Texas Chronic Wasting Disease CWD TSE Prion 1,393 Confirmed Cases To Date July 2026
Texas Chronic Wasting Disease CWD TSE Prion 1,393 Confirmed Cases To Date July 2026
Positive Number CWD Positive Confirmation Date Free Range Captive County Facility Species Sex Age
1393 7/2/26 Breeder Deer Duval White-tailed Deer F 2.9
1392 7/2/26 Breeder Deer Duval White-tailed Deer M 2
1391 7/2/26 Breeder Deer Duval White-tailed Deer M 3.9
1390 7/2/206 Breeder Deer Zavala White-tailed Deer F 2
1389 7/2/26 Breeder Deer Zavala White-tailed Deer M 2.9
1388 7/15/26 Breeder Deer Gillespie White-tailed Deer F 2.9
1387 7/2/26 Breeder Deer Navarro White-tailed Deer F 2.9
1386 6/25/26 Breeder Deer Navarro White-tailed Deer F 2.8
1385 6/24/26 Breeder Deer Navarro White-tailed Deer F 5.8
1384 5/27/26 Breeder Deer Mason White-tailed Deer M 1.8
1383 5/27/26 Breeder Deer Mason White-tailed Deer M 3.8
1382 6/25/26 Free Range El Paso Mule Deer M 3.5
1381 6/25/26 Free Range El Paso Mule Deer F 3.5
1380 6/25/26 Free Range El Paso Mule Deer M 2.5
1379 5/27/26 Breeder Deer Duval White-tailed Deer M 3.8
1378 5/12/26 Breeder Deer Gillespie White-tailed Deer M 3.7
1377 4/27/26 Breeder Release Site Zavala White-tailed Deer F 7.5
1376 4/27/26 Breeder Deer Gillespie White-tailed Deer M 2.7
1375 4/27/26 Free Range Hartley White-tailed Deer M 3.5
1374 4/27/26 Free Range Hartley Mule Deer M 3.5
1373 4/27/26 Free Range Dallam Mule Deer M 3.5
1372 4/30/26 Breeder Deer Gillespie White-tailed Deer M 5.7
1371 4/30/26 Breeder Deer Gillespie White-tailed Deer M 5.7
1370 4/30/26 Breeder Deer Gillespie White-tailed Deer M 5.7
1369 4/22/26 Breeder Deer Zavala White-tailed Deer M 2.8
1368 4/22/26 Breeder Release Site Kaufman White-tailed Deer F 2.5
1367 4/21/26 Free Range Val Verde White-tailed Deer M 8.5
1366 4/17/26 Breeder Deer Cherokee White-tailed Deer F 0.8
1365 4/17/26 Breeder Deer Cherokee White-tailed Deer F 1.8
1364 4/21/26 Free Range El Paso Mule Deer M 5.5
1363 3/4/26 Free Range Dallam Mule Deer M 2.5
1362 4/15/26 Breeder Deer Real White-tailed Deer F 4.7
1361 4/15/26 Breeder Deer Real White-tailed Deer Unknown N/A
1360 4/15/26 Breeder Deer Real White-tailed Deer F 10.5
1359 4/15/26 Breeder Deer Real White-tailed Deer M 2.6
1358 4/15/26 Breeder Deer Real White-tailed Deer M 3.4
1357 4/15/26 Breeder Deer Real White-tailed Deer F 2.6
1356 4/15/26 Breeder Deer Real White-tailed Deer Unknown N/A
1355 4/15/26 Breeder Deer Real White-tailed Deer F 7.7
1354 4/15/26 Breeder Deer Real White-tailed Deer Unknown N/A
1353 4/15/26 Breeder Deer Real White-tailed Deer F 2.6
1352 4/15/26 Breeder Deer Real White-tailed Deer Unknown N/A
1351 4/15/26 Breeder Deer Real White-tailed Deer Unknown N/A
1350 4/15/26 Breeder Deer Real White-tailed Deer F 3.6
1349 4/15/26 Breeder Deer Real White-tailed Deer Unknown N/A
1348 5/7/26 Breeder Deer Real White-tailed Deer M 1.5
1347 5/7/26 Breeder Deer Real White-tailed Deer M 1.5
1346 5/7/26 Breeder Deer Real White-tailed Deer M 6.7
1345 5/7/26 Breeder Deer Real White-tailed Deer F 7.7
1344 5/7/26 Breeder Deer Real White-tailed Deer M 1.5
1343 5/7/26 Breeder Deer Real White-tailed Deer F 2.6
1342 5/7/26 Breeder Deer Real White-tailed Deer F N/A
1341 5/7/26 Breeder Deer Real White-tailed Deer M 2.6
1340 5/7/26 Breeder Deer Real White-tailed Deer M 1.5
1339 5/7/26 Breeder Deer Real White-tailed Deer M N/A
1338 5/7/26 Breeder Deer Real White-tailed Deer M 2.6
1337 5/7/26 Breeder Deer Real White-tailed Deer M 2.6
1336 5/7/26 Breeder Deer Real White-tailed Deer M 2.6
1335 5/7/26 Breeder Deer Real White-tailed Deer F 4.7
1334 5/7/26 Breeder Deer Real White-tailed Deer F 6.7
1333 5/7/26 Breeder Deer Real White-tailed Deer F 4.7
1332 5/7/26 Breeder Deer Real White-tailed Deer F 4.7
1331 5/7/26 Breeder Deer Real White-tailed Deer F 4.7
1330 5/7/26 Breeder Deer Real White-tailed Deer F 10.7
1329 5/7/26 Breeder Deer Real White-tailed Deer F 4.7
1328 5/7/26 Breeder Deer Real White-tailed Deer M 4.7
1327 5/7/26 Breeder Deer Real White-tailed Deer M 4.7
1326 5/7/26 Breeder Deer Real White-tailed Deer M 4.7
1325 5/7/26 Breeder Deer Real White-tailed Deer M 4.6
1324 5/7/26 Breeder Deer Real White-tailed Deer F 7.7
1323 5/7/26 Breeder Deer Real White-tailed Deer F 4.7
1322 5/7/26 Breeder Deer Real White-tailed Deer F 5.7
1321 5/7/26 Breeder Deer Real White-tailed Deer F 4.7
1320 5/7/26 Breeder Deer Real White-tailed Deer F 4.7
1319 5/7/26 Breeder Deer Real White-tailed Deer M 2.5
1318 5/7/26 Breeder Deer Real White-tailed Deer F 5.7
1317 5/7/26 Breeder Deer Real White-tailed Deer F 4.7
1316 5/7/26 Breeder Deer Real White-tailed Deer F N/A
1315 5/7/26 Breeder Deer Real White-tailed Deer F 2.6
1314 5/7/26 Breeder Deer Real White-tailed Deer F 2.6
1313 5/7/26 Breeder Deer Real White-tailed Deer F 2.6
1312 5/7/26 Breeder Deer Real White-tailed Deer F 4.7
1311 5/7/26 Breeder Deer Real White-tailed Deer F 4.6
1310 5/7/26 Breeder Deer Real White-tailed Deer M 2.5
1309 5/7/26 Breeder Deer Real White-tailed Deer M 2.5
1308 5/7/26 Breeder Deer Real White-tailed Deer F 4.7
1307 5/7/26 Breeder Deer Real White-tailed Deer F 4.7
1306 5/7/26 Breeder Deer Real White-tailed Deer F 4.7
1305 5/7/26 Breeder Deer Real White-tailed Deer F 4.6
1304 5/7/26 Breeder Deer Real White-tailed Deer F 4.6
1303 5/7/26 Breeder Deer Real White-tailed Deer F 5.7
1302 5/7/26 Breeder Deer Real White-tailed Deer M 1.5
1301 5/7/26 Breeder Deer Real White-tailed Deer F 4.7
1300 5/7/26 Breeder Deer Real White-tailed Deer M 3.7
1299 5/7/26 Breeder Deer Real White-tailed Deer M N/A
1298 5/7/26 Breeder Deer Real White-tailed Deer M 2.6
1297 5/7/26 Breeder Deer Real White-tailed Deer M N/A
1296 5/7/26 Breeder Deer Real White-tailed Deer M N/A
1295 5/7/26 Breeder Deer Real White-tailed Deer M N/A
1294 5/7/26 Breeder Deer Real White-tailed Deer M N/A
1293 5/7/26 Breeder Deer Real White-tailed Deer M 2.6
1292 5/7/26 Breeder Deer Real White-tailed Deer M N/A
1291 5/7/26 Breeder Deer Real White-tailed Deer M 2.6
1290 5/7/26 Breeder Deer Real White-tailed Deer F 3.5
1289 5/7/26 Breeder Deer Real White-tailed Deer M 2.6
1288 5/7/26 Breeder Deer Real White-tailed Deer M N/A
1287 5/7/26 Breeder Deer Real White-tailed Deer M N/A
1286 5/7/26 Breeder Deer Real White-tailed Deer M N/A
1285 5/7/26 Breeder Deer Real White-tailed Deer M N/A
1284 5/7/26 Breeder Deer Real White-tailed Deer M 3.7
1283 5/7/26 Breeder Deer Real White-tailed Deer M 3.6
1282 5/7/26 Breeder Deer Real White-tailed Deer M 3.5
1281 3/5/26 Breeder Deer Mason White-tailed Deer M 1.6
1280 3/3/26 Breeder Release Site Zavala White-tailed Deer M 3.5
1279 3/3/26 Breeder Release Site Zavala White-tailed Deer M 3.5
1278 3/3/26 Breeder Release Site Limestone White-tailed Deer N/A 2.5
1277 3/5/26 Breeder Deer Medina White-tailed Deer F 3.5
1276 3/3/26 Breeder Deer Gillespie White-tailed Deer M 3.5
1275 3/4/26 Breeder Deer Zavala White-tailed Deer M 1.5
1274 3/4/26 Breeder Deer Trinity White-tailed Deer F 10.5
1273 3/4/26 Breeder Deer Trinity White-tailed Deer F 2.6
1272 3/4/26 Free Range El Paso Mule Deer F 3.5
1271 2/19/26 Breeder Deer Duval White-tailed Deer M 2.4
1270 2/19/26 Breeder Deer Zavala White-tailed Deer M 3.5
1269 2/17/26 Breeder Deer Kaufman White-tailed Deer M 2.5
1268 2/19/26 Free Range Uvalde White-tailed Deer M 2.5
1267 2/17/26 Breeder Release Site Medina White-tailed Deer N/A 5
1266 2/17/26 Breeder Release Site Medina White-tailed Deer N/A 4
1265 2/17/26 Breeder Deer Kaufman White-tailed Deer M 2.5
1264 2/23/26 Breeder Release Site Gillespie White-tailed Deer N/A 2.5
1263 2/23/26 Breeder Release Site Medina White-tailed Deer N/A 6
1262 2/6/26 Breeder Release Site Medina White-tailed Deer N/A 7
1261 2/23/26 Breeder Deer Kaufman White-tailed Deer M 2.5
1260 2/23/26 Breeder Deer Kaufman White-tailed Deer M 2.5
1259 2/23/26 Breeder Deer Zavala White-tailed Deer M 3.4
1258 2/2/26 Breeder Deer Duval White-tailed Deer F 2.3
1257 2/2/26 Breeder Deer Duval White-tailed Deer F 2.3
1256 2/2/26 Breeder Deer Duval White-tailed Deer F 2.3
1255 2/2/26 Breeder Deer Duval White-tailed Deer F 2.3
1254 2/2/26 Breeder Deer Duval White-tailed Deer F 2.3
1253 2/2/26 Breeder Deer Duval White-tailed Deer F 2.3
1252 2/2/26 Breeder Deer Duval White-tailed Deer F 2.3
1251 2/2/26 Breeder Deer Duval White-tailed Deer F 2.3
1250 2/2/26 Breeder Deer Duval White-tailed Deer F 2.3
1249 2/2/26 Breeder Deer Duval White-tailed Deer F 2.3
1248 2/13/26 Breeder Release Site Duval White-tailed Deer F 3.5
1247 1/28/26 Breeder Release Site Kaufman White-tailed Deer M 4.5
1246 1/28/26 Breeder Release Site Hunt White-tailed Deer M 4.5
1245 1/28/26 Free Range Hudspeth Mule Deer M 5.5
1244 1/28/26 Free Range El Paso Mule Deer M 6.5
1243 1/6/26 Breeder Release Site Limestone White-tailed Deer N/A 4
1242 1/6/26 Breeder Release Site Limestone White-tailed Deer N/A 3
1241 1/6/26 Breeder Release Site Medina Red Deer F 4
1240 1/6/26 Breeder Release Site Medina Red Deer F 5
1239 1/28/26 Free Range Hartley Mule Deer M 2.5
1238 1/6/26 Breeder Deer Zavala White-tailed Deer N/A 4.4
1237 12/31/25 Breeder Release Site Limestone White-tailed Deer N/A 2.5
1236 12/31/25 Breeder Release Site Limestone White-tailed Deer N/A 2.5
1235 1/6/26 Breeder Release Site Limestone White-tailed Deer N/A 3.5
1234 1/6/26 Breeder Release Site Limestone White-tailed Deer N/A 3.5
1233 1/9/26 Breeder Deer Kimble White-tailed Deer M 5.4
1232 1/9/26 Breeder Deer Kimble White-tailed Deer F 2.4
1231 1/9/26 Breeder Deer Kimble White-tailed Deer F 4.4
1230 1/9/26 Breeder Deer Kimble White-tailed Deer F 2.4
1229 1/9/26 Breeder Deer Kimble White-tailed Deer F 4.4
1228 1/9/26 Breeder Deer Kimble White-tailed Deer F 6.4
1227 1/9/26 Breeder Deer Kimble White-tailed Deer F 2.4
1226 1/9/26 Breeder Deer Kimble White-tailed Deer F 7.4
1225 12/10/25 Breeder Deer Gillespie White-tailed Deer F 4.3
1224 12/15/25 Breeder Release Site Hunt White-tailed Deer M 2.5
1223 12/10/25 Free Range Medina White-tailed Deer M 1.5
1222 12/5/25 Breeder Release Site Kaufman White-tailed Deer M 2.3
1221 12/5/25 Breeder Release Site Kaufman White-tailed Deer M 6.5
1220 12/5/25 Breeder Release Site Kaufman White-tailed Deer M 2.5
1219 12/5/25 Free Range Hutchinson Mule Deer M 2.5
1218 11/18/25 Free Range Ochiltree Mule Deer M 6.5
1217 11/10/25 Breeder Release Site Kaufman White-tailed Deer M 4.5
1216 11/10/25 Free Range El Paso Mule Deer F 2.5
1215 10/27/25 Breeder Deer Tom Green White-tailed Deer F 5.3
1214 10/27/25 Breeder Deer Tom Green White-tailed Deer F 3
1213 11/4/25 Breeder Deer Frio White-tailed Deer M 3.2
1212 11/4/25 Breeder Deer Frio White-tailed Deer M 5.3
1211 11/4/25 Breeder Deer Navarro White-tailed Deer F 6.3
1210 10/31/25 Breeder Release Site Limestone White-tailed Deer F 2
1209 10/31/25 Breeder Release Site Limestone White-tailed Deer F 5
1208 11/4/25 Breeder Deer Frio White-tailed Deer M 2.3
1207 11/5/25 Breeder Deer Frio White-tailed Deer F 2.3
1206 11/5/25 Breeder Deer Frio White-tailed Deer F 2.3
1205 11/5/25 Breeder Deer Frio White-tailed Deer F 4.1
1204 11/5/25 Breeder Deer Frio White-tailed Deer F 9.3
1203 11/5/25 Breeder Deer Frio White-tailed Deer F 2.3
1202 11/5/25 Breeder Deer Frio White-tailed Deer F 4.3
1201 11/5/25 Breeder Deer Frio White-tailed Deer F 4.3
1200 10/31/25 Breeder Deer Frio White-tailed Deer F 2.3
1199 10/31/25 Breeder Release Site Kaufman White-tailed Deer M 2.3
1198 9/30/25 Breeder Deer Kerr White-tailed Deer M 4.1
1197 9/30/25 Breeder Deer Kerr White-tailed Deer F 1.1
1196 9/30/25 Breeder Deer Kerr White-tailed Deer F 1.1
1195 9/30/25 Breeder Deer Kerr White-tailed Deer F 1.1
1194 9/30/25 Breeder Deer Kerr White-tailed Deer M 1.1
1193 9/29/25 Breeder Deer Washington White-tailed Deer F 3.1
snip…see all;
Chronic Wasting Disease CWD TSE Prion Update July 2026
Texas CWD Update July 2026
Texas Agriculture Alert
Department Texas Parks and Wildlife
Created 06/09/2026
Disease / Pest Type Chronic Wasting Disease
Summary
A mule deer has been confirmed positive for Chronic Wasting Disease (CWD) in El Paso County. Due to the number and distribution of CWD detections in free-ranging deer in this county, additional information regarding CWD detections will only be posted to the website linked above from this time forward.
Additional Information
https://tpwd.texas.gov/huntwild/wild/diseases/cwd/
https://agalert.tamu.edu/details/2/6
Texas Agriculture Alert
Department
Texas Parks and Wildlife
Created 05/21/2026
Disease / Pest Type Chronic Wasting Disease
Summary
A white-tailed deer has been confirmed positive for Chronic Wasting Disease (CWD) in Cherokee County. The property where this CWD positive deer was located has not previously had a CWD detection. Visit the link provided for additional information regarding CWD surveillance and management in Texas.
Additional Information
https://tpwd.texas.gov/huntwild/wild/diseases/cwd/
https://agalert.tamu.edu/details/2/3
Texas Agriculture Alert
Department
Texas Parks and Wildlife
Created 05/21/2026
Disease / Pest Type Chronic Wasting Disease
Summary
A white-tailed deer has been confirmed positive for Chronic Wasting Disease (CWD) in Zavala County. The property where this CWD positive deer was located has not previously had a CWD detection. Visit the link provided for additional information regarding CWD surveillance and management in Texas.
Additional Information
https://tpwd.texas.gov/huntwild/wild/diseases/cwd/
https://agalert.tamu.edu/details/2/4
SEE NEW DASHBOARD FOR CWD POSITIVES!
https://experience.arcgis.com/experience/8f6c27330c444a19b4b57beb7ffabb8b/page/Dashboard#data_s=id%3AdataSource_3-1966d773e34-layer-10%3A29
Texas CWD total by calendar years
https://tpwd.texas.gov/huntwild/wild/diseases/cwd/positive-cases/listing-cwd-cases-texas.phtml#texasCWD
Counties where CWD Exposed Deer were Released
https://tpwd.texas.gov/documents/257/CWD-Trace-OutReleaseSites.pdf
Number of CWD Exposed Deer Released by County
https://tpwd.texas.gov/documents/258/CWD-Trace-OutReleaseSites-NbrDeer.pdf
***> TRUCKING CWD TSE PRION
“CWD spreads among wild populations at a relatively slow rate, limited by the natural home range and dispersed nature of wild animals.”
***> NOW HOLD YOUR HORSES, Chronic Wasting Disease CWD of Cervid can spread rather swiftly, traveling around 50 MPH, from the back of truck and trailer, and Here in Texas, we call it ‘Trucking CWD’…
Preventive Veterinary Medicine Volume 234, January 2025, 106385
Use of biosecurity practices to prevent chronic wasting disease in Minnesota cervid herds
Vehicles or trailers that entered the farm were used to transport other live cervids, cervid carcasses, or cervid body parts in past 3 years in 64.3 % (95 % CI 46.3–82.3) of larger elk/reindeer herds compared to 13.6 % (95 % CI 4.7–22.4) of smaller deer herds.
Snip…
Identifying the exact pathway of initial CWD transmission to cervid herds is often not possible, in part due to many potential pathways of transmission for the infection, including both direct and indirect contact with infected farmed or wild cervids (Kincheloe et al., 2021). That study identified that transmissions from infected farmed cervids may occur from direct contact with the movement of cervids from one herd to another and from indirect contact with the sharing of equipment, vehicles, clothing, reproductive equipment, and potentially through semen or embryos.
https://www.sciencedirect.com/science/article/abs/pii/S016758772400271X
“Chronic Wasting Disease (CWD) is a fatal neurological disease and can devastate deer populations by silently spreading through direct animal contact and contaminated environments. Without close monitoring, illegal movement of captive deer increases the risk of introducing CWD to areas it is not known to exist, potentially leading to widespread outbreaks which will impact more than just the health of Texas deer.”
https://tpwd.texas.gov/newsmedia/releases/?req=20250227b
THURSDAY, AUGUST 14, 2025
Texas Game Wardens Near Conclusion of ‘Ghost Deer’ Case with 24 Suspects, 1,400 Charges Filed Statewide
https://chronic-wasting-disease.blogspot.com/2025/08/texas-game-wardens-near-conclusion-of.html
https://prpsc.proboards.com/thread/178/texas-game-wardens-conclusion-ghost
Texas CWD TSE Prion Cases Climb To 1,377 Cases Detected to date May 19, 2026
https://chronic-wasting-disease.blogspot.com/2026/05/texas-cwd-tse-prion-cases-climb-to-1377.html
https://prpsc.proboards.com/thread/218/texas-cases-detected-date-2026
MONDAY, MAY 05, 2026
TEXAS ANIMAL HEALTH COMMISSION AGENCY STRATEGIC PLAN FISCAL YEARS 2025 - 2029 CWD TSE Prion
https://chronic-wasting-disease.blogspot.com/2026/05/texas-animal-health-commission-agency.html
https://prpsc.proboards.com/thread/216/animal-health-commission-agency-strategic
Chronic Wasting Disease CWD TSE Prion, Economical, Environmental, Zoonotic, Risk Factors 2026
https://transmissiblespongiformencephalopathy.blogspot.com/2026/02/chronic-wasting-disease-cwd-tse-prion.html
Cervid CWD TSE PrP Transmission to Humans, has it already happened and being masked as sporadic CJD, What if? July 2026 Review
https://chronic-wasting-disease.blogspot.com/2026/07/cervid-cwd-tse-prp-transmission-to.html
https://prpsc.proboards.com/thread/224/cervid-transmission-humans-july-review
***> USA Report, Scrapie, CWD, BSE, TSE, Cattle, Sheep, Pigs, Cervid, Humans, Zoonotic, 2026
April 2026
https://fdabse589.blogspot.com/2026/04/usa-report-scrapie-cwd-bse-tse-cattle.html
https://www.researchgate.net/publication/403956772_USA_Report_Scrapie_CWD_BSE_TSE_Cattle_Sheep_Pigs_Cervid_Humans_Zoonotic_2026
Tuesday, June 9, 2026
Hazard identification for the risks to terrestrial animal health from the import of hay and straw, BSE, CWD, Scrapie, TSE, Prion
https://www.gov.uk/government/publications/hazard-identification-for-the-risks-to-terrestrial-animal-health-from-the-import-of-hay-and-straw/hazard-identification-for-the-risks-to-terrestrial-animal-health-from-the-import-of-hay-and-straw
https://defrauktseprp.blogspot.com/2026/06/hazard-identification-for-risks-to.html
“They analysed over 300 brain and lymph node samples from 178 wild pigs living across Arkansas and Texas, USA…the researchers identified CWD prions in up to 37% of the lymph node samples and 15% of brain samples. The lowest detection rates were in the Texas samples (below 16%), matching the lower CWD prevalence in the state’s cervid population.”
Chronic wasting disease prions in cervids and wild pigs in North America Preliminary Outbreak Assessment DEFRA 26 January 2026 Department for Environment, Food and Rural Affairs
Preliminary Outbreak Assessment
Chronic wasting disease prions in cervids and wild pigs in North America
26 January 2026
Disease report
Chronic wasting disease (CWD) is a fatal neurodegenerative disease of cervids, such as deer, elk, moose and reindeer. It is caused by prions – infectious proteins that cause normal cellular prion proteins to misfold (CIDRAP, 2025). The disease is widespread in captive and free-ranging cervids in North America (Figure 1). For the first time, CWD prions have also been detected in the tissues of wild pigs (Sus scrofa) caught in CWD-affected areas of the USA (Soto et al. 2025). This discovery emerged from a study designed to investigate potential interactions between wild pigs and CWD prions, as wild pigs often coexist with cervids, which can shed prions into the environment. The following assessment discusses the epidemiology of CWD in North America and the detection of CWD prions in wild pigs. It also considers the potential implications for Great Britain.
snip…
While the spread of CWD across North America is often described as ‘rapid,’ it has been suggested that this may reflect widening disease surveillance, rather than a ‘real-time’ indication of geographic spread. CWD epidemics appear to develop relatively slowly compared with other wildlife diseases (EFSA BIOHAZ Panel, 2023). Field and modelling data from North America suggest that it may take 15 to 20 years for CWD prevalence to reach 1% in free-ranging cervid populations, although more rapid transmission may occur in captive populations. The surveillance sensitivity in North America means that the disease may have been present for 10 years or more in some areas before it was detected (Miller et al., 2000).
The prevalence of CWD in affected populations or species varies across North America. In captive herds, prevalence may reach 100% over time, while in affected free-ranging populations, reported prevalence ranges from <1% to >30%. Most clinical cases are observed in cervids 2 to 7 years old, especially males, which is believed to be due to behavioural differences rather than differences in susceptibility between sexes (EFSA BIOHAZ Panel, 2023). At least 13 different risk factors may contribute towards CWD spread in North America, such as host genetics, high deer density or inappropriate disposal of deer carcasses and slaughter by-products (EFSA BIOHAZ Panel, 2019).
Approaches towards CWD control and surveillance in captive and free-ranging deer vary widely across North America within and between jurisdictions (CIDRAP, 2025). A summary of the measures in place in each US state and Canadian province is available from the CWD Alliance (2026), a coalition of wildlife conservation agencies, dedicated to providing accurate information on CWD and supporting strategies to minimise its impact on free-ranging cervids. Wildlife agencies rely on voluntary testing of hunted deer carcasses as the main mechanism for CWD surveillance and management, usually using post-mortem ELISA or immunohistochemistry methods (CIDRAP, 2025).
In the USA, Animal and Plant Health Inspection Service (APHIS) operates the CWD Herd Certification Programme (HCP) in collaboration with state and wildlife agencies. This is a voluntary scheme which aims to provide a consistent, national approach to controlling CWD in farmed cervids and preventing interstate spread by establishing control measures such as fencing, detailed record keeping and CWD testing of all cervids over 12 months old that die for any reason. The Canadian Food Inspection Agency (CFIA) operates a similar programme, the CWD Herd Certification Programme. As of December 2025, 28 states were participating in the USA’s CWD HCP and 5 Canadian provinces and one Canadian territory were participating in the Canadian programme (USDA, 2025b, CFIA, 2025).
Control methods fall within three general categories: prevention, containment, and control and suppression. Prevention and containment aim to prevent CWD
Department for Environment, Food and Rural Affairs
introduction into areas where it has not previously been reported and to limit its geographical spread once it has been introduced, respectively. Both tend to include regulatory measures such as bans on the movement of live cervids, cervid carcasses or specified risk materials. Control and suppression aim to stabilise or reduce infection rates within a herd or population through measures such as selective or random culling (EFSA BIOHAZ Panel, 2017).
Despite control efforts, CWD has continued to spread among captive and free- ranging cervids in North America, with increasing prevalence in affected areas (Uehlinger et al., 2016, CFSPH, 2024). Eradicating CWD from North America appears infeasible due to its extent of geographic spread and epidemiological characteristics, such as environmental persistence (EFSA BIOHAZ Panel, 2017).
CWD in wild pigs in the USA
Wild pigs are an invasive population in the USA, especially in the south (Figure 2). They comprise escaped domestic swine, Eurasian wild boar and hybrids of the two (Smyser et al., 2020). Wild pigs frequently coexist with cervids in areas where CWD is endemic and may be exposed to CWD prions through rooting in contaminated soil, scavenging deer carcasses and predation on fawns. These ecological interactions provide multiple routes by which wild pigs could encounter prions from infected deer (Soto et al. 2025).
Under experimental conditions, domestic pigs can become infected with CWD by oral and intracerebral routes, suggesting that wild pigs might also be susceptible. Domestic pigs rarely develop clinical signs of CWD but accumulate prions in the lymphoid tissues in their heads and gut, suggesting that, like cervids, they could shed the prions in saliva and faeces (Moore et al., 2017).
Against this background, Soto et al. (2025) investigated potential interactions between wild pigs and CWD prions. They analysed over 300 brain and lymph node samples from 178 wild pigs living across Arkansas and Texas, USA. The animals were captured by the United States Department of Agriculture (USDA) between 2020 and 2021. None of the pigs included in the study were reported to be displaying clinical signs of disease.
Using an ultra-sensitive laboratory method (protein misfolding cyclic amplification (PMCA)), the researchers identified CWD prions in up to 37% of the lymph node samples and 15% of brain samples. The lowest detection rates were in the Texas samples (below 16%), matching the lower CWD prevalence in the state’s cervid population. These findings indicate that wild pigs are naturally exposed to CWD prions in areas where the disease is present (Soto et al., 2025).
When intracerebrally inoculated with tissues from wild pigs, a small proportion of mice expressing deer prion protein developed subclinical prion infection. No transmission was detected in mice expressing pig prion protein. This suggests that wild pig tissues only contain low levels of infectious prions and that wild pigs are relatively resistant to natural infection. However, they could still contribute to CWD transmission, influencing its epidemiology, geographic distribution and interspecies spread (Soto et al., 2025).
Department for Environment, Food and Rural Affairs
While their exact role and importance in CWD transmission is unclear, wild pigs have considerable home ranges in North America (1.1 to 5.32 km on average), which may increase when food is scarce. This mobility could complicate efforts to control the disease if they play a role in its transmission (Soto et al., 2025).
The USDA’s APHIS does not currently conduct active surveillance for CWD in wild pigs (USDA, 2025a).
Figure 2. Geographic distribution of wild pigs (purple) in the USA as of 27 January 2025, comprising escaped domestic pigs, Eurasian wild boar and hybrids of the two (adapted from USDA, 2026). Yellow (Texas) and green (Arkansas) circles indicate the states where CWD prions were detected in wild pig tissues.
snip…
Conclusion
CWD has continued to spread among captive and free-ranging cervids in North America since it was first detected in the 1960s. The finding of CWD prions in wild pigs in the USA suggests they could contribute towards transmission of the disease, influencing its epidemiology, geographic distribution and interspecies spread. However, further research is needed to confirm this. CWD has never been reported in Great Britain and the current risk of CWD prions being introduced into Great Britain’s wild pig or cervid population ranges from very low to negligible.
Readers are reminded to be vigilant for signs of CWD. Information on how to spot the disease can be found here. Suspected cases must be reported immediately to the Defra Rural Services Helpline on 03000 200 301. In Wales, call 0300 303 8268. In Scotland, contact your local Field Services Office. Failure to do so is an offence. We will continue to monitor the situation.
Authors • Lawrence Finn • Dr. Lauren Perrin • John Spiropoulos • Dr. Helen RobertsDepartment for Environment, Food and Rural Affairs
References
snip…see;
https://assets.publishing.service.gov.uk/media/697a3b013c71d838df6bd413/CWD_Prions_in_Cervids_and_Wild_Pigs_in_North_America.pdf
“They analysed over 300 brain and lymph node samples from 178 wild pigs living across Arkansas and Texas, USA…the researchers identified CWD prions in up to 37% of the lymph node samples and 15% of brain samples. The lowest detection rates were in the Texas samples (below 16%), matching the lower CWD prevalence in the state’s cervid population.”
Detection of Prions in Wild Pigs (Sus scrofa) from Areas with Reported Chronic Wasting Disease Cases, United State Volume 31, Number 1—January 2025
https://wwwnc.cdc.gov/eid/article/31/1/24-0401_article
Camel Prion Disease, Tataouine, Tunisia, 2019–2021 CDC Dispatch August 2026
Volume 32, Number 8—August 2026
Dispatch
Camel Prion Disease, Tataouine, Tunisia, 2019–2021
We report 6 cases of camel prion disease in dromedaries in Tunisia, confirming widespread occurrence in North Africa.
The recent detection of sheep scrapie in the same area of Tunisia as CPrD (15) raises the question of possible links, although no similarities have yet been shown.
https://wwwnc.cdc.gov/eid/article/32/8/25-1474_article
snip…
The detection of PrPSc in all available lymph nodes, together with the relatively young age of some camels, supports the hypothesis that CPrD is a transmissible prion disease. As in scrapie and chronic wasting disease, lymphoid involvement may enable extraneural propagation and serve as a source of environmental contamination (2–5), raising concerns about prion shedding and persistence.
snip…
Taken together, the data from Algeria and Tunisia suggest that CPrD may be endemic in certain areas. Despite the limited number of reported cases so far, the absence of active surveillance programs in those countries, unlike in Europe for scrapie and bovine spongiform encephalopathy (BSE), raises the possibility of a higher number of undetected cases. Given frequent cross-border movements and the extensive pastoral systems in which camels roam widely, contact between herds in Tunisia and herds in Algeria is frequent and has possible implications for disease circulation.
CPrD may substantially affect camel health and production, given that dromedaries are long-lived animals with extended reproductive and productive roles and are vital to pastoralist communities in arid regions. Its detection in areas where camels are central to food production underscores the need to understand its epidemiology, transmission, and long-term impact.
The emergence of a new animal prion disease also raises public health concerns. Although only BSE has shown zoonotic potential, its history highlights the necessity of monitoring all animal prion diseases. Long incubation and absence of early clinical markers complicate risk assessment, as exemplified by the decade-long delay between cattle BSE and the variant Creutzfeldt–Jakob disease epidemic in humans (2).
Given the potential implications for animal and human health and the possible socioeconomic impact in regions where dromedaries play a key role, ongoing surveillance and in-depth characterization of CPrD are essential to enhance our comprehension of its epidemiology, host range, and zoonotic potential.
Dr. Amara is emeritus professor of histology and pathological anatomy at the National School of Veterinary Medicine of Sidi Thabet in Sidi Thabet, Tunisia. He is also vice president of the Expert Committee on Animal Health at the National Center for Veterinary Surveillance in Tunisia. His primary research interests include prion diseases in ruminants and dromedaries.
https://wwwnc.cdc.gov/eid/article/32/8/25-1474_article
Identification of Camel Prion Disease in Tunisia: evidence of an emerging prion disease in North Africa
https://proceedings.science/prion-2025/papers?prod_proceedings_papers%5BrefinementList%5D%5Btrack.title.en%5D%5B0%5D=Animal%20prion%20diseases
https://camelusprp.blogspot.com/2026/02/
https://camelusprp.blogspot.com/2026/
Texas Creutzfeldt Jakob Disease CJD TSE Prion Disease 2025
Update as of 10/15/25, NO REPORT TO UPDATE…
https://cjdtexas.blogspot.com/2026/03/texas-creutzfeldt-jakob-disease-deaths.html
https://www.dshs.texas.gov/creutzfeldt-jakob-disease-cjd/creutzfeldt-jakob-disease-cjd-data
US NATIONAL PRION DISEASE PATHOLOGY SURVEILLANCE CENTER CJD TSE REPORT SEPTEMBER 2025
From the first full year of reporting CJD TSE in the US in 2000, where 90 cases of CJD was reported that year, to today, where in September 2025, the number of CJD cases reported in the last full year reporting, which would have been 2024, the number of CJD cases for 2024 was 249 cases. So, from the first full year 2000 CJD cases were 90 cases confirmed in that year, to 2024, where 2024 CJD statistics rose to 249 confirmed CJD cases in a single year. A dramatic increase in deaths, from figures that don’t seem to be dramatic. But thes figures today, they are not from “better surveillance”, that dog don’t hunt no more. They have been saying this for over 25 years, year after year, well it’s time to call it for what it is, Human Transmissible Spongiform Encephalopathy TSE Prion cases are rising, and it’s NOT because of better surveillance, or just a “happenstance of bad luck, that 85%+ of all human cases, sporadic CJD, including VPSPr, just happen spontaneously, no, it’s because of unknown environmental factors, and or iatrogenic factors, imho…terry
US NATIONAL PRION DISEASE PATHOLOGY SURVEILLANCE CENTER CJD TSE REPORT SEPTEMBER 2025
https://prionunitusaupdate.blogspot.com/2025/10/us-national-prion-disease-pathology.html
So, this is what we leave our children and grandchildren?
Terry S. Singeltary Sr.
Texas CWD Update July 2026
Texas Agriculture Alert
Department Texas Parks and Wildlife
Created 06/09/2026
Disease / Pest Type Chronic Wasting Disease
Summary
A mule deer has been confirmed positive for Chronic Wasting Disease (CWD) in El Paso County. Due to the number and distribution of CWD detections in free-ranging deer in this county, additional information regarding CWD detections will only be posted to the website linked above from this time forward.
Additional Information
https://tpwd.texas.gov/huntwild/wild/diseases/cwd/
https://agalert.tamu.edu/details/2/6
Texas Agriculture Alert
Department
Texas Parks and Wildlife
Created 05/21/2026
Disease / Pest Type Chronic Wasting Disease
Summary
A white-tailed deer has been confirmed positive for Chronic Wasting Disease (CWD) in Cherokee County. The property where this CWD positive deer was located has not previously had a CWD detection. Visit the link provided for additional information regarding CWD surveillance and management in Texas.
Additional Information
https://tpwd.texas.gov/huntwild/wild/diseases/cwd/
https://agalert.tamu.edu/details/2/3
Texas Agriculture Alert
Department
Texas Parks and Wildlife
Created 05/21/2026
Disease / Pest Type Chronic Wasting Disease
Summary
A white-tailed deer has been confirmed positive for Chronic Wasting Disease (CWD) in Zavala County. The property where this CWD positive deer was located has not previously had a CWD detection. Visit the link provided for additional information regarding CWD surveillance and management in Texas.
Additional Information
https://tpwd.texas.gov/huntwild/wild/diseases/cwd/
https://agalert.tamu.edu/details/2/4
SEE NEW DASHBOARD FOR CWD POSITIVES!
https://experience.arcgis.com/experience/8f6c27330c444a19b4b57beb7ffabb8b/page/Dashboard#data_s=id%3AdataSource_3-1966d773e34-layer-10%3A29
Texas CWD total by calendar years
https://tpwd.texas.gov/huntwild/wild/diseases/cwd/positive-cases/listing-cwd-cases-texas.phtml#texasCWD
Counties where CWD Exposed Deer were Released
https://tpwd.texas.gov/documents/257/CWD-Trace-OutReleaseSites.pdf
Number of CWD Exposed Deer Released by County
https://tpwd.texas.gov/documents/258/CWD-Trace-OutReleaseSites-NbrDeer.pdf
***> TRUCKING CWD TSE PRION
“CWD spreads among wild populations at a relatively slow rate, limited by the natural home range and dispersed nature of wild animals.”
***> NOW HOLD YOUR HORSES, Chronic Wasting Disease CWD of Cervid can spread rather swiftly, traveling around 50 MPH, from the back of truck and trailer, and Here in Texas, we call it ‘Trucking CWD’…
Preventive Veterinary Medicine Volume 234, January 2025, 106385
Use of biosecurity practices to prevent chronic wasting disease in Minnesota cervid herds
Vehicles or trailers that entered the farm were used to transport other live cervids, cervid carcasses, or cervid body parts in past 3 years in 64.3 % (95 % CI 46.3–82.3) of larger elk/reindeer herds compared to 13.6 % (95 % CI 4.7–22.4) of smaller deer herds.
Snip…
Identifying the exact pathway of initial CWD transmission to cervid herds is often not possible, in part due to many potential pathways of transmission for the infection, including both direct and indirect contact with infected farmed or wild cervids (Kincheloe et al., 2021). That study identified that transmissions from infected farmed cervids may occur from direct contact with the movement of cervids from one herd to another and from indirect contact with the sharing of equipment, vehicles, clothing, reproductive equipment, and potentially through semen or embryos.
https://www.sciencedirect.com/science/article/abs/pii/S016758772400271X
“Chronic Wasting Disease (CWD) is a fatal neurological disease and can devastate deer populations by silently spreading through direct animal contact and contaminated environments. Without close monitoring, illegal movement of captive deer increases the risk of introducing CWD to areas it is not known to exist, potentially leading to widespread outbreaks which will impact more than just the health of Texas deer.”
https://tpwd.texas.gov/newsmedia/releases/?req=20250227b
THURSDAY, AUGUST 14, 2025
Texas Game Wardens Near Conclusion of ‘Ghost Deer’ Case with 24 Suspects, 1,400 Charges Filed Statewide
https://chronic-wasting-disease.blogspot.com/2025/08/texas-game-wardens-near-conclusion-of.html
https://prpsc.proboards.com/thread/178/texas-game-wardens-conclusion-ghost
Texas CWD TSE Prion Cases Climb To 1,377 Cases Detected to date May 19, 2026
https://chronic-wasting-disease.blogspot.com/2026/05/texas-cwd-tse-prion-cases-climb-to-1377.html
https://prpsc.proboards.com/thread/218/texas-cases-detected-date-2026
MONDAY, MAY 05, 2026
TEXAS ANIMAL HEALTH COMMISSION AGENCY STRATEGIC PLAN FISCAL YEARS 2025 - 2029 CWD TSE Prion
https://chronic-wasting-disease.blogspot.com/2026/05/texas-animal-health-commission-agency.html
https://prpsc.proboards.com/thread/216/animal-health-commission-agency-strategic
Chronic Wasting Disease CWD TSE Prion, Economical, Environmental, Zoonotic, Risk Factors 2026
https://transmissiblespongiformencephalopathy.blogspot.com/2026/02/chronic-wasting-disease-cwd-tse-prion.html
Cervid CWD TSE PrP Transmission to Humans, has it already happened and being masked as sporadic CJD, What if? July 2026 Review
https://chronic-wasting-disease.blogspot.com/2026/07/cervid-cwd-tse-prp-transmission-to.html
https://prpsc.proboards.com/thread/224/cervid-transmission-humans-july-review
***> USA Report, Scrapie, CWD, BSE, TSE, Cattle, Sheep, Pigs, Cervid, Humans, Zoonotic, 2026
April 2026
https://fdabse589.blogspot.com/2026/04/usa-report-scrapie-cwd-bse-tse-cattle.html
https://www.researchgate.net/publication/403956772_USA_Report_Scrapie_CWD_BSE_TSE_Cattle_Sheep_Pigs_Cervid_Humans_Zoonotic_2026
Tuesday, June 9, 2026
Hazard identification for the risks to terrestrial animal health from the import of hay and straw, BSE, CWD, Scrapie, TSE, Prion
https://www.gov.uk/government/publications/hazard-identification-for-the-risks-to-terrestrial-animal-health-from-the-import-of-hay-and-straw/hazard-identification-for-the-risks-to-terrestrial-animal-health-from-the-import-of-hay-and-straw
https://defrauktseprp.blogspot.com/2026/06/hazard-identification-for-risks-to.html
“They analysed over 300 brain and lymph node samples from 178 wild pigs living across Arkansas and Texas, USA…the researchers identified CWD prions in up to 37% of the lymph node samples and 15% of brain samples. The lowest detection rates were in the Texas samples (below 16%), matching the lower CWD prevalence in the state’s cervid population.”
Chronic wasting disease prions in cervids and wild pigs in North America Preliminary Outbreak Assessment DEFRA 26 January 2026 Department for Environment, Food and Rural Affairs
Preliminary Outbreak Assessment
Chronic wasting disease prions in cervids and wild pigs in North America
26 January 2026
Disease report
Chronic wasting disease (CWD) is a fatal neurodegenerative disease of cervids, such as deer, elk, moose and reindeer. It is caused by prions – infectious proteins that cause normal cellular prion proteins to misfold (CIDRAP, 2025). The disease is widespread in captive and free-ranging cervids in North America (Figure 1). For the first time, CWD prions have also been detected in the tissues of wild pigs (Sus scrofa) caught in CWD-affected areas of the USA (Soto et al. 2025). This discovery emerged from a study designed to investigate potential interactions between wild pigs and CWD prions, as wild pigs often coexist with cervids, which can shed prions into the environment. The following assessment discusses the epidemiology of CWD in North America and the detection of CWD prions in wild pigs. It also considers the potential implications for Great Britain.
snip…
While the spread of CWD across North America is often described as ‘rapid,’ it has been suggested that this may reflect widening disease surveillance, rather than a ‘real-time’ indication of geographic spread. CWD epidemics appear to develop relatively slowly compared with other wildlife diseases (EFSA BIOHAZ Panel, 2023). Field and modelling data from North America suggest that it may take 15 to 20 years for CWD prevalence to reach 1% in free-ranging cervid populations, although more rapid transmission may occur in captive populations. The surveillance sensitivity in North America means that the disease may have been present for 10 years or more in some areas before it was detected (Miller et al., 2000).
The prevalence of CWD in affected populations or species varies across North America. In captive herds, prevalence may reach 100% over time, while in affected free-ranging populations, reported prevalence ranges from <1% to >30%. Most clinical cases are observed in cervids 2 to 7 years old, especially males, which is believed to be due to behavioural differences rather than differences in susceptibility between sexes (EFSA BIOHAZ Panel, 2023). At least 13 different risk factors may contribute towards CWD spread in North America, such as host genetics, high deer density or inappropriate disposal of deer carcasses and slaughter by-products (EFSA BIOHAZ Panel, 2019).
Approaches towards CWD control and surveillance in captive and free-ranging deer vary widely across North America within and between jurisdictions (CIDRAP, 2025). A summary of the measures in place in each US state and Canadian province is available from the CWD Alliance (2026), a coalition of wildlife conservation agencies, dedicated to providing accurate information on CWD and supporting strategies to minimise its impact on free-ranging cervids. Wildlife agencies rely on voluntary testing of hunted deer carcasses as the main mechanism for CWD surveillance and management, usually using post-mortem ELISA or immunohistochemistry methods (CIDRAP, 2025).
In the USA, Animal and Plant Health Inspection Service (APHIS) operates the CWD Herd Certification Programme (HCP) in collaboration with state and wildlife agencies. This is a voluntary scheme which aims to provide a consistent, national approach to controlling CWD in farmed cervids and preventing interstate spread by establishing control measures such as fencing, detailed record keeping and CWD testing of all cervids over 12 months old that die for any reason. The Canadian Food Inspection Agency (CFIA) operates a similar programme, the CWD Herd Certification Programme. As of December 2025, 28 states were participating in the USA’s CWD HCP and 5 Canadian provinces and one Canadian territory were participating in the Canadian programme (USDA, 2025b, CFIA, 2025).
Control methods fall within three general categories: prevention, containment, and control and suppression. Prevention and containment aim to prevent CWD
Department for Environment, Food and Rural Affairs
introduction into areas where it has not previously been reported and to limit its geographical spread once it has been introduced, respectively. Both tend to include regulatory measures such as bans on the movement of live cervids, cervid carcasses or specified risk materials. Control and suppression aim to stabilise or reduce infection rates within a herd or population through measures such as selective or random culling (EFSA BIOHAZ Panel, 2017).
Despite control efforts, CWD has continued to spread among captive and free- ranging cervids in North America, with increasing prevalence in affected areas (Uehlinger et al., 2016, CFSPH, 2024). Eradicating CWD from North America appears infeasible due to its extent of geographic spread and epidemiological characteristics, such as environmental persistence (EFSA BIOHAZ Panel, 2017).
CWD in wild pigs in the USA
Wild pigs are an invasive population in the USA, especially in the south (Figure 2). They comprise escaped domestic swine, Eurasian wild boar and hybrids of the two (Smyser et al., 2020). Wild pigs frequently coexist with cervids in areas where CWD is endemic and may be exposed to CWD prions through rooting in contaminated soil, scavenging deer carcasses and predation on fawns. These ecological interactions provide multiple routes by which wild pigs could encounter prions from infected deer (Soto et al. 2025).
Under experimental conditions, domestic pigs can become infected with CWD by oral and intracerebral routes, suggesting that wild pigs might also be susceptible. Domestic pigs rarely develop clinical signs of CWD but accumulate prions in the lymphoid tissues in their heads and gut, suggesting that, like cervids, they could shed the prions in saliva and faeces (Moore et al., 2017).
Against this background, Soto et al. (2025) investigated potential interactions between wild pigs and CWD prions. They analysed over 300 brain and lymph node samples from 178 wild pigs living across Arkansas and Texas, USA. The animals were captured by the United States Department of Agriculture (USDA) between 2020 and 2021. None of the pigs included in the study were reported to be displaying clinical signs of disease.
Using an ultra-sensitive laboratory method (protein misfolding cyclic amplification (PMCA)), the researchers identified CWD prions in up to 37% of the lymph node samples and 15% of brain samples. The lowest detection rates were in the Texas samples (below 16%), matching the lower CWD prevalence in the state’s cervid population. These findings indicate that wild pigs are naturally exposed to CWD prions in areas where the disease is present (Soto et al., 2025).
When intracerebrally inoculated with tissues from wild pigs, a small proportion of mice expressing deer prion protein developed subclinical prion infection. No transmission was detected in mice expressing pig prion protein. This suggests that wild pig tissues only contain low levels of infectious prions and that wild pigs are relatively resistant to natural infection. However, they could still contribute to CWD transmission, influencing its epidemiology, geographic distribution and interspecies spread (Soto et al., 2025).
Department for Environment, Food and Rural Affairs
While their exact role and importance in CWD transmission is unclear, wild pigs have considerable home ranges in North America (1.1 to 5.32 km on average), which may increase when food is scarce. This mobility could complicate efforts to control the disease if they play a role in its transmission (Soto et al., 2025).
The USDA’s APHIS does not currently conduct active surveillance for CWD in wild pigs (USDA, 2025a).
Figure 2. Geographic distribution of wild pigs (purple) in the USA as of 27 January 2025, comprising escaped domestic pigs, Eurasian wild boar and hybrids of the two (adapted from USDA, 2026). Yellow (Texas) and green (Arkansas) circles indicate the states where CWD prions were detected in wild pig tissues.
snip…
Conclusion
CWD has continued to spread among captive and free-ranging cervids in North America since it was first detected in the 1960s. The finding of CWD prions in wild pigs in the USA suggests they could contribute towards transmission of the disease, influencing its epidemiology, geographic distribution and interspecies spread. However, further research is needed to confirm this. CWD has never been reported in Great Britain and the current risk of CWD prions being introduced into Great Britain’s wild pig or cervid population ranges from very low to negligible.
Readers are reminded to be vigilant for signs of CWD. Information on how to spot the disease can be found here. Suspected cases must be reported immediately to the Defra Rural Services Helpline on 03000 200 301. In Wales, call 0300 303 8268. In Scotland, contact your local Field Services Office. Failure to do so is an offence. We will continue to monitor the situation.
Authors • Lawrence Finn • Dr. Lauren Perrin • John Spiropoulos • Dr. Helen RobertsDepartment for Environment, Food and Rural Affairs
References
snip…see;
https://assets.publishing.service.gov.uk/media/697a3b013c71d838df6bd413/CWD_Prions_in_Cervids_and_Wild_Pigs_in_North_America.pdf
“They analysed over 300 brain and lymph node samples from 178 wild pigs living across Arkansas and Texas, USA…the researchers identified CWD prions in up to 37% of the lymph node samples and 15% of brain samples. The lowest detection rates were in the Texas samples (below 16%), matching the lower CWD prevalence in the state’s cervid population.”
Detection of Prions in Wild Pigs (Sus scrofa) from Areas with Reported Chronic Wasting Disease Cases, United State Volume 31, Number 1—January 2025
https://wwwnc.cdc.gov/eid/article/31/1/24-0401_article
Camel Prion Disease, Tataouine, Tunisia, 2019–2021 CDC Dispatch August 2026
Volume 32, Number 8—August 2026
Dispatch
Camel Prion Disease, Tataouine, Tunisia, 2019–2021
We report 6 cases of camel prion disease in dromedaries in Tunisia, confirming widespread occurrence in North Africa.
The recent detection of sheep scrapie in the same area of Tunisia as CPrD (15) raises the question of possible links, although no similarities have yet been shown.
https://wwwnc.cdc.gov/eid/article/32/8/25-1474_article
snip…
The detection of PrPSc in all available lymph nodes, together with the relatively young age of some camels, supports the hypothesis that CPrD is a transmissible prion disease. As in scrapie and chronic wasting disease, lymphoid involvement may enable extraneural propagation and serve as a source of environmental contamination (2–5), raising concerns about prion shedding and persistence.
snip…
Taken together, the data from Algeria and Tunisia suggest that CPrD may be endemic in certain areas. Despite the limited number of reported cases so far, the absence of active surveillance programs in those countries, unlike in Europe for scrapie and bovine spongiform encephalopathy (BSE), raises the possibility of a higher number of undetected cases. Given frequent cross-border movements and the extensive pastoral systems in which camels roam widely, contact between herds in Tunisia and herds in Algeria is frequent and has possible implications for disease circulation.
CPrD may substantially affect camel health and production, given that dromedaries are long-lived animals with extended reproductive and productive roles and are vital to pastoralist communities in arid regions. Its detection in areas where camels are central to food production underscores the need to understand its epidemiology, transmission, and long-term impact.
The emergence of a new animal prion disease also raises public health concerns. Although only BSE has shown zoonotic potential, its history highlights the necessity of monitoring all animal prion diseases. Long incubation and absence of early clinical markers complicate risk assessment, as exemplified by the decade-long delay between cattle BSE and the variant Creutzfeldt–Jakob disease epidemic in humans (2).
Given the potential implications for animal and human health and the possible socioeconomic impact in regions where dromedaries play a key role, ongoing surveillance and in-depth characterization of CPrD are essential to enhance our comprehension of its epidemiology, host range, and zoonotic potential.
Dr. Amara is emeritus professor of histology and pathological anatomy at the National School of Veterinary Medicine of Sidi Thabet in Sidi Thabet, Tunisia. He is also vice president of the Expert Committee on Animal Health at the National Center for Veterinary Surveillance in Tunisia. His primary research interests include prion diseases in ruminants and dromedaries.
https://wwwnc.cdc.gov/eid/article/32/8/25-1474_article
Identification of Camel Prion Disease in Tunisia: evidence of an emerging prion disease in North Africa
https://proceedings.science/prion-2025/papers?prod_proceedings_papers%5BrefinementList%5D%5Btrack.title.en%5D%5B0%5D=Animal%20prion%20diseases
https://camelusprp.blogspot.com/2026/02/
https://camelusprp.blogspot.com/2026/
Texas Creutzfeldt Jakob Disease CJD TSE Prion Disease 2025
Update as of 10/15/25, NO REPORT TO UPDATE…
https://cjdtexas.blogspot.com/2026/03/texas-creutzfeldt-jakob-disease-deaths.html
https://www.dshs.texas.gov/creutzfeldt-jakob-disease-cjd/creutzfeldt-jakob-disease-cjd-data
US NATIONAL PRION DISEASE PATHOLOGY SURVEILLANCE CENTER CJD TSE REPORT SEPTEMBER 2025
From the first full year of reporting CJD TSE in the US in 2000, where 90 cases of CJD was reported that year, to today, where in September 2025, the number of CJD cases reported in the last full year reporting, which would have been 2024, the number of CJD cases for 2024 was 249 cases. So, from the first full year 2000 CJD cases were 90 cases confirmed in that year, to 2024, where 2024 CJD statistics rose to 249 confirmed CJD cases in a single year. A dramatic increase in deaths, from figures that don’t seem to be dramatic. But thes figures today, they are not from “better surveillance”, that dog don’t hunt no more. They have been saying this for over 25 years, year after year, well it’s time to call it for what it is, Human Transmissible Spongiform Encephalopathy TSE Prion cases are rising, and it’s NOT because of better surveillance, or just a “happenstance of bad luck, that 85%+ of all human cases, sporadic CJD, including VPSPr, just happen spontaneously, no, it’s because of unknown environmental factors, and or iatrogenic factors, imho…terry
US NATIONAL PRION DISEASE PATHOLOGY SURVEILLANCE CENTER CJD TSE REPORT SEPTEMBER 2025
https://prionunitusaupdate.blogspot.com/2025/10/us-national-prion-disease-pathology.html
So, this is what we leave our children and grandchildren?
Terry S. Singeltary Sr.
posted by Terry S. Singeltary Sr. at
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