Texas Pair Convicted in Illegal Deer Breeding Operation TPWD
News Release Media Contact: Steve Lightfoot, 512-389-4701,
steve.lightfoot@tpwd.texas.gov
Oct. 21, 2014
Pair Convicted in Illegal Deer Breeding Operation AUSTIN – The latest
chapter in a decade long series of criminal and wildlife disease investigations
involving a former South Texas deer breeder ended recently when a Corpus Christi
area couple pleaded guilty to 50 charges of Unlawful Possession and/or Sale of
Live Game Animals.
Frank Thomas Shumate Jr., 51, and Kalub Rogers Shumate, 31, were each
assessed $14,127.50 in fines and agreed to surrender the ability to apply for a
deer breeder permit or a hunting lease license for all time. Mr. Shumate also
agreed to surrender his hunting license through the end of the 2015 license year
and Ms. Shumate through the end of the 2017 license year.
The criminal cases were adjudicated in the office of Hon. Caroline
Korzekwa, Karnes County Justice Court Precinct. 2.
Retired San Antonio attorney Rene Barrientos served as special prosecutor
in the case with approval and support from the Karnes County Attorney. He also
coordinated a global agreement in Travis County District Court to resolve a
pending civil case against Ms. Rogers and recover $34,080 in restitution related
to expenses incurred by TPWD staff while conducting a deer herd inventory
inspection and disease sampling at a deer breeding facility permitted to Ms.
Rogers.
Investigation into Mr. Shumate’s deer breeding activities began in March
2004 and led to multiple charges in three counties. Two years later, his deer
breeder violations resulted in 10 convictions on criminal charges in Jim Wells
County, 5 convictions in Nueces County and 5 convictions in Webb County. As a
result of these findings, Mr. Shumate agreed to relinquish his Scientific
Breeder’s Permit and liquidate all deer held in captivity in his deer breeding
facility in Nueces County.
In advance of losing his deer breeding privileges, Mr. Shumate allegedly
initiated a plan to have a new deer breeding facility permitted in the name of
Kalub Rogers on his property in Karnes County, where he then transferred more
than 100 deer from his Nueces County facility.
Over time the TPWD Special Operations Unit received numerous reports that
Mr. Shumate was reportedly still in the deer business and was buying and selling
deer for which he was not legally authorized by TPWD. An investigation revealed
that Mr. Shumate conducted sales of at least 78 white-tailed deer from Ms.
Rogers’ deer breeding facility to ranches for release into the wild for stocking
purposes since September 2010. Mr. Shumate received a minimum of $171,466 in
payments for white-tailed deer he unlawfully sold, which according to records
submitted to TPWD, were transported from Ms. Rogers’ deer breeding facility. The
investigation further indicated that Kalub Rogers was holding a deer breeder
permit in her name on behalf of her husband Frank Thomas Shumate Jr.
Ms. Rogers’ facility came under additional scrutiny in 2012, initially as a
result of a delinquent annual report required of all permitted deer breeding
facilities. Criminal charges were filed when a subsequent herd inspection and
inventory revealed significant irregularities and discrepancies, including 162
inventoried deer that were missing from the facility. During the herd
inspection, TPWD wildlife biologists noted the remaining deer in the facility to
be in poor condition and numerous decayed deer carcasses were observed
throughout the pens.
The observation of 142 deer of unknown origin was cause for additional
concern and tissue samples from several deer were tested for both Chronic
Wasting Disease and Bovine Tuberculosis. Neither disease was detected.
“Unscrupulous actions by these two individuals are not only a threat to all
the law-abiding deer breeders who are carefully monitoring and managing their
facilities, but also to the state’s free-ranging deer, which can be exposed to
unnecessary disease risk from these illegitimate activities,” said Mitch
Lockwood, TPWD Big Game Program Director. “Captive and free-ranging deer are too
important to our state to have them compromised by the actions of a few.”
SL 2014-10-21
> The observation of 142 deer of unknown origin was cause for additional
concern and tissue samples from several deer were tested for both Chronic
Wasting Disease and Bovine Tuberculosis. Neither disease was detected.
‘’several deer’’, two deer tested for cwd?...seriously???
they did not want to find cwd apparently.
I am not surprised...tss
MR. KURZAWSKI: Thank you, commissioner.
COMMISSIONER DUGGINS: All right, good. We are at Item 7, Rules Concerning
Implementation of Senate Bill 820, Deer Permits, recommended adoption of
proposed changes, Mitch Lockwood. Welcome.
MR. LOCKWOOD: Thank you. Good morning, Mr. Chairman, Commissioners. As we
discussed yesterday, the 83rd Texas Legislature enacted Senate Bill 820, which
requires the Department to issue a deer breeders permit with a duration of one
year, three years, or five years.
Well, we already had the authority to issue a deer breeders permit for
multiple years; but as I discussed yesterday, we had opted to limit the duration
of those permits to one year and one reason for that was to try and not remove
an incentive to submit annual reports in a timely fashion. But I admit that it
did not prove to be a very effective incentive.
Nonetheless, Senate Bill 820 did amend Parks and Wildlife Code to ensure
that we do issue a three-year or a five-year permit to someone who meets a
couple of criteria. But the rule -- the statute also gives the Commission the
authority to establish some additional criteria, which is the purpose or the
intent of this proposal. In essence, we're proposing only one additional
criteria, which is that we would issue a multiyear permit to someone who has
been in substantial compliance with the deer breeder regulations and statutes
for the previous three years.
The idea behind this is to try an prevent invoking or at least minimize the
risk of invoking a very lengthy and inefficient permit revocation process. You
know, there may be some instances in which some permitted individuals just
refuse to comply with annual reporting requirements, which we find to be very
important in order to reconcile these herd inventories that affect the entire
industry. And so in the event someone does refuse to comply with those
requirements, it may give cause to revoke this longterm or multiyear permit.
We think we can avoid implementing or invoking that lengthy process if we
can ensure or at least issue this permit to someone who has demonstrated for the
previous three years that they have complied with that reporting requirement and
with some other regulations. Again, we don't propose any change to the annual
permit fee of $200 and so a three-year permit would that $200 times three years
at $600 and a five-year permit would be a thousand dollars.
So with that, staff recommend that the Texas Parks and Wildlife Commission
adopts amendments to 53.14 concerning deer management and removal permits and
65.603 concerning application of permit issuance, with changes as necessary to
the proposed text as published in the October 4th, 2013, issue of the Texas
Register.
We have received two public comments, in addition to some input I received
from the breeder user group who is supportive of this proposal. Again, they did
want to qualify the term compliance by stating "substantial compliance" and so
with that, they're supportive of this proposal. Of the two comments we received,
one was in favor, one was in opposition; but the comments that accompanied the
opposing view was -- really were not germane to this proposal. He was opposed to
the practice of deer breeding altogether.
And so that concludes my presentation, and I'll be glad to entertain any
questions you might have.
Singeltary Submission TAHC on CWD rule proposal
Saturday, July 07, 2012
TEXAS Animal Health Commission Accepting Comments on Chronic Wasting
Disease Rule Proposal
Considering the seemingly high CWD prevalence rate in the Sacramento and
Hueco Mountains of New Mexico, CWD may be well established in the population and
in the environment in Texas at this time.
Media Contacts:
Steve Lightfoot, TPWD, 512-389-4701, steve.lightfoot@tpwd.state.tx.us
Yvonne "Bonnie" Ramirez, TAHC, 512-719-0710,
bonnie.ramirez@tahc.state.tx.us
FOR IMMEDIATE RELEASE
July 10, 2012
Chronic Wasting Disease Detected in Far West Texas
AUSTIN -- Samples from two mule deer recently taken in far West Texas have
been confirmed positive for Chronic Wasting Disease (CWD). These are the first
cases of CWD detected in Texas deer. Wildlife officials believe the event is
currently isolated in a remote part of the state near the New Mexico border.
snip...
###
Texas Animal Health Commission (TAHC) 2105 Kramer Lane Austin, Texas 78758
1-800-550-8242
TEXAS CHRONIC WASTING DISEASE CWD Four New Positives Found in Trans Pecos
News Release
Media Contact: Steve Lightfoot, 512-389-4701,
steve.lightfoot@tpwd.state.tx.us
Feb. 11, 2013
Four New Positives Found in Trans Pecos CWD Surveillance
Disease not discovered outside Containment Zone
AUSTIN – Nearly 300 tissue samples were collected from hunter harvested
mule deer from the Trans Pecos ecoregion of far West Texas during the 2012-13
season for Chronic Wasting Disease (CWD) testing. Texas A&M Veterinary
Medical Diagnostic Laboratory and National Veterinary Services Laboratories
(NVSL) have confirmed CWD in four of those samples. All CWD-positive deer were
harvested within the CWD Containment Zone.
SL 2013-02-11
7 MR. BROWN: Madam Chairman, Members, 8 my name is Kirby Brown with the
Texas Wildlife 9 Association. We want to thank the Commission and 10 the staff
for their efforts in this. And, Madam 11 Chairman, your leadership in this
action that's 12 going forward, we appreciate the rapid response, 13 the
professionalism and the science being brought 14 to bear on the issue. We think
that's all very 15 important. 16 We are continuing to work with the 17 Texas
Animal Health Commission on the issue. Our 18 meeting over the next few weeks --
in fact, Monday 19 we have a meeting with Commissioner Wood. And 20 although our
membership continues to have real 21 concern about CWD and entry into the wild,
we 22 think the voluntary actions that are taking place 23 are proceeding at a
rapid pace at this point in 24 time. And we'd like to see that continue. And we
25 believe that's a process, especially with the . 38 1 response that we can get
for herd health plans 2 from the Texas Animal Health Commission staff, 3 which
is -- they're literally overwhelmed right 4 now with so many issues that they
have on their 5 plate. We think this is a good process, we think 6 it's an
effective process, and we appreciate your 7 consideration in that regard. So
with that, 8 that's all I have to say. Thank you very much. 9 CHAIRMAN IDSAL:
After Karl, we have 10 Ellis Gilleland. 11 MR. KINSEL: I second entirely what 12
Kirby just said, but I also wanted to bring it to 13 y'all's attention that the
letter is drafted going 14 out to all scientific breeders, all 467, both TWA 15
and TDA have individually drafted that letter. We 16 intend to do that jointly
to continue showing our 17 joint support and our joint efforts. As soon as 18 we
get clarification from TAHC, that will go as a 19 cover letter on form 0008 of
the Texas Animal 20 Health Commission to enroll as many as possible 21 into the
program. Thank you. 22 CHAIRMAN IDSAL: We may have some 23 questions.
Commissioner Fitzsimons has a question 24 of Mr. Kinsel.
snip...
19 The second thing I want to say is, 20 voluntary law does not work.
George W. Bush 21 approved that. The cold fired power plants 22 throughout this
state that are grandfathered from 23 30, 40 years ago are still belching. They
keep 24 the shell, they rebuild the guts and keep the same 25 within the
grandfather clause. Voluntary -- you . 43 1 lawyers know that. People will take
advantage of 2 voluntary law. It does not work. And it's not in 3 your mission
statement. Everybody want to do 4 this? No. Okay. Don't do it. Everybody don't 5
want to do it? Yeah, okay, do it. That's not 6 government. That's not what
you're being paid to 7 do. So forget about the voluntary stuff. Make a 8 law.
Jerry gave you a good first step. Make a 9 law, there it is. Now, get the
voluntary 10 compliance once you've got the law. That's where 11 you want the
voluntary. Not voluntary eons and 12 eons. There's no -- that's not law. That's
an 13 anarchy. 14 The third thing I want to mention, 15 there's no precedence
for voluntary law. The 16 precedence are coherent laws. We are a nation of 17
laws. Rabies -- let's bring it down to home. 18 Rabies in Texas. Take away the
rabies law and 19 what have we got? We've got a bunch of dead 20 people without
rabies law. There's got to be a 21 CWD law or in a year or two or five years
from now 22 we're going to all be in trouble and go back and 23 all these
(inaudible), well, let's go back and see 24 what happened. Well, we don't have
to do that. 25 Let's take the action now. Get the consensus, get . 44 1 the
voluntary, yeah, yeah, yeah, make a law. Now, 2 do it, and there's a penalty.
Please, the 3 politics are not a factor in this. The heck with 4 them. They can
start dipping ice cream at Dairy 5 Queen or something. They don't have to raise
a 6 deer. Thank you. 7 CHAIRMAN IDSAL: Does staff have any 8 comments or
response on these comments? Does the 9 Commission have any comments? 10 If there
are no comments, is there a 11 motion on this item? 12 COMMISSIONER MONTGOMERY:
Joseph. 13 CHAIRMAN IDSAL: Joseph, did you 14 have a comment? 15 COMMISSIONER
FITZSIMONS: I would 16 move we would adopt the second recommendation. 17
COMMISSIONER MONTGOMERY: Which is 18 to withdraw -- to postpone it? 19
COMMISSIONER RAMOS: To postpone it. 20 CHAIRMAN IDSAL: To postpone it. 21
COMMISSIONER RAMOS: If that's your 22 motion, I'll second that. 23 CHAIRMAN
IDSAL: All in favor? 24 COMMISSIONER FITZSIMONS: And 25 republish.
Monday, February 11, 2013
TEXAS CHRONIC WASTING DISEASE CWD Four New Positives Found in Trans
Pecos
Thursday, October 03, 2013
*** TAHC ADOPTS CWD RULE THAT the amendments __REMOVE__ the requirement for
a specific fence height for captives ***
Texas Animal Health Commission (TAHC)
October 3, 2013
Monday, June 23, 2014
*** PRION 2014 CONFERENCE CHRONIC WASTING DISEASE CWD
Tuesday, July 01, 2014
CHRONIC WASTING DISEASE CWD TSE PRION DISEASE, GAME FARMS, AND POTENTIAL
RISK FACTORS THERE FROM
Thursday, July 03, 2014
How Chronic Wasting Disease is affecting deer population and what’s the
risk to humans and pets?
Saturday, July 07, 2012
TEXAS Animal Health Commission Accepting Comments on Chronic Wasting
Disease Rule Proposal
Considering the seemingly high CWD prevalence rate in the Sacramento and
Hueco Mountains of New Mexico, CWD may be well established in the population and
in the environment in Texas at this time.
Tuesday, July 10, 2012
Chronic Wasting Disease Detected in Far West Texas
Monday, February 11, 2013
TEXAS CHRONIC WASTING DISEASE CWD Four New Positives Found in Trans
Pecos
Thursday, October 03, 2013
*** TAHC ADOPTS CWD RULE THAT the amendments __REMOVE__ the requirement for
a specific fence height for captives ***
Texas Animal Health Commission (TAHC)
October 3, 2013
Saturday, October 18, 2014
Chronic wasting disease threatens Canadian agriculture, Alberta MLA
says
10/21/2014
Tenth Pennsylvania Captive Deer Tests Positive for Chronic Wasting
Disease
News for Immediate Release Oct. 21, 2014 Harrisburg – The Pennsylvania
Department of Agriculture today announced a tenth captive deer has tested
positive for Chronic Wasting Disease in Pennsylvania since the disease was
discovered on a captive deer farm in 2012. The seven year old white-tailed deer
died at a farm in Reynoldsville, Jefferson County, on Sept. 18, and tested
positive for the disease at the Pennsylvania Veterinary Laboratory in
Harrisburg. Four Jefferson County deer farms where the deer had lived are under
quarantine. Currently 31 captive deer farms across the state are quarantined,
where deer cannot be moved on or off the properties. The investigation continues
and additional herds may be quarantined. There is no evidence that humans or
livestock can get the disease, according to the Centers for Disease Control and
Prevention. Chronic Wasting Disease attacks the brain of infected deer, elk and
moose, producing small lesions that eventually result in death. Animals can get
the disease through direct contact with saliva, feces and urine from an infected
animal. Symptoms include weight loss, excessive salivation, increased drinking
and urination, and abnormal behavior like stumbling, trembling and depression.
Infected deer and elk may also allow unusually close approach by humans or
natural predators. The disease is fatal and there is no known treatment or
vaccine. The first cases of CWD in Pennsylvania were detected when two Adams
County deer tested positive for Chronic Wasting Disease in 2012. Surveillance
for the disease has been ongoing in Pennsylvania since 1998. The Department of
Agriculture coordinates a mandatory surveillance program for more than 23,000
captive deer on 1,100 breeding farms, hobby farms and shooting preserves. Ten
captive deer have tested positive since 2012. The Pennsylvania Game Commission
collects samples from hunter-harvested deer and elk and those that appear sick
or behave abnormally. Since 1998, the commission has tested more than 48,000
free-ranging deer and elk for the disease. Six wild deer have tested positive
for the disease since 2013. For more information, visit
www.agriculture.state.pa.us and click on the “Chronic Wasting Disease
Information” button. Media contact: Samantha Elliott Krepps, 717-787-5085 ###
>>> There is no evidence that humans or livestock can get the
disease, according to the Centers for Disease Control and Prevention.
hang on now, what do you call this ;
> First transmission of CWD to transgenic mice over-expressing bovine
prion protein gene (TgSB3985)
PRION 2014 - PRIONS: EPIGENETICS and NEURODEGENERATIVE DISEASES – Shaping
up the future of prion research
Animal TSE Workshop 10.40 – 11.05 Talk Dr. L. Cervenakova First
transmission of CWD to transgenic mice over-expressing bovine prion protein gene
(TgSB3985)
FORGOT TO ADD THIS ONE...
P.126: Successful transmission of chronic wasting disease (CWD) into mice
over-expressing bovine prion protein (TgSB3985)
Larisa Cervenakova,1 Christina J Sigurdson,2 Pedro Piccardo,3 Oksana
Yakovleva,1 Irina Vasilyeva,1 Jorge de Castro,1 Paula Saá,1 and Anton Cervenak1
1American Red Cross, Holland Laboratory; Rockville, MD USA; 2University of
California; San Diego, CA USA; 3Lab TSE/OBRR /CBER/FDA; Rockville, MD USA
Keywords: chronic wasting disease, transmission, transgenic mouse, bovine
prion protein
Background. CWD is a disease affecting wild and farmraised cervids in North
America. Epidemiological studies provide no evidence of CWD transmission to
humans. Multiple attempts have failed to infect transgenic mice expressing human
PRNP gene with CWD. The extremely low efficiency of PrPCWD to convert normal
human PrPC in vitro provides additional evidence that transmission of CWD to
humans cannot be easily achieved. However, a concern about the risk of CWD
transmission to humans still exists. This study aimed to establish and
characterize an experimental model of CWD in TgSB3985 mice with the following
attempt of transmission to TgHu mice.
Materials and Methods. TgSB3985 mice and wild-type FVB/ NCrl mice were
intracranially injected with 1% brain homogenate from a CWD-infected Tga20 mouse
(CWD/Tga20). TgSB3985 and TgRM (over-expressing human PrP) were similarly
injected with 5% brain homogenates from CWD-infected white-tailed deer (CWD/WTD)
or elk (CWD/Elk). Animals were observed for clinical signs of neurological
disease and were euthanized when moribund. Brains and spleens were removed from
all mice for PrPCWD detection by Western blotting (WB). A histological analysis
of brains from selected animals was performed: brains were scored for the
severity of spongiform change, astrogliosis, and PrPCWD deposition in ten brain
regions.
Results. Clinical presentation was consistent with TSE. More than 90% of
TgSB3985 and wild-type mice infected with CWD/Tga20, tested positive for PrPres
in the brain but only mice in the latter group carried PrPCWD in their spleens.
We found evidence for co-existence or divergence of two CWD/ Tga20 strains based
on biochemical and histological profiles. In TgSB3985 mice infected with CWD-elk
or CWD-WTD, no animals tested positive for PrPCWD in the brain or in the spleen
by WB. However, on neuropathological examination we found presence of amyloid
plaques that stained positive for PrPCWD in three CWD/WTD- and two
CWD/Elk-infected TgSB3985 mice. The neuropathologic profiles in CWD/WTD- and
CWD/Elkinfected mice were similar but unique as compared to profiles of BSE,
BSE-H or CWD/Tg20 agents propagated in TgSB3985 mice. None of CWD-infected TgRM
mice tested positive for PrPCWD by WB or by immunohistochemical detection.
Conclusions. To our knowledge, this is the first established experimental
model of CWD in TgSB3985. We found evidence for co-existence or divergence of
two CWD strains adapted to Tga20 mice and their replication in TgSB3985 mice.
Finally, we observed phenotypic differences between cervid-derived CWD and
CWD/Tg20 strains upon propagation in TgSB3985 mice. Further studies are underway
to characterize these strains.
TSS
UPDATED CORRESPONDENCE FROM AUTHORS OF THIS STUDY I.E. COLBY, PRUSINER ET
AL, ABOUT MY CONCERNS OF THE DISCREPANCY BETWEEN THEIR FIGURES AND MY FIGURES OF
THE STUDIES ON CWD TRANSMISSION TO CATTLE ;
CWD to cattle figures CORRECTION
Greetings,
I believe the statement and quote below is incorrect ;
"CWD has been transmitted to cattle after intracerebral inoculation,
although the infection rate was low (4 of 13 animals [Hamir et al. 2001]). This
finding raised concerns that CWD prions might be transmitted to cattle grazing
in contaminated pastures."
Please see ;
Within 26 months post inoculation, 12 inoculated animals had lost weight,
revealed abnormal clinical signs, and were euthanatized. Laboratory tests
revealed the presence of a unique pattern of the disease agent in tissues of
these animals. These findings demonstrate that when CWD is directly inoculated
into the brain of cattle, 86% of inoculated cattle develop clinical signs of the
disease.
" although the infection rate was low (4 of 13 animals [Hamir et al.
2001]). "
shouldn't this be corrected, 86% is NOT a low rate. ...
kindest regards,
Terry S. Singeltary Sr. P.O. Box 42 Bacliff, Texas USA 77518
Thank you!
Thanks so much for your updates/comments. We intend to publish as rapidly
as possible all updates/comments that contribute substantially to the topic
under discussion.
re-Prions David W. Colby1,* and Stanley B. Prusiner1,2 + Author
Affiliations
1Institute for Neurodegenerative Diseases, University of California, San
Francisco, San Francisco, California 94143 2Department of Neurology, University
of California, San Francisco, San Francisco, California 94143 Correspondence:
stanley@ind.ucsf.edu
Mule deer, white-tailed deer, and elk have been reported to develop CWD. As
the only prion disease identified in free-ranging animals, CWD appears to be far
more communicable than other forms of prion disease. CWD was first described in
1967 and was reported to be a spongiform encephalopathy in 1978 on the basis of
histopathology of the brain. Originally detected in the American West, CWD has
spread across much of North America and has been reported also in South Korea.
In captive populations, up to 90% of mule deer have been reported to be positive
for prions (Williams and Young 1980). The incidence of CWD in cervids living in
the wild has been estimated to be as high as 15% (Miller et al. 2000). The
development of transgenic (Tg) mice expressing cervid PrP, and thus susceptible
to CWD, has enhanced detection of CWD and the estimation of prion titers
(Browning et al. 2004; Tamgüney et al. 2006). Shedding of prions in the feces,
even in presymptomatic deer, has been identified as a likely source of infection
for these grazing animals (Williams and Miller 2002; Tamgüney et al. 2009b). CWD
has been transmitted to cattle after intracerebral inoculation, although the
infection rate was low (4 of 13 animals [Hamir et al. 2001]). This finding
raised concerns that CWD prions might be transmitted to cattle grazing in
contaminated pastures.
snip...
----- Original Message -----
From: David Colby To: flounder9@verizon.net
Cc: stanley@XXXXXXXX
Sent: Tuesday, March 01, 2011 8:25 AM
Subject: Re: FW: re-Prions David W. Colby1,* and Stanley B. Prusiner1,2 +
Author Affiliations
Dear Terry Singeltary,
Thank you for your correspondence regarding the review article Stanley
Prusiner and I recently wrote for Cold Spring Harbor Perspectives. Dr. Prusiner
asked that I reply to your message due to his busy schedule. We agree that the
transmission of CWD prions to beef livestock would be a troubling development
and assessing that risk is important. In our article, we cite a peer-reviewed
publication reporting confirmed cases of laboratory transmission based on
stringent criteria. The less stringent criteria for transmission described in
the abstract you refer to lead to the discrepancy between your numbers and ours
and thus the interpretation of the transmission rate. We stand by our assessment
of the literature--namely that the transmission rate of CWD to bovines appears
relatively low, but we recognize that even a low transmission rate could have
important implications for public health and we thank you for bringing attention
to this matter. Warm Regards, David Colby -- David Colby, PhDAssistant Professor
Department of Chemical Engineering University of Delaware
===========END...TSS==============
SNIP...SEE FULL TEXT ;
UPDATED DATA ON 2ND CWD STRAIN Wednesday, September 08, 2010 CWD PRION
CONGRESS SEPTEMBER 8-11 2010
Sunday, August 19, 2012
Susceptibility of cattle to the agent of chronic wasting disease from elk
after intracranial inoculation 2012
Research Project: TRANSMISSION, DIFFERENTIATION, AND PATHOBIOLOGY OF
TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHIES Location: Virus and Prion Research
Unit
Thursday, November 21, 2013
*** Assessing the susceptibility of transgenic mice over-expressing deer
prion protein to bovine spongiform encephalopathy
The present study was designed to assess the susceptibility of the
prototypic mouse line, Tg(CerPrP)1536+/- to bovine spongiform encephalopathy
(BSE) prions, which have the ability to overcome species barriers.
Tg(CerPrP)1536+/- mice challenged with red deer-adapted BSE resulted in a
90-100% attack rates, BSE from cattle failed to transmit, indicating agent
adaptation in the deer.
*** The potential impact of prion diseases on human health was greatly
magnified by the recognition that interspecies transfer of BSE to humans by beef
ingestion resulted in vCJD. While changes in animal feed constituents and
slaughter practices appear to have curtailed vCJD, there is concern that CWD of
free-ranging deer and elk in the U.S. might also cross the species barrier.
Thus, consuming venison could be a source of human prion disease. Whether BSE
and CWD represent interspecies scrapie transfer or are newly arisen prion
diseases is unknown. Therefore, the possibility of transmission of prion disease
through other food animals cannot be ruled out. There is evidence that vCJD can
be transmitted through blood transfusion. There is likely a pool of unknown size
of asymptomatic individuals infected with vCJD, and there may be asymptomatic
individuals infected with the CWD equivalent. These circumstances represent a
potential threat to blood, blood products, and plasma supplies.
NOW, what is the latest on human risk factors to CWD strains ???
SNIP...SEE FULL TEXT HERE ;
Tuesday, October 21, 2014
Pennsylvania Department of Agriculture Tenth Pennsylvania Captive Deer
Tests Positive for Chronic Wasting Disease CWD TSE PRION DISEASE
Singeltary Response to USDA, and USDA
RESPONSE TO SINGELTARY ON HARVARD BSE RISK ASSESSMENT
Owens, Julie
From: Terry S. Singeltary Sr. [flounder9@verizon.net]
Sent: Monday, July 24, 2006 1:09 PM
To: FSIS RegulationsComments
Subject: [Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine
Spongiform Encephalopathy (BSE) Page 1 of 98
FSIS, USDA, REPLY TO SINGELTARY
Transmissible Spongiform Encephalopathy TSE Prion Disease North America
2014
Transmissible Spongiform Encephalopathy TSE Prion Disease have now been
discovered in a wide verity of species across North America. typical C-BSE,
atypical L-type BASE BSE, atypical H-type BSE, atypical H-G BSE, of the bovine,
typical and atypical Scrapie strains, in sheep and goats, with atypical Nor-98
Scrapie spreading coast to coast in about 5 years. Chronic Wasting Disease CWD
in cervid is slowly spreading without any stopping it in Canada and the USA and
now has mutated into many different strains. Transmissible Mink Encephalopathy
TME outbreaks. These Transmissible Spongiform Encephalopathy TSE Prion Disease
have been silently mutating and spreading in different species in North America
for decades.
The USDA, FDA, et al have assured us of a robust Triple BSE TSE prion
Firewall, of which we now know without a doubt, that it was nothing but ink on
paper. Since the 1997 mad cow feed ban in the USA, literally tons and tons of
banned mad cow feed has been put out into commerce, never to return, as late as
December of 2013, serious, serious breaches in the FDA mad cow feed ban have
been documented. The 2004 enhanced BSE surveillance program was so flawed, that
one of the top TSE prion Scientist for the CDC, Dr. Paul Brown stated ; Brown,
who is preparing a scientific paper based on the latest two mad cow cases to
estimate the maximum number of infected cows that occurred in the United States,
said he has "absolutely no confidence in USDA tests before one year ago" because
of the agency's reluctance to retest the Texas cow that initially tested
positive.
see ;
The BSE surveillance and testing have also been proven to be flawed, and
the GAO and OIG have both raised serious question as to just how flawed it has
been (see GAO and OIG reports). North America has more documented TSE prion
disease, in different documented species (excluding the Zoo BSE animals in the
EU), then any other place on the Globe. This does not include the very
likelihood that TSE prion disease in the domestic feline and canine have been
exposed to high doses of the TSE prion disease vid pet food. To date, it’s still
legal to include deer from cwd zone into pet food or deer food. Specified Risk
Material i.e. SRM bans still being breach, as recently as just last month.
nvCJD or what they now call vCJD, another case documented in Texas last
month, with very little information being released to the public on about this
case? with still the same line of thought from federal officials, ‘it can’t
happen here’, so another vCJD blamed on travel of a foreign animal disease from
another country, while ignoring all the BSE TSE Prion risk factors we have here
in the USA and Canada, and the time that this victim and others, do spend in the
USA, and exposed to these risk factors, apparently do not count in any way with
regard to risk factor. a flawed process of risk assessment.
sporadic CJD, along with new TSE prion disease in humans, of which the
young are dying, of which long duration of illness from onset of symptoms to
death have been documented, only to have a new name added to the pot of prion
disease i.e. sporadic GSS, sporadic FFI, and or VPSPR. I only ponder how a
familial type disease could be sporadic with no genetic link to any family
member? when the USA is the only documented Country in the world to have
documented two different cases of atypical H-type BSE, with one case being
called atypical H-G BSE with the G meaning Genetic, with new science now showing
that indeed atypical H-type BSE is very possible transmitted to cattle via oral
transmission (Prion2014). sporadic CJD and VPSPR have been rising in Canada,
USA, and the UK, with the same old excuse, better surveillance. You can only use
that excuse for so many years, for so many decades, until one must conclude that
CJD TSE prion cases are rising. a 48% incease in CJD in Canada is not just a
blip or a reason of better surveillance, it is a mathematical rise in numbers.
More and more we are seeing more humans exposed in various circumstance in the
Hospital, Medical, Surgical arenas to the TSE Prion disease, and at the same
time in North America, more and more humans are becoming exposed to the TSE
prion disease via consumption of the TSE prion via deer and elk, cattle, sheep
and goats, and for those that are exposed via or consumption, go on to further
expose many others via the iatrogenic modes of transmission of the TSE prion
disease i.e. friendly fire. I pondered this mode of transmission via the victims
of sporadic FFI, sporadic GSS, could this be a iatrogenic event from someone
sub-clinical with sFFI or sGSS ? what if?
Two decades have passed since Dr. Ironside first confirmed his first ten
nvCJD victims in 1995. Ten years later, 2005, we had Dr. Gambetti and his first
ten i.e. VPSPR in younger victims. now we know that indeed VPSPR is
transmissible. yet all these TSE prion disease and victims in the USA and Canada
are being pawned off as a spontaneous event, yet science has shown, the
spontaneous theory has never been proven in any natural case of TSE prion
disease, and scientist have warned, that they have now linked some sporadic CJD
cases to atypical BSE, to atypical Scrapie, and to CWD, yet we don’t here about
this in the public domain. We must make all human and animal TSE prion disease
reportable in every age group, in ever state and internationally, we must have a
serious re-evaluation and testing of the USA cattle herds, and we must ban
interstate movement of all cervids. Any voluntary effort to do any of this will
fail. Folks, we have let the industry run science far too long with regards to
the TSE prion disease. While the industry and their lobbyist continues to funnel
junk science to our decision policy makers, Rome burns. ...end
REFERENCES
Sunday, June 29, 2014
Transmissible Spongiform Encephalopathy TSE Prion Disease North America
2014
Tuesday, August 12, 2014
MAD COW USDA TSE PRION COVER UP or JUST IGNORANCE, for the record AUGUST
2014
Terry S. Singeltary Sr.
posted by Terry S. Singeltary Sr. at
7:25 PM
0 Comments:
Post a Comment
Subscribe to Post Comments [Atom]
<< Home