December 20, 2011


The CWD infection rate was nearly 80%, the highest ever in a North American captive herd.

Despite the five year premise plan and site decontamination, The WI DNR has concerns over the bioavailability of infectious prions at this site to wild white-tail deer should these fences be removed. Current research indicates that prions can persist in soil for a minimum of 3 years.

However, Georgsson et al. (2006) concluded that prions that produced scrapie disease in sheep remained bioavailable and infectious for at least 16 years in natural Icelandic environments, most likely in contaminated soil.

Additionally, the authors reported that from 1978-2004, scrapie recurred on 33 sheep farms, of which 9 recurrences occurred 14-21 years after initial culling and subsequent restocking efforts; these findings further emphasize the effect of environmental contamination on sustaining TSE infectivity and that long-term persistence of prions in soils may be substantially greater than previously thought.


SUMMARY: The Department has obtained on agreement to purchase 80 acres of land from Patricia Casey for $465,000 for the Statewide Wildlife Habitat program, In Portage County, The item is being submitted because the price exceeds the appraised value of $371,000 and because the Department will prohibit Nature-Based Outdoor Activities on the land.

The properly is located in south central Portage County about 12 miles southeast of Plover in the Town of Almond. The property, which includes a single family residence, a metal building, and a storage shed, was operated us a deer farm until 2006 at which time it was closed down because of an outbreak of chronic wasting disease (CWD). All the deer in the operation were destroyed and the operation has stood vacant for the last 5 years per U .S. Dcpnrtmeut of Agriculture requirements. During this time and until May 24, 2011, the fences around this facility must be maintained and the premise cannot be used as a deer farm, though other animals such ns cattle and horses would be permitted. After May 24, all such restrictions will expire. This site, known as the Hall Farm, had the highest prevalence of CWO positive deer recorded at any deer farm in North America.

Based on available science, the Department believes that there is an unacceptable potential risk of exposure to CWD causing prions to wild cervids in this area should the premise fencing be removed. To minimize this risk, the Department believes that the fences should remain intact and in place until science can demonstrate that there is no longer any potential risk. After extensive consideration of several options, the Department maintains that the purchase and subsequent management of the properly and fences is the only realistic option.

The Department proposes to prohibit all public use of the property in order to ensure confinement and control of contaminated soils and limit any potential spread of Chronic Wasting Disease from the property to surrounding lands and wild deer populations and to allow for research of prions and prion related diseases such as Chronic Wasting Disease. The property is currently surrounded by a deer fence and removal of that fence to allow public use, or public use of any form inside the fenced area would be incompatible with the primary purpose of acquiring the properly. The Department has determined that it is necessary to prohibit all public access on the site to accommodate the Department's primary purpose for the acquisition and its Intended use of the property for research and wildlife management.

Acquisition of this properly will minimize any potential risk to local cervids from the CWD causing prions that may exist within the fenced area. The Department will consider sale of the house at a later date if local zoning can be modified for a lot size that would not contain contaminated soil. State ownership will allow the Department to maintain the deer proof fence, thereby protecting wild deer from CWD infection from the contaminated soil on this former deer farm.

RECOMMENDATION: That the Board approve the purchase of 80 acres of land for $465,000 for the Statewide Wildlife Habitat Program in Portage County and approve the restrictions on public use of the site.

snip...see full text and much more here ;


Thursday, February 09, 2012

Colorado Farm-Raised Deer Farms and CWD there from 2012 report Singeltary et al

Monday, February 13, 2012

Stop White-tailed Deer Farming from Destroying Tennessee’s Priceless Wild Deer Herd oppose HB3164

Tuesday, February 14, 2012

Oppose Indiana House Bill 1265 game farming cervids

Wednesday, February 15, 2012

West Virginia Deer Farming Bill backed by deer farmers advances, why ? BE WARNED CWD


2001 - 2002


Subject: CWD testing in Texas

Date: Sun, 25 Aug 2002 19:45:14 –0500

From: Kenneth Waldrup



Dear Dr. Singletary,

In Fiscal Year 2001, seven deer from Texas were tested by the National Veterinary Services Laboratory (NVSL) for CWD (5 fallow deer and 2 white-tailed deer). In Fiscal Year 2002, seven elk from Texas were tested at NVSL (no deer). During these two years, an additional six elk and one white-tailed deer were tested at the Texas Veterinary Medical Diagnostic Laboratory (TVMDL). In Fiscal Year 2002, four white-tailed deer (free-ranging clinical suspects) and at least eight other white-tailed deer have been tested at TVMDL. One elk has been tested at NVSL. All of these animals have been found negative for CWD. Dr. Jerry Cooke of the Texas Parks and Wildlife Department also has records of 601 clinically ill white-tailed deer which were necropsied at Texas A&M during the late 1960's and early 1970's, and no spongiform encepalopathies were noted.

Thank you for your consideration.

Ken Waldrup, DVM, PhD Texas Animal Health Commission



Captive Cervids

There have been no reported CWD infections of captive elk or deer in Texas. There is currently no mandatory surveillance program for susceptible cervids kept on game farms, although, there has been voluntary surveillance since 1999, which requires owners of participating herds to maintain an annual herd inventory and submit samples for all mortalities of animals over 16 months of age.


SO, i thought i would just see where these Ecoregions were, and just how the CWD testing was distributed. YOU would think that with the cluster of CWD bordering TEXAS at the WPMR in NM, you would have thought this would be where the major CWD testing samples were to have been taken? wrong! let's have a look at the sample testing. here is map of CWD in NM WPMR bordering TEXAS;


NEXT, let's have a look at the overall distribution of CWD in Free-Ranging Cervids and see where the CWD cluster in NM WSMR borders TEXAS;

Current Distribution of Chronic Wasting Disease in Free-Ranging Cervids

NOW, the MAP of the Exoregion where the samples were taken to test for CWD;


Ecoregions of TEXAS

IF you look at the area around the NM WSMR where the CWD cluster was and where it borders TEXAS, that ecoregion is called Trans Pecos region. Seems if my Geography and my Ciphering is correct ;-) that region only tested 55% of it's goal. THE most important area on the MAP and they only test some 96 samples, this in an area that has found some 7 positive animals? NOW if we look at the only other border where these deer from NM could cross the border into TEXAS, this area is called the High Plains ecoregion, and again, we find that the sampling for CWD was pathetic. HERE we find that only 9% of it's goal of CWD sampling was met, only 16 samples were tested from some 175 that were suppose to be sampled.

AS i said before;

> SADLY, they have not tested enough from the total population to

> know if CWD is in Texas or not.

BUT now, I will go one step further and state categorically that they are not trying to find it. just the opposite it seems, they are waiting for CWD to find them, as with BSE/TSE in cattle, and it will eventually...

snip...see full text ;

see more on warning from New Mexico about CWD waltzing across Texas in the next two links ;

Monday, March 26, 2012

Texas Prepares for Chronic Wasting Disease CWD Possibility in Far West Texas

Monday, March 26, 2012


Sunday, January 22, 2012

Chronic Wasting Disease CWD cervids interspecies transmission

Thursday, January 26, 2012

The Risk of Prion Zoonoses

Science 27 January 2012: Vol. 335 no. 6067 pp. 411-413 DOI: 10.1126/science.1218167

Thursday, January 26, 2012

Facilitated Cross-Species Transmission of Prions in Extraneural Tissue

Science 27 January 2012: Vol. 335 no. 6067 pp. 472-475 DOI: 10.1126/science.1215659

Tuesday, February 14, 2012

White House budget proposes cuts to ag programs including TSE PRION disease aka mad cow type disease





Captive CWD discovered 1967

Free ranging CWD discovered 1981



*** Chronic Wasting Disease CWD CDC REPORT MARCH 2012 ***

Saturday, February 18, 2012

Occurrence, Transmission, and Zoonotic Potential of Chronic Wasting Disease

CDC Volume 18, Number 3—March 2012


Long-term effects of CWD on cervid populations and ecosystems remain unclear as the disease continues to spread and prevalence increases. In captive herds, CWD might persist at high levels and lead to complete herd destruction in the absence of human culling. Epidemiologic modeling suggests the disease could have severe effects on free-ranging deer populations, depending on hunting policies and environmental persistence (8,9). CWD has been associated with large decreases in free-ranging mule deer populations in an area of high CWD prevalence (Boulder, Colorado, USA) (5).


Reasons for Caution There are several reasons for caution with respect to zoonotic and interspecies CWD transmission. First, there is strong evidence that distinct CWD strains exist (36). Prion strains are distinguished by varied incubation periods, clinical symptoms, PrPSc conformations, and CNS PrPSc depositions (3,32). Strains have been identified in other natural prion diseases, including scrapie, BSE, and CJD (3). Intraspecies and interspecies transmission of prions from CWD-positive deer and elk isolates resulted in identification of >2 strains of CWD in rodent models (36), indicating that CWD strains likely exist in cervids. However, nothing is currently known about natural distribution and prevalence of CWD strains. Currently, host range and pathogenicity vary with prion strain (28,37). Therefore, zoonotic potential of CWD may also vary with CWD strain. In addition, diversity in host (cervid) and target (e.g., human) genotypes further complicates definitive findings of zoonotic and interspecies transmission potentials of CWD. Intraspecies and interspecies passage of the CWD agent may also increase the risk for zoonotic CWD transmission. The CWD prion agent is undergoing serial passage naturally as the disease continues to emerge. In vitro and in vivo intraspecies transmission of the CWD agent yields PrPSc with an increased capacity to convert human PrPc to PrPSc (30). Interspecies prion transmission can alter CWD host range (38) and yield multiple novel prion strains (3,28). The potential for interspecies CWD transmission (by cohabitating mammals) will only increase as the disease spreads and CWD prions continue to be shed into the environment. This environmental passage itself may alter CWD prions or exert selective pressures on CWD strain mixtures by interactions with soil, which are known to vary with prion strain (25), or exposure to environmental or gut degradation. Given that prion disease in humans can be difficult to diagnose and the asymptomatic incubation period can last decades, continued research, epidemiologic surveillance, and caution in handling risky material remain prudent as CWD continues to spread and the opportunity for interspecies transmission increases. Otherwise, similar to what occurred in the United Kingdom after detection of variant CJD and its subsequent link to BSE, years of prevention could be lost if zoonotic transmission of CWD is subsequently identified, SNIP...SEE FULL TEXT ;

*** Chronic Wasting Disease CWD CDC REPORT MARCH 2012 ***

Saturday, February 18, 2012

Occurrence, Transmission, and Zoonotic Potential of Chronic Wasting Disease

CDC Volume 18, Number 3—March 2012

see much more here ;


October 1994

Mr R.N. Elmhirst Chairman British Deer Farmers Association Holly Lodge Spencers Lane BerksWell Coventry CV7 7BZ

Dear Mr Elmhirst,


Thank you for your recent letter concerning the publication of the third annual report from the CJD Surveillance Unit. I am sorry that you are dissatisfied with the way in which this report was published.

The Surveillance Unit is a completely independant outside body and the Department of Health is committed to publishing their reports as soon as they become available. In the circumstances it is not the practice to circulate the report for comment since the findings of the report would not be amended. In future we can ensure that the British Deer Farmers Association receives a copy of the report in advance of publication.

The Chief Medical Officer has undertaken to keep the public fully informed of the results of any research in respect of CJD. This report was entirely the work of the unit and was produced completely independantly of the the Department.

The statistical results reqarding the consumption of venison was put into perspective in the body of the report and was not mentioned at all in the press release. Media attention regarding this report was low key but gave a realistic presentation of the statistical findings of the Unit. This approach to publication was successful in that consumption of venison was highlighted only once by the media ie. in the News at one television proqramme.

I believe that a further statement about the report, or indeed statistical links between CJD and consumption of venison, would increase, and quite possibly give damaging credence, to the whole issue. From the low key media reports of which I am aware it seems unlikely that venison consumption will suffer adversely, if at all.



Consumption of venison and veal was much less widespread among both cases and controls. For both of these meats, there was evidence of a trend with increasing frequency of consumption being associated with increasing risk of CJD. These associations were largely unchanged when attention was restricted to pairs with data obtained from relatives. ...


Thursday, May 26, 2011

Travel History, Hunting, and Venison Consumption Related to Prion Disease Exposure, 2006-2007 FoodNet Population Survey

Journal of the American Dietetic Association Volume 111, Issue 6 , Pages 858-863, June 2011.

NOR IS THE FDA recalling this CWD positive elk meat for the well being of the dead elk ;

Wednesday, March 18, 2009

Noah's Ark Holding, LLC, Dawson, MN RECALL Elk products contain meat derived from an elk confirmed to have CWD NV, CA, TX, CO, NY, UT, FL, OK RECALLS AND FIELD CORRECTIONS: FOODS CLASS II

now, let’s see what the authors said about this casual link, personal communications years ago. see where it is stated NO STRONG evidence. so, does this mean there IS casual evidence ????

“Our conclusion stating that we found no strong evidence of CWD transmission to humans”

From: TSS (


Date: September 30, 2002 at 7:06 am PST

From: "Belay, Ermias"


Cc: "Race, Richard (NIH)" ; ; "Belay, Ermias"

Sent: Monday, September 30, 2002 9:22 AM


Dear Sir/Madam,

In the Archives of Neurology you quoted (the abstract of which was attached to your email), we did not say CWD in humans will present like variant CJD.

That assumption would be wrong. I encourage you to read the whole article and call me if you have questions or need more clarification (phone: 404-639-3091). Also, we do not claim that "no-one has ever been infected with prion disease from eating venison." Our conclusion stating that we found no strong evidence of CWD transmission to humans in the article you quoted or in any other forum is limited to the patients we investigated.

Ermias Belay, M.D. Centers for Disease Control and Prevention

-----Original Message-----


Sent: Sunday, September 29, 2002 10:15 AM

To:;; ebb8@CDC.GOV


Sunday, November 10, 2002 6:26 PM ......snip........end..............TSS

Thursday, April 03, 2008

A prion disease of cervids: Chronic wasting disease

2008 1: Vet Res. 2008 Apr 3;39(4):41

A prion disease of cervids: Chronic wasting disease

Sigurdson CJ.


*** twenty-seven CJD patients who regularly consumed venison were reported to the Surveillance Center***,


full text ;

Saturday, March 5, 2011


Sunday, February 12, 2012

National Prion Disease Pathology Surveillance Center Cases Examined1 (August 19, 2011) including Texas

blog has been updated with some information on the Wisconsin deer czar from Texas and old data on game farms and cwd. ...

We received news that James Kroll, a private-land deer manager from Texas

At the time, Kroll — nicknamed Dr. Deer — and his university research team were the only Texans using artificial insemination (A.I.) on deer. On Kroll’s advice, Grace adopted A.I. as a way to introduce superior genetics into his herd, thus becoming one of the first commercial deer breeders in North America known to use A.I.

It would become the key to success for Mustang Creek Ranch, which today markets stocker bucks, semen, open and bred does, and breeder bucks.

The event was the third annual Whitetail Workshop and Intro to A.I. School, led primarily by Dr. James Kroll, director of the Institute for White-tailed Deer Management and Research Center at Stephen F. Austin State University.

According to Bill Grace, owner of Mustang Creek Ranch and host of the workshop, the large attendance was indicative of a growing interest in deer breeding, an industry in which superior bucks often sell for six figures.


The Texas Deer Association, which was founded by Kroll, Grace and several other breeders in 1999, now has more than 2,000 members.

should have known. seems the good Dr. is/was an expert straw bred buck raiser/adviser, get those BIG DEER/RACKS $$$ GM deer I guess? I hope he does solve Wisconsin CWD problem, don’t get me wrong. bbbut, it is game farming, raising GM deer and elk, high/low fence operations that are a big part of the problem, in my opinion. like I said, I think Texas has a problem with CWD that he should be addressing if he is such an expert on CWD. ...tss


Game farm rules argued pro and con in Montana

December 12, 1999

By ERIN P. BILLINGS, Gazette State Bureau

Since 1992, the state has recorded 39 animal escapes from Montana game farm operations. During the same period there were 22 cases in which native wildlife got into a game farm. Here's a look at the number of animals escaping from game farms vs. the number of wildlife getting into the farms from the outside.

1992: Six escapes of game farm animals compared with seven animals entering the pen.

1993: Six escapes compared with one animal getting in from the outside.

1994: Nine escapes compared with one animal getting in from the outside.

1995: Six escapes compared with two animals getting in.

1996: Four escapes compared with two animals getting in.

1997: Two escapes compared with two getting in.

1998: Four escapes compared with six getting in.

1999: Two escapes compared with two getting in from the outside.<

seems to me it would be the game farm operation fault if i farmed deer escaped, OR if a wild deer or elk entered. but who would then own the wild deer? can't ever let him go, IF you are trying to keep CWD from the wild herds. by only testing dead deer at different ages per state, with sub-clinical CWD TSE, you never know when your farmed deer are infected. you could be selling them and or their products from state to state for years, before a game farm ever knows it's infected. by the time it's documented, it's too late. ...tss

How CWD spreads from Colorado

15 Jul 00 webmaster opinion

Fore background on CWD, see 1, 2, 3, 4

McCombie, in the article above, obtained access under Wisconsin's Freedom of Information Act to various CWD documents that since have been circulating on the internet, which the webmaster believes are authentic being plausible, consistent with previously disclosed information, and bearing signatures on official state stationery. These letters suggest that widespread traffic in elk coupled with minimal or non-existent state regulation and monitoring has the potential to spread CWD far and wide. Indeed, Colorado, Wyoming, Nebraska, Montana, Oklahoma, and Saskatchawan have announced cases; South Dakota,Missouri, Vermont, and numerous others have documented trace-back or trace-forward herds.

The first document, dated 17 Apr 98, on state letterhead from Nebraska State Veterinarian Dr. Larry Williams to Dr. Clarence Siroky, Wisconsin State Veterinarian, is a routine herd trace-forward letter. It states that on 9 April 98, an elk from a "domesticated cervine herd " at Heartland Elk Ranch in Cody, Nebraska, was diagnosed positive for CWD. The letter states that on 7 Feb 98 an elk (tagged as "Blue 40") was sold to T/D's Elk Farm in Bloomer, Wisconsin.

The webmaster is not aware of any follow-up on "Blue 40" or any other animal on either recipient Wisconsin farm; these animals are not necessarily infected or a risk though the potential is there. None of the game farms mentioned here did anything improper, imprudent, or illegal -- CWD emerged as a serious concern subsequent to these animal transfers; there was no way of knowing the status of a given game farm at the time. There is no indication that anyone sold or purchased an animal known to be at risk of CWD. The game farms here cannot be singled out as being at more risk than other game farms nationwide which have trace-back connections, direct or indirect, to Colorado.

As noted in McCombie's article, the first shipment of farm elk from areas with CWD in the wild occurred in 1992, with 66 Colorado elk going to a game farm in Plymouth, Wisconsin. The above is apparently the first shipment to Wisconsin from an elk farm with a confirmed case of CWD.

The second public document, dated 27 May 98, on state letterhead from Dr. Robert Ehlenfeldt, Wisconsin Bureau of Animal Disease Control, to the owner of an elk game farm in West Bend, Wisconsin, provides trace-back provided by the State of Colorado, which according to the letter, identifies the Elk Echo Ranch in Stoneham, Colorado as the source herd for the CWD-diagnosed elk in Nebraska and further imported Wisconsin elk. it is important to stress that no CWD is suspected or diagnosed so far in Wisconsin cervids at any facility.

Dr. Ehlenfeldt says that "the state of Wisconsion currently has no rules covering CWD and is taking no action at this time. No restrictions are being placed on your herd."

Stoneham, Colorado is located near the Pawnee National Grasslands in Weld County, which borders to the east Larimer County and the epicenter of every case of CWD, the Foothills Research Station in Ft. Collins. It is entirely possible that surplus elk were sold from the Foothills Research Station to the game farm.

In the webmaster's opinion, the primary responsibility for the creation and spread of CWD falls upon the incompetent and unethical cervid nutrition researchers at the Foothills Research Station who let 67 cervids die of an infectious disease (said by staff in 1967, repeat 1967, to resemble scrapie) over a 13 year period without conducting an autopsy, while selling off surplus infected animals to zoos, game farms, and 5 other research facilities, not to mention releasing infected animals to the wild.

This facility was so shabbily constructed and maintained that according to their annual reports, feral dogs in the Ft. Collins area were repeatedly allowed to dig under the fences to attack and kill captive deer. In short, CWD, like killer bees, the algae Caulerpa taxifolia, and many others, was released into the environment by careless research scientists.

McCombie reports that in Dec 1999, researchers Mike Miller, Beth Williams, et al, reported on CWD at a Geneva, Switzerland TSE conference hosted by the World Health Organization conference. Gregory Raymond, in Caughey's lab, spoke on prion conversions in vitro. CWD prions converted human prions to a reliable proxy of the infectious form. This was counterbalanced by saying conversion is "weak" and transmission rates "low."

Raymod states: "PrP CWD induced moderate conversion of ovine PrP-sen and similarly weak conversions of human and bovine PrP-sen molecules. The efficiency of PrP CWD -induced conversion of human PrP-sen was similar to that induced by PrP BSE and less than that induced by ov-PrP Sc."

Similar to that induced by PrP BSE? Hello, is anybody home??? We have a BSE-to-human epidemic epidemic going on. Yet Colorado DoW still sold game tags to Larimer County in the fall of 1999 despite their people speaking at this WHO conference. Surely this is not public policy based on the best available science.

In their recommendations, they concluded that "there is currently no evidence that CWD in Cervidae is transmitted to humans..." (safe enough: no one has a method for diagnosing such an event) but go on to recommend that people don't eat CWD infected animals or feed them to other animals. See section 4.3, with recommendations at the end, of this pdf.

Note a single Colorado herd sourced both Nebraska herds [see below] and also supplied animals to 2 Wisconsin game farms; Colorado has not publicly released a full set of trace-forwards from this herd nor is the source of its animals known. A second herd in Colorado had a positive case in an animal from an unidentified, possibly third Colorado game farm.

The third document, dated 23 Sept 98, on Wisconsin Commerical Deer & Elk Farmer's Association letterhead from its president Mike Monson to the Wisconsin State Veterinarian, states that "the mention of a moratorium [on importing elk from CWD affected states until a diagnostic test is available, suggested by Wisconsin Dept of Natural Resourses] is not only premature, but shows, in my opinion that some people in the DNR are out to get us. We made it out of the department regulation [from DNR to the friendlier state ag dept] and as you know we have been a very cooperative and clean industry. A moratorium imposed by the DNR smacks of the way the DNR has always looked at our industry and why the industry has been so slow to grow, and provide the venison the US consumer is demanding...."

Recall that "A Model Program for Surveillance, Control, and Eradication of Chronic Wasting Disease (CWD) in Domestic Elk" presented at the October 1998 meeting of the United States Animal Health Association (USAHA) and developed through the initiative of the North American Elk Breeders Association, sets minimum standards for herd surveillance and monitoring by calling for voluntary CWD testing of all elk 16 months or older that die in a herd. An annual herd inventory with verification by a state or federal animal health officer would be made, and elk herds would obtain a graded status depending upon the number of years monitored without evidence of CWD. Animals could be transferred among herds of equal or lesser status.

In the event of a diagnosis of CWD in a captive elk herd, the Model outlines mandatory steps for herd disposition. The length of the quarantine period depends upon whether the state veterinarian believes there is evidence of spread of CWD within the herd as opposed to a single case. The minimum quarantine for high risk animals (those that were in contact with a CWD-positive elk) is 4 years. Herd surveillance, which includes mandatory death reporting and CWD testing on all dead animals, must be done for 5 years after the last CWD case is diagnosed. [Dr.Victor Nettles, SCWDS Briefs, Jan 99, a newsletter from the Southeastern Center for Wildlife Disease Study at the University of Georgia]

There are provisions for sacrificing and testing the high-risk animals in the herd. High risk animals are elk that were pen-mates of an affected elk for any time up to 1 year prior to the death of the affected animal. Trace-back and trace-forward elk herds must be monitored for 3 years from the date of exposure to the affected animal.

The Model has been distributed to all state veterinarians. Although the surveillance aspect in the Model is intended to be voluntary among elk farmers, it is likely that many state veterinarians will make it mandatory. [Few did.]

The questionnaire CWD Survey by SCWDS found 6 of 31 responding states identified and pursued diagnostic evaluation on high-risk symptomatic animals; individuals in 13 states in addition to Colorado and Wyoming collected brains from 1,830 normal wild cervids for surveillance.

Another CWD case in Nebraska occurred in a captive elk in Nebraska in a captive herd of approximately 10 animals in Cheyenne County, Nebraska near Scottsbluff. It died on April 26, 1999. The Nebraska Game and Parks Commission will periodically check this herd and wild elk and deer in the vicinity for clinical signs of CWD.

According to the SCWDS newsletter, all captive elk on this newly diagnosed farm came from a single source herd in Colorado, which was the same source herd [Elk Echo Ranch in Stoneham, Colorado] where the first CWD elk in Nebraska of April 1998 was diagnosed (SCWDS BRIEFS Vol. 14, No. 1). There is an investigation underway to determine if other Nebraska elk herds may have received elk from this Colorado herd as well. There were no movements of elk from the second affected Nebraska herd since it was established in March 1997. Nebraska Game and Parks Commission personnel collected a wild elk that had been interacting with the captive elk through the fence; the brainstem from this wild elk was negative for CWD.

In September, 1999 according to this same wildlife veterinarian newsletter, CWD was diagnosed in a single elk from a small alternative livestock farm in Colorado. The herd was placed under quarantine and arrangements are being made for the state to purchase and depopulate the animals [not explained why public should pay for this]. Approximately 18 months prior to the CWD diagnosis, the affected elk was purchased from a larger commercial herd in Colorado, and this larger herd remains under quarantine until the completion of an epidemiological investigation. Colorado initiated mandatory CWD surveillance of privately-owned cervids in 1998. {SCWDS Briefs, John Fischer, October 1999]

remember, this was in 1999. ...TSS

see full text and more here ;

Thursday, March 29, 2012



Sunday, March 25, 2012

Three more cases of CWD found in free-ranging deer in Macon County

Tuesday, January 24, 2012

CWD found in two free-ranging deer from Macon County Missouri

Friday, October 21, 2011

Chronic Wasting Disease Found in Captive Deer Missouri

Friday, February 26, 2010

Chronic wasting disease found in Missouri deer

Captive Deer & Elk (Cervids) in Missouri

Captive Cervids: Elk, elk-hybrids, red deer, roe deer, sika deer, white-tailed deer, mule deer, moose, and related species in the Cervidae family, raised under confinement or agricultural conditions for the production of meat or other agricultural products or for sport or exhibition, and free-ranging cervidae when they are captured for any purpose.

The majority of farm-raised cervidae in Missouri are either Rocky Mountain elk, considered livestock which fall under the regulations of the Missouri Department of Agriculture, or White-tailed deer, wildlife, which fall under the regulations of the Missouri Department of Conservation.

For information concerning health requirements governing the admission of CWD status herds of Elk, elk-hybrids, red deer, roe deer, sika deer, white-tailed deer, mule deer, and moose sold, traded, exchanged, leased, donated, relinquished or otherwise involved in a change of ownership contact the Division of Animal Health at (573) 751-2267, or e-mail


from a TSE Prion aka mad cow disease point of regulatory efforts, the USDA et al fumbled, bumbled, lost the football so many times, well, see for yourself. there is too much evidence to post here, for those interested, please see below, these links will lead you to that evidence. in short, the USDA (USA) had the gold card for trading, and boasted all the time about it, up and until that fateful day in Dec. of 2003. all things changed, you see, the gold card consisted of the USDA being able to corner the markets. simply because the USDA never intended to find any BSE with the infamous, enhanced, 2004 BSE surveillance program. from day one that program was set up to fail, and the BSE Harvard Risk assessment proved this. from bumbled testing protocols where you never would have a positive, only testing one part of the brain, with only the less sensitive testing, or, where the gosh darn guy collecting the suspect mad cow brains for the USDA to be tested, IS BRINGING ONLY HEALTHY BRAINS TO BE TESTED, to a failed BSE feed ban. the august 4, 1997, partial and voluntary mad cow feed ban was nothing but ink on paper. where as late as 10 years after said mad cow feed ban, some 10,000,000 pounds of suspect, banned, mad cow feed was fed out in commerce, where today, said feed ban still fails. remember .005 grams of BSE TSE prion tainted feed, is enough to kill a small herd of cows. from that Washington mad cow old dave capped, to the 1st documented mad cow in Texas they covered up i.e. stumbling and staggering highly suspect mad cow that officials in Austin made the final decision to rush that madcow to the render, to be chopped up for pet food, BEFORE ANY TEST COULD BE DONE. next mad cow in Texas, i.e. the infamous FONG madcow ............maybe TSSMADCOW too, that stumbling and staggering mad cow...went missing in action for a bit. I confirmed it some 7 months before myself and others finally got the USDA to confirm that madcow via Weybridge, and literally, an act of Congress, thanks to the Honorable Phyllis Fong of the OIG, and some scientist around the world...and me too...really, that’s my dang madcow. 7 months later, and after the gosh darn USDA during this 7 months, where that texas madcow tissue samples sat up on a shelf, or in a freezer, for the BSE MRR to be ratified. and it was. this made legal, the trading of all strains of TSE Prion mad cow disease to be traded globally, the Bovine Spongiform Encephalopathy Minimal Risk Region i.e. BSE MRR. IT did away with the Bovine Spongiform Encephalopathy Geographical Risk Assessments i.e BSE GBR. the BSE GBR was set forth with real science put forth, the BSE MRR was junk science, set up for trade $$$ you see, humans are expendable, due to the long incubation period. a few body bags will not matter. but they are slowly mounting, and the huge human health risk factor here is the iatrogenic aspect, i.e. friendly fire, pass it forward mode, i.e. blood, tissue, surgery, dental, that’s when all this becomes every persons problem. see correspondence between myself and TAHC. well, from there we go to Alabama. now, the Washington madcow was supposedly a Canadian mad cow sent to the USA. the USA and Canada traded cattle, livestock, feed, byproducts, like two lovers swapping spit. The Washington Canadian/USA madcow was a typical c-BSE madcow. Now, the second madcow in Texas, the one they finally decided to test after 7+ months, of a 48 hour confirmation turn around, so the BSE MRR policy could be ratified, was an atypical h-BSE madcow. moooving right along to the Alabama mad cow. the Alabama madcow was a strange type, it was atypical g-h-BSE i.e. genetic h-type atypical BSE. this is important, in the fact this cow matches exactly with the rising subtype of sporadic CJD in humans, what I call tpsCJD type pending sporadic CJD. right after that, the USDA decided to shut testing down to numbers so small (OIE standards), the odds of finding a madcow is nil. I am telling you, whether you want to hear it or not, North America, we have a problem, and the problem is slowly catching up with us $$$ really...

Thursday, February 16, 2012

Bovine Spongiform Encephalopathy BSE


Thursday, February 23, 2012


Monday, November 30, 2009


madcow disease was in the USA decades ago...

Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME.


The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle...

Response to Public Comments

on the

Harvard Risk Assessment of Bovine Spongiform Encephalopathy Update, October 31, 2005


The United States Department of Agriculture’s Food Safety and Inspection Service (FSIS) held a public meeting on July 25, 2006 in Washington, D.C. to present findings from the Harvard Risk Assessment of Bovine Spongiform Encephalopathy Update, October 31, 2005 (report and model located on the FSIS website:

Comments on technical aspects of the risk assessment were then submitted to FSIS.

Comments were received from Food and Water Watch, Food Animal Concerns Trust (FACT), Farm Sanctuary, R-CALF USA, Linda A Detwiler, and Terry S. Singeltary.

This document provides itemized replies to the public comments received on the 2005 updated Harvard BSE risk assessment. Please bear the following points in mind:

Suppressed peer review of Harvard study October 31, 2002.

October 31, 2002 Review of the Evaluation of the Potential for Bovine Spongiform Encephalopathy in the United States Conducted by the Harvard Center for Risk Analysis, Harvard School of Public Health and Center for Computational Epidemiology, College of Veterinary Medicine, Tuskegee University Final Report Prepared for U.S. Department of Agriculture Food Safety and Inspection Service Office of Public Health and Science Prepared by RTI Health, Social, and Economics Research Research Triangle Park, NC 27709 RTI Project Number 07182.024

Sunday, February 14, 2010

[Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine Spongiform Encephalopathy (BSE)



Terry S. Singeltary

From: Terry S. Singeltary Sr. []

Sent: Thursday, September 08, 2005 6:17 PM


Subject: [Docket No. 03-025IFA] FSIS Prohibition of the Use of Specified Risk Materials for Human Food and Requirements for the Disposition of Non-Ambulatory Disabled Cattle

Greetings FSIS,

I would kindly like to submit the following to [Docket No. 03-025IFA] FSIS Prohibition of the Use of Specified Risk Materials for Human Food and Requirements for the Disposition of Non-Ambulatory Disabled Cattle


When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE. In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.

This study will contribute to a correct definition of specified risk material (SRM) in atypical BSE. The incumbent of this position will develop new and transfer existing, ultra-sensitive methods for the detection of atypical BSE in tissue of experimentally infected cattle.

Thursday, August 12, 2010

Seven main threats for the future linked to prions

First threat The TSE road map defining the evolution of European policy for protection against prion diseases is based on a certain numbers of hypotheses some of which may turn out to be erroneous. In particular, a form of BSE (called atypical Bovine Spongiform Encephalopathy), recently identified by systematic testing in aged cattle without clinical signs, may be the origin of classical BSE and thus potentially constitute a reservoir, which may be impossible to eradicate if a sporadic origin is confirmed.

***Also, a link is suspected between atypical BSE and some apparently sporadic cases of Creutzfeldt-Jakob disease in humans. These atypical BSE cases constitute an unforeseen first threat that could sharply modify the European approach to prion diseases.

Second threat snip...

EFSA Journal 2011 The European Response to BSE: A Success Story

This is an interesting editorial about the Mad Cow Disease debacle, and it's ramifications that will continue to play out for decades to come ;

Monday, October 10, 2011 EFSA Journal 2011

The European Response to BSE: A Success Story


EFSA and the European Centre for Disease Prevention and Control (ECDC) recently delivered a scientific opinion on any possible epidemiological or molecular association between TSEs in animals and humans (EFSA Panel on Biological Hazards (BIOHAZ) and ECDC, 2011). This opinion confirmed Classical BSE prions as the only TSE agents demonstrated to be zoonotic so far but the possibility that a small proportion of human cases so far classified as "sporadic" CJD are of zoonotic origin could not be excluded. Moreover, transmission experiments to non-human primates suggest that some TSE agents in addition to Classical BSE prions in cattle (namely L-type Atypical BSE, Classical BSE in sheep, transmissible mink encephalopathy (TME) and chronic wasting disease (CWD) agents) might have zoonotic potential.


see follow-up here about North America BSE Mad Cow TSE prion risk factors, and the ever emerging strains of Transmissible Spongiform Encephalopathy in many species here in the USA, including humans ;

Saturday, June 25, 2011

Transmissibility of BSE-L and Cattle-Adapted TME Prion Strain to Cynomolgus Macaque "BSE-L in North America may have existed for decades"

Monday, March 19, 2012

Infectivity in Skeletal Muscle of Cattle with Atypical Bovine Spongiform Encephalopathy

PLoS One. 2012; 7(2): e31449.

Sunday, March 11, 2012

APHIS Proposes New Bovine Spongiform Encephalopathy Import Regulations in Line with International Animal Health Standards Proposal Aims to Ensure Health of the U.S. Beef Herd, Assist in Negotiations

Thursday, March 29, 2012

atypical Nor-98 Scrapie has spread from coast to coast in the USA 2012

NIAA Annual Conference April 11-14, 2011San Antonio, Texas

really, in my opinion, the USDA et al hung the cervid industry out to dry decades ago, in relations to the CWD TSE prion disease. now, they want to just deregulate, by proxy, i.e. LAX OVERSIGHT, or no oversight at all, and the almighty hunt for the dollar $$$ the DNR does/did not want CWD in your state. the USDA et al don’t care $$$


MOM DOD 12/14/97 hvCJD ‘confirmed’

Terry S. Singeltary Sr.

P.O. Box 42

Bacliff, Texas USA 77518



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