Missouri Gov. Nixon vetoes two bills defining captive deer as livestock
Gov. Nixon vetoes two bills defining captive deer as livestock
July 8, 2014
Governor points out Missouri Constitution gives Conservation Commission
sole regulatory authority over wildlife; says bill provisions are clearly
unconstitutional Columbia, MOGov.
Jay Nixon today vetoed two bills that would have redefined the term
“livestock” to include captive deer in order to eliminate the role of the
Missouri Department of Conservation in regulating white-tailed deer. The
Governor said those provisions of Senate Bill 506 and House Bill 1326 would go
against longstanding successful conservation practices and also would clearly
violate the Missouri Constitution, which gives exclusive authority over game and
wildlife resources to the Missouri Conservation Commission.
“For more than 75 years, our Department of Conservation has been held up as
a model for wildlife management agencies across the country because of its
incredible success,” Gov. Nixon said. “Redefining deer as livestock to remove
the regulatory role of Department defies both its clear record of achievement as
well as common sense. White-tailed deer are wildlife and also game animals – no
matter if they’re roaming free, or enclosed in a fenced area.”
In his veto message, the Governor cites the exclusive authority of the
Missouri Conservation Commission provided by the Missouri Constitution under
Article IV, Section 40(a). He also said that under the stewardship of the
Missouri Department of Conservation, the state’s population of white-tailed deer
has grown from fewer than 2,000 in the early 1930s to an estimated 1.3 million
today, and that the 500,000 deer hunters contribute $1 billion to Missouri’s
economy.
“Growing and managing our deer herd and fostering the hunting opportunities
that we enjoy takes hard work and sound science, and the Department of
Conservation should be commended for employing both to preserve this important
part of our heritage, not stripped of its authority to do so in order to protect
narrow interests,” the veto message reads.
Gov. Nixon noted that “it is unfortunate that the legislature insisted on
amending this unconstitutional provision to two pieces of legislation that
otherwise contain worthy provisions advancing Missouri agriculture.”
The Governor discussed his actions on the two bills at a special meeting
today of the Missouri Conservation Commission in Columbia. The veto message on
Senate Bill 506 can be found here,
and the veto message on House Bill 1326 can be found here.
PRION 2014 CONFERENCE
CHRONIC WASTING DISEASE CWD
A FEW FINDINGS ;
Conclusions. To our knowledge, this is the first established experimental
model of CWD in TgSB3985. We found evidence for co-existence or divergence of
two CWD strains adapted to Tga20 mice and their replication in TgSB3985 mice.
Finally, we observed phenotypic differences between cervid-derived CWD and
CWD/Tg20 strains upon propagation in TgSB3985 mice. Further studies are underway
to characterize these strains.
We conclude that TSE infectivity is likely to survive burial for long time
periods with minimal loss of infectivity and limited movement from the original
burial site. However PMCA results have shown that there is the potential for
rainwater to elute TSE related material from soil which could lead to the
contamination of a wider area. These experiments reinforce the importance of
risk assessment when disposing of TSE risk materials.
The results show that even highly diluted PrPSc can bind efficiently to
polypropylene, stainless steel, glass, wood and stone and propagate the
conversion of normal prion protein. For in vivo experiments, hamsters were ic
injected with implants incubated in 1% 263K-infected brain homogenate. Hamsters,
inoculated with 263K-contaminated implants of all groups, developed typical
signs of prion disease, whereas control animals inoculated with non-contaminated
materials did not.
Our data establish that meadow voles are permissive to CWD via peripheral
exposure route, suggesting they could serve as an environmental reservoir for
CWD. Additionally, our data are consistent with the hypothesis that at least two
strains of CWD circulate in naturally-infected cervid populations and provide
evidence that meadow voles are a useful tool for CWD strain typing.
Conclusion. CWD prions are shed in saliva and urine of infected deer as
early as 3 months post infection and throughout the subsequent >1.5 year
course of infection. In current work we are examining the relationship of
prionemia to excretion and the impact of excreted prion binding to surfaces and
particulates in the environment.
Conclusion. CWD prions (as inferred by prion seeding activity by RT-QuIC)
are shed in urine of infected deer as early as 6 months post inoculation and
throughout the subsequent disease course. Further studies are in progress
refining the real-time urinary prion assay sensitivity and we are examining more
closely the excretion time frame, magnitude, and sample variables in
relationship to inoculation route and prionemia in naturally and experimentally
CWD-infected cervids.
Conclusions. Our results suggested that the odds of infection for CWD is
likely controlled by areas that congregate deer thus increasing direct
transmission (deer-to-deer interactions) or indirect transmission
(deer-to-environment) by sharing or depositing infectious prion proteins in
these preferred habitats. Epidemiology of CWD in the eastern U.S. is likely
controlled by separate factors than found in the Midwestern and endemic areas
for CWD and can assist in performing more efficient surveillance efforts for the
region.
Conclusions. During the pre-symptomatic stage of CWD infection and
throughout the course of disease deer may be shedding multiple LD50 doses per
day in their saliva. CWD prion shedding through saliva and excreta may account
for the unprecedented spread of this prion disease in nature.
Monday, June 23, 2014
*** PRION 2014 CONFERENCE CHRONIC WASTING DISEASE CWD
Tuesday, July 01, 2014
*** CHRONIC WASTING DISEASE CWD TSE PRION DISEASE, GAME FARMS, AND
POTENTIAL RISK FACTORS THERE FROM ***
Thursday, July 03, 2014
*** How Chronic Wasting Disease is affecting deer population and what’s the
risk to humans and pets? ***
Monday, July 07, 2014
Governor Nixon Hosting Press Conference to “Act” on Captive Deer
Legislation in Missouri
TSS
posted by Terry S. Singeltary Sr. at
11:32 AM
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