Missouri Suspends Issuing Permits for New Deer Breeders and Big-game Hunting Facilities
Commission approves suspension of issuing permits for new deer breeders and
big-game hunting facilities
Published on: Aug. 24, 2012
Posted by Joe Jerek
JEFFERSON CITY, Mo. – At its Aug. 24 meeting in Jefferson City, the
Missouri Conservation Commission approved changes to the Wildlife Code of
Missouri that indefinitely suspend issuing permits for new big-game hunting
facilities and new wildlife breeding facilities in Missouri that hold
white-tailed deer or mule deer.
The regulation changes to suspend the issuance of new permits do not apply
to wildlife breeders and game ranches with existing permits. The suspension of
issuing permits does not include wildlife-breeders or game ranches who wish to
hold approved wildlife species other than white-tailed deer or mule deer.
Renewal of existing permits for hunting and breeding facilities will be
considered under established requirements of the Wildlife Code.
“The Missouri Department of Conservation (MDC) is responsible for managing
and protecting the wildlife resources of the state and we take that
responsibility very seriously,” says MDC Deputy Director Tom Draper. “With
Chronic Wasting Disease now in Missouri, this suspension of issuing permits for
new deer breeders and hunting ranches is one of several actions we are taking to
help protect free-ranging deer from CWD, and to help ensure the health of
captive deer and other cervids.”
MDC permit records show there are 27 permitted big-game hunting preserves
in Missouri with white-tailed deer, and 277 permitted wildlife breeders with
white-tailed deer.
MDC has held numerous open houses to share information and get public
feedback on the issue of Chronic Wasting Disease (CWD) and Department actions to
contain the disease.
MDC provided current information on CWD and the proposed suspension of
issuing permits for new big-game ranches and wildlife breeders that hold
white-tailed deer or mule deer to members of the Missouri Whitetail Breeders and
Hunting Ranch Association at the Association’s annual conference on Aug.
4.
Draper adds that MDC continues to work with landowners, deer hunters,
members of the captive cervid industry and others on the issue of CWD and
welcomes related comments at mdc.mo.gov/node/17901.
Chronic Wasting Disease is a fatal disease that attacks the nervous systems
of cervids, such as white-tailed, mule and other types of deer. It is
transmitted by animal-to-animal contact or soil-to-animal contact. It can spread
through activities that unnaturally concentrate animals, the natural movement
and dispersal of infected free-ranging deer, the transportation of live captive
deer with CWD or the transportation and improper disposal of infected
carcasses.
According to the Missouri Department of Agriculture, there is no evidence
from existing research that CWD can spread to domestic livestock, such as sheep
or cattle. According to the Missouri Department of Health and Senior Services
(MDHSS), there is no evidence that CWD can infect people.
The first two cases of CWD in the state were found in 2010 and 2011 at two
private big-game hunting preserves in Linn and Macon counties. Following those
discoveries, the first two cases of CWD in free-ranging deer were confirmed in
2012 in Macon County. Missouri’s confirmed cases of CWD total 11 in captive deer
from the private hunting preserves and five in free-ranging deer harvested in
Macon County.
With the help of hunters, MDC has tested more than 35,000 free-ranging deer
for CWD from all parts of the state since 2002 and up to 2012. As a result of
that testing, MDC scientists have determined it is highly unlikely that CWD has
been present in the state prior to its recent discovery in northeast
Missouri.
Draper says that the Code changes allow time for MDC to further assess the
CWD situation, continue to engage stakeholders, plan for the future and identify
and utilize the best and most current science to manage the disease.
New federal regulations for the interstate movement and disease
certification of captive deer and other cervids were recently open for review
and comment through the Federal Register at www.federalregister.gov. Additional
information is pending publication. Draper says that the Code changes also give
MDC, deer breeders and others time to review these new regulations.
“Conservation efforts such as providing good habitat and progressive deer
management practices on both public and private land make Missouri a great place
to hunt deer,” Draper says. “The cultural, social and economic importance that
white-tailed deer provide the people of our state is, and will continue to be,
one of our top priorities.”
According to MDC, Missouri has more than 507,000 deer hunters who spend
about $690 million in the state each year on deer hunting and related
activities. This has an overall economic impact of $1.1 billion in Missouri each
year and supports almost 12,000 jobs. Many Missourians also enjoy viewing deer.
A 2009 Gallup survey found that about 91% of Missourians are somewhat or very
interested in observing deer in the outdoors.
Other actions the Conservation Commission and MDC have taken to limit the
spread of CWD in Missouri include regulation changes, recommendations and
continuing sampling of harvested deer to test for CWD.
The Conservation Commission approved a regulation change in May that
restricts activities that are likely to unnaturally concentrate white-tailed
deer and promote the spread of CWD. The regulation will become effective Oct.
30. It bans the placement of grain, salt products, minerals and other consumable
natural or manufactured products in the CWD Containment Zone, which consists of
Adair, Chariton, Linn, Macon, Randolph and Sullivan counties. The regulation
includes exceptions for backyard feeding of wildlife and normal agricultural,
forest management, crop and wildlife food production practices.
The Conservation Commission also approved a regulation change in May that
rescinds the antler-point restriction (four-point rule) in the CWD Containment
Zone, which became effective July 1. Yearling and adult male deer have been
found to exhibit CWD at higher rates than female deer so a reduction in the
number of male deer can help limit the spread of CWD. The dispersal of yearling
males from their natal or birth range in search of territory and mates is also
one of the primary means of expanding the distribution of CWD.
MDC also encourages hunters who harvest deer in the CWD Containment Zone
not to take whole deer carcasses or certain carcass parts out of the area.
MDC will also continue to work with hunters who harvest deer in the CWD
Containment Zone to collect samples for CWD testing.
Detailed information can be found in MDC’s “2012 Fall Deer & Turkey
Hunting Regulations and Information” booklet available at MDC offices, from
permit vendors and online at
Monday, June 11, 2012
OHIO Captive deer escapees and non-reporting
Saturday, February 04, 2012
Wisconsin 16 age limit on testing dead deer Game Farm CWD Testing Protocol
Needs To Be Revised
Monday, June 11, 2012
OHIO Captive deer escapees and non-reporting
Thursday, February 09, 2012
50 GAME FARMS IN USA INFECTED WITH CHRONIC WASTING DISEASE
Tuesday, June 05, 2012
Captive Deer Breeding Legislation Overwhelmingly Defeated During 2012
Legislative Session
Saturday, June 09, 2012
USDA Establishes a Herd Certification Program for Chronic Wasting Disease
in the United States
Oral.29: Susceptibility of Domestic Cats to CWD Infection
Amy Nalls, Nicholas J. Haley, Jeanette Hayes-Klug, Kelly Anderson, Davis
M. Seelig, Dan S. Bucy, Susan L. Kraft, Edward A. Hoover and Candace K.
Mathiason† Colorado State University; Fort Collins, CO USA†Presenting author;
Email: ckm@lamar.colostate.edu
Domestic and non-domestic cats have been shown to be susceptible to one
prion disease, feline spongiform encephalopathy (FSE), thought to be transmitted
through consumption of bovine spongiform encephalopathy (BSE) contaminated meat.
Because domestic and free ranging felids scavenge cervid carcasses, including
those in CWD affected areas, we evaluated the susceptibility of domestic cats to
CWD infection experimentally. Groups of n = 5 cats each were inoculated either
intracerebrally (IC) or orally (PO) with CWD deer brain homogenate. Between
40–43 months following IC inoculation, two cats developed mild but progressive
symptoms including weight loss, anorexia, polydipsia, patterned motor behaviors
and ataxia—ultimately mandating euthanasia. Magnetic resonance imaging (MRI) on
the brain of one of these animals (vs. two age-matched controls) performed just
before euthanasia revealed increased ventricular system volume, more prominent
sulci, and T2 hyperintensity deep in the white matter of the frontal hemisphere
and in cortical grey distributed through the brain, likely representing
inflammation or gliosis. PrPRES and widely distributed peri-neuronal vacuoles
were demonstrated in the brains of both animals by immunodetection assays. No
clinical signs of TSE have been detected in the remaining primary passage cats
after 80 months pi. Feline-adapted CWD was sub-passaged into groups (n=4 or 5)
of cats by IC, PO, and IP/SQ routes. Currently, at 22 months pi, all five IC
inoculated cats are demonstrating abnormal behavior including increasing
aggressiveness, pacing, and hyper responsiveness. Two of these cats have
developed rear limb ataxia. Although the limited data from this ongoing study
must be considered preliminary, they raise the potential for cervid-to-feline
transmission in nature. www.landesbioscience.com Prion
UPDATED CORRESPONDENCE FROM AUTHORS OF THIS STUDY I.E. COLBY, PRUSINER ET
AL, ABOUT MY CONCERNS OF THE DISCREPANCY BETWEEN THEIR FIGURES AND MY FIGURES OF
THE STUDIES ON CWD TRANSMISSION TO CATTLE ;
----- Original Message -----
From: David Colby
To: flounder9@verizon.net
Cc: stanley@XXXXXXXX
Sent: Tuesday, March 01, 2011 8:25 AM
Subject: Re: FW: re-Prions David W. Colby1,* and Stanley B. Prusiner1,2 +
Author Affiliations
Dear Terry Singeltary,
Thank you for your correspondence regarding the review article Stanley
Prusiner and I recently wrote for Cold Spring Harbor Perspectives. Dr. Prusiner
asked that I reply to your message due to his busy schedule. We agree that the
transmission of CWD prions to beef livestock would be a troubling development
and assessing that risk is important. In our article, we cite a peer-reviewed
publication reporting confirmed cases of laboratory transmission based on
stringent criteria. The less stringent criteria for transmission described in
the abstract you refer to lead to the discrepancy between your numbers and ours
and thus the interpretation of the transmission rate. We stand by our assessment
of the literature--namely that the transmission rate of CWD to bovines appears
relatively low, but we recognize that even a low transmission rate could have
important implications for public health and we thank you for bringing attention
to this matter.
Warm Regards, David Colby
--
David Colby, PhDAssistant ProfessorDepartment of Chemical
EngineeringUniversity of Delaware
====================END...TSS==============
SNIP...SEE FULL TEXT ;
UPDATED DATA ON 2ND CWD STRAIN
Wednesday, September 08, 2010
CWD PRION CONGRESS SEPTEMBER 8-11 2010
Thursday, May 31, 2012
CHRONIC WASTING DISEASE CWD PRION2012 Aerosol, Inhalation transmission,
Scrapie, cats, species barrier, burial, and more
Tuesday, January 24, 2012
CWD found in two free-ranging deer from Macon County Missouri
Friday, October 21, 2011
Chronic Wasting Disease Found in Captive Deer Missouri
Friday, February 26, 2010
Chronic wasting disease found in Missouri deer
Sunday, January 22, 2012
Chronic Wasting Disease CWD cervids interspecies transmission
*** Chronic Wasting Disease CWD CDC REPORT MARCH 2012 ***
Saturday, February 18, 2012
Occurrence, Transmission, and Zoonotic Potential of Chronic Wasting Disease
CDC Volume 18, Number 3—March 2012
CWD has been identified in free-ranging cervids in 15 US states and 2
Canadian provinces and in ≈ 100 captive herds in 15 states and provinces and in
South Korea (Figure 1, panel B).
SNIP...
Long-term effects of CWD on cervid populations and ecosystems remain
unclear as the disease continues to spread and prevalence increases. In captive
herds, CWD might persist at high levels and lead to complete herd destruction in
the absence of human culling. Epidemiologic modeling suggests the disease could
have severe effects on free-ranging deer populations, depending on hunting
policies and environmental persistence (8,9). CWD has been associated with large
decreases in free-ranging mule deer populations in an area of high CWD
prevalence (Boulder, Colorado, USA) (5).
PLEASE STUDY THIS MAP, COMPARE FARMED CWD TO WILD CWD...TSS
Saturday, February 18, 2012
Occurrence, Transmission, and Zoonotic Potential of Chronic Wasting Disease
CDC Volume 18, Number 3—March 2012
Friday, August 24, 2012
Diagnostic accuracy of rectal mucosa biopsy testing for chronic wasting
disease within white-tailed deer (Odocoileus virginianus) herds in North America
TSS
posted by Terry S. Singeltary Sr. at
11:25 AM
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