Sunday, July 13, 2014

Missouri Governor sends message with vetoes of captive deer bills Making a strong statement

Driftwood Outdoors: Making a strong statement
Governor sends message with vetoes of captive deer bills .
Brandon Butler, outdoors columnist,
Sunday, July 13, 2014
Deer are wildlife, not livestock. Making a more obvious statement would be difficult.
During Gov. Nixon’s announcement of his vetoes of SB 506 and HB 1326, which would reclassify captive deer as livestock, he proclaimed the reclassification of “wildlife” to “livestock” would violate the Missouri Constitution.
Nixon said, “White-tailed deer are wildlife, and they are also a game animal. Putting them behind a fence does not change that fact.”
You would think violating our Constitution would be the end of these bills, but no. Certain legislators are declaring they will override the Governor’s vetoes and force control of captive deer on the Missouri Department of Agriculture, which has openly testified against the transfer.
The Governor has stated the bills are unconstitutional and has vetoed them. The Missouri Department of Conservation doesn’t want these bills to pass. The Missouri Department of Agriculture doesn’t want these bills to pass. The Kansas City Star, St. Louis Post-Dispatch, Jefferson City News Tribune, Poplar Bluff Daily American Republic and many more media sources across Missouri have denounced the bills. The Conservation Federation of Missouri, Quality Deer Management Association, Whitetails Unlimited, National Wild Turkey Federation, Mule Deer Foundation, National Wildlife Federation, Sierra Club and just about every other conservation organization you can name publicly opposes these bills.
And most importantly, the vast majority of our state’s citizens, represented by an outpouring of letters, emails and phone calls to the Governor’s office, which was reported in an Associated Press article, don’t want these bills to pass.
So you’re probably left scratching your head wondering why in the world we’re still talking about calling deer livestock. Why would legislators who are elected by the people and paid with public tax dollars focus any of their efforts on overriding unconstitutional legislation the vast majority of our state’s citizens are against?
In a Poplar Bluff Daily American Republic article, Rep. Casey Guernsey said MDC had made him and other legislators mad, calling the department an example of “bad government,” so he was intent on fast-tracking and passing this bill as a way, in no uncertain words, to punish them with a veto-proof majority”
So because they are “mad,” some of your elected senators and representatives want to punish MDC by stripping their control of wildlife management so a few shooting ranches can continue to ship potentially diseased, drugged, out-of-state deer into fenced facilities where only the wealthiest can afford to pay tens-of-thousands of dollars to kill genetically mutated “livestock.” Doesn’t seem right, does it? And you know who really suffers? Not MDC, you. And everyone who believes wildlife is a public resource.
It goes much deeper, though. If you’re a sportsman, or just someone who enjoys nature, you may not realize just how incredibly fortunate you are to live in Missouri. The system of conservation governance we have here in our state is unique, and successful beyond the wildest dreams of most other states. In 1936, when forest, fish and wildlife were at all time lows, the people of Missouri made a bold statement by passing a constitutional amendment that took control of our state’s distraught natural resources away from the politicians who had propagated such decimation and placed the responsibility of conservation management with a citizen led Conservation Commission.
Because the General Assembly does not have authority over MDC, these same “mad” legislators argue MDC is “accountable to no one.” Nothing could be further from the truth. MDC is accountable to everyone. For nearly 80 years, MDC has answered to citizens like you and me. The conservation-minded Missourians who came before us fought to establish our unique and successful model of conservation governance. MDC continues to honor their legacy.
In Missouri, wildlife scientists who have the necessary funding to do their jobs at the highest level manage conservation. The reason why our fish and wildlife thrive here is because politicians can’t just hand out favors to their friends like a few of them are trying to do now.
Most of our legislators are proud to live in a state with such an esteemed system of science-based conservation management, and are happy to leave our world-class system alone. Yet, a select few are hell-bent on destroying what nearly 80 years of sound-science and public resource management has built. I suggest you find out where your legislators stand.
The few leading the fight to override the vetoes of SB 506 and HB 1326 are claiming MDC is infringing on the private property rights of high-fence captive shooting facilities, because these “farmers” nurture and care for these animals. If you believe shipping a drugged deer from out-of-state to a fenced-facility in a gooseneck trailer and then letting someone pay to shoot it is nurturing and caring, then we have vastly different definitions of those words.
And if these politicians want to talk about private property rights, then what about the private property rights of the landowners who border or are in close proximity to these facilities? Their private property rights are at risk every time another out-of-state deer is trucked into a facility, potentially bringing with it Chronic Wasting Disease (CWD) or another deadly wildlife disease. Once CWD is passed through the fence onto a neighbor’s land, what do you think happens to that neighbor’s land value through no fault of their own? Should the private property rights of a single landowner override the private property rights of all their neighbors? I’m not a lawyer, but I would think your private property rights end when they threaten the rights of everyone around you.
“Short-sighted and cynical assaults like this are the very reason why the Conservation Commission must and shall remain a strong and independent custodian of our state’s time honored traditions and precious resources,” Gov. Nixon said.
The differences between the two sides of this entire captive cervid issue are many, but it essentially boils down to this. Those opposed are citizen conservationists passionate about wildlife and ethical hunting standards, who give selflessly in support of public resources. Those in favor are financially invested.
Gov. Nixon said it’s time for conservation to stop playing defense and to go on offense. I echo his sentiment. If you have yet to engage in this fight, the time is now.
Every Missouri conservationist must take action. Talk about this issue in your community. Write letters to your local newspapers. Contact your legislators, and insist your family and friends do the same. Tell your legislators pointedly you support the vetoes of SB 506 and HB 1326.
See you down the trail …
Brandon Butler is an outdoors columnist for the News Tribune. Contact him at
here is the video of the speech the good Governor Nixon made on the veto. great speech ''deer are NOT livestock''... ''we cannot head back to the bad old days, when wildlife decisions, were made by officials worried about the next election, rather than professionals committed to doing what's best for the next generation, and the ones to follow that''... BRAVO!
*** Spraker suggested an interesting explanation for the occurrence of CWD. The deer pens at the Foot Hills Campus were built some 30-40 years ago by a Dr. Bob Davis. At or abut that time, allegedly, some scrapie work was conducted at this site. When deer were introduced to the pens they occupied ground that had previously been occupied by sheep. ... also, see where even decades back, the USDA had the same thought as they do today with CWD, not their problem...see page 27 below as well, where USDA stated back then, the same thing they stated in the state of Pennsylvania, not their damn business, once they escape, and they said the same thing about CWD in general back then ; ”The occurrence of CWD must be viewed against the contest of the locations in which it occurred. It was an incidental and unwelcome complication of the respective wildlife research programmes. Despite it’s subsequent recognition as a new disease of cervids, therefore justifying direct investigation, no specific research funding was forthcoming.
***The USDA veiwed it as a wildlife problem and consequently not their province!” 26.
”The occurrence of CWD must be viewed against the contest of the locations in which it occurred. It was an incidental and unwelcome complication of the respective wildlife research programmes. Despite it’s subsequent recognition as a new disease of cervids, therefore justifying direct investigation, no specific research funding was forthcoming. The USDA veiwed it as a wildlife problem and consequently not their province!” 26.
sound familiar $$$
Sunday, January 06, 2013
USDA TO PGC ONCE CAPTIVES ESCAPE *** "it‘s no longer its business.”
Tuesday, May 28, 2013
Chronic Wasting Disease CWD quarantine Louisiana via CWD index herd Pennsylvania Update May 28, 2013
*** 6 doe from Pennsylvania CWD index herd still on the loose in Louisiana, quarantine began on October 18, 2012, still ongoing, Lake Charles premises.
Saturday, June 29, 2013
Tuesday, June 11, 2013
*** CWD GONE WILD, More cervid escapees from more shooting pens on the loose in Pennsylvania
Monday, June 24, 2013
The Effects of Chronic Wasting Disease on the Pennsylvania Cervid Industry Following its Discovery
Thursday, October 03, 2013
*** TAHC ADOPTS CWD RULE THAT the amendments __REMOVE__ the requirement for a specific fence height for captives ***
Texas Animal Health Commission (TAHC)
October 3, 2013
According to Wisconsin’s White-Tailed Deer Trustee Dr. James Kroll, people who call for more public hunting opportunities are “pining for socialism.”
He further states, “(Public) Game management is the last bastion of communism.”
“Game Management,” says James Kroll, driving to his high-fenced, two-hundred-acre spread near Nacogdoches, “is the last bastion of communism.”
Kroll, also known as Dr. Deer, is the director of the Forestry Resources Institute of Texas at Stephen F. Austin State University, and the “management” he is referring to is the sort practiced by the State of Texas.
The 55-year-old Kroll is the leading light in the field of private deer management as a means to add value to the land. His belief is so absolute that some detractors refer to him as Dr. Dough, implying that his eye is on the bottom line more than on the natural world.
Kroll, who has been the foremost proponent of deer ranching in Texas for more than thirty years, doesn’t mind the controversy and certainly doesn’t fade in the heat. People who call for more public lands are “cocktail conservationists,” he says, who are really pining for socialism. He calls national parks “wildlife ghettos” and flatly accuses the government of gross mismanagement. He argues that his relatively tiny acreage, marked by eight-foot fences and posted signs warning off would-be poachers, is a better model for keeping what’s natural natural while making money off the land.
What does this all mean?
My initial reaction, which is one that I predicted when Kroll was named to the state’s deer trustee position, is that his team’s final recommendations — if implemented — will be heavily skewed toward the state’s larger landowners (500+ acres) and folks who own small parcels in areas comprised mostly of private land. It is also my prediction that the final recommendations (again, if implemented) will do little, if anything, to improve deer herds and deer hunting on Wisconsin’s 5.7 million acres of public land. Where does this leave the public-land hunter? “It will suck to be you,” said one deer manager who asked to remain anonymous out of fear for his job. “The resources and efforts will go toward improving the private land sector. This is all about turning deer hunting away from the Public Land Doctrine and more toward a European-style of management — like they have in Texas.”
Friday, June 01, 2012
Monday, February 11, 2013
TEXAS CHRONIC WASTING DISEASE CWD Four New Positives Found in Trans Pecos
Wednesday, September 04, 2013
***cwd - cervid captive livestock escapes, loose and on the run in the wild...
Saturday, February 04, 2012
*** Wisconsin 16 age limit on testing dead deer Game Farm CWD Testing Protocol Needs To Be Revised
Approximately 4,200 fawns, defined as deer under 1 year of age, were sampled from the eradication zone over the last year. The majority of fawns sampled were between the ages of 5 to 9 months, though some were as young as 1 month.
*** Two of the six fawns with CWD detected were 5 to 6 months old.
All six of the positive fawns were taken from the core area of the CWD eradication zone where the highest numbers of positive deer have been identified.
Infectious agent of sheep scrapie may persist in the environment for at least 16 years
Gudmundur Georgsson1, Sigurdur Sigurdarson2 and Paul Brown3
New studies on the heat resistance of hamster-adapted scrapie agent: Threshold survival after ashing at 600°C suggests an inorganic template of replication
Prion Infected Meat-and-Bone Meal Is Still Infectious after Biodiesel Production
Detection of protease-resistant cervid prion protein in water from a CWD-endemic area
A Quantitative Assessment of the Amount of Prion Diverted to Category 1 Materials and Wastewater During Processing
Rapid assessment of bovine spongiform encephalopathy prion inactivation by heat treatment in yellow grease produced in the industrial manufacturing process of meat and bone meals
Survival and Limited Spread of TSE Infectivity after Burial
Sunday, September 01, 2013
hunting over gut piles and CWD TSE prion disease
Sunday, April 13, 2014
Mineral licks: motivational factors for visitation and accompanying disease risk at communal use sites of elk and deer
Environmental Geochemistry and Health
Monday, June 18, 2012
natural cases of CWD in eight Sika deer (Cervus nippon) and five Sika/red deer crossbreeds captive Korea and Experimental oral transmission to red deer (Cervus elaphus elaphus)
spreading cwd around...tss
Between 1996 and 2002, chronic wasting disease was diagnosed in 39 herds of farmed elk in Saskatchewan in a single epidemic. All of these herds were depopulated as part of the Canadian Food Inspection Agency’s (CFIA) disease eradication program. Animals, primarily over 12 mo of age, were tested for the presence CWD prions following euthanasia. Twenty-one of the herds were linked through movements of live animals with latent CWD from a single infected source herd in Saskatchewan, 17 through movements of animals from 7 of the secondarily infected herds.
***The source herd is believed to have become infected via importation of animals from a game farm in South Dakota where CWD was subsequently diagnosed (7,4). A wide range in herd prevalence of CWD at the time of herd depopulation of these herds was observed. Within-herd transmission was observed on some farms, while the disease remained confined to the introduced animals on other farms.
spreading cwd around...tss
Friday, May 13, 2011
Chronic Wasting Disease (CWD) outbreaks and surveillance program in the Republic of Korea Chronic Wasting Disease (CWD) outbreaks and surveillance program in the Republic of Korea
Hyun-Joo Sohn, Yoon-Hee Lee, Min-jeong Kim, Eun-Im Yun, Hyo-Jin Kim, Won-Yong Lee, Dong-Seob Tark, In- Soo Cho, Foreign Animal Disease Research Division, National Veterinary Research and Quarantine Service, Republic of Korea
Chronic wasting disease (CWD) has been recognized as an important prion disease in native North America deer and Rocky mountain elks. The disease is a unique member of the transmissible spongiform encephalopathies (TSEs), which naturally affects only a few species. CWD had been limited to USA and Canada until 2000.
On 28 December 2000, information from the Canadian government showed that a total of 95 elk had been exported from farms with CWD to Korea. These consisted of 23 elk in 1994 originating from the so-called “source farm” in Canada, and 72 elk in 1997, which had been held in pre export quarantine at the “source farm”.Based on export information of CWD suspected elk from Canada to Korea, CWD surveillance program was initiated by the Ministry of Agriculture and Forestry (MAF) in 2001.
All elks imported in 1997 were traced back, however elks imported in 1994 were impossible to identify. CWD control measures included stamping out of all animals in the affected farm, and thorough cleaning and disinfection of the premises. In addition, nationwide clinical surveillance of Korean native cervids, and improved measures to ensure reporting of CWD suspect cases were implemented.
Total of 9 elks were found to be affected. CWD was designated as a notifiable disease under the Act for Prevention of Livestock Epidemics in 2002.
Additional CWD cases - 12 elks and 2 elks - were diagnosed in 2004 and 2005.
Since February of 2005, when slaughtered elks were found to be positive, all slaughtered cervid for human consumption at abattoirs were designated as target of the CWD surveillance program. Currently, CWD laboratory testing is only conducted by National Reference Laboratory on CWD, which is the Foreign Animal Disease Division (FADD) of National Veterinary Research and Quarantine Service (NVRQS).
In July 2010, one out of 3 elks from Farm 1 which were slaughtered for the human consumption was confirmed as positive. Consequently, all cervid – 54 elks, 41 Sika deer and 5 Albino deer – were culled and one elk was found to be positive. Epidemiological investigations were conducted by Veterinary Epidemiology Division (VED) of NVRQS in collaboration with provincial veterinary services.
Epidemiologically related farms were found as 3 farms and all cervid at these farms were culled and subjected to CWD diagnosis. Three elks and 5 crossbreeds (Red deer and Sika deer) were confirmed as positive at farm 2.
All cervids at Farm 3 and Farm 4 – 15 elks and 47 elks – were culled and confirmed as negative.
Further epidemiological investigations showed that these CWD outbreaks were linked to the importation of elks from Canada in 1994 based on circumstantial evidences.
In December 2010, one elk was confirmed as positive at Farm 5. Consequently, all cervid – 3 elks, 11 Manchurian Sika deer and 20 Sika deer – were culled and one Manchurian Sika deer and seven Sika deer were found to be positive. This is the first report of CWD in these sub-species of deer. Epidemiological investigations found that the owner of the Farm 2 in CWD outbreaks in July 2010 had co-owned the Farm 5.
In addition, it was newly revealed that one positive elk was introduced from Farm 6 of Jinju-si Gyeongsang Namdo. All cervid – 19 elks, 15 crossbreed (species unknown) and 64 Sika deer – of Farm 6 were culled, but all confirmed as negative.
: Corresponding author: Dr. Hyun-Joo Sohn (+82-31-467-1867, E-mail: 2011 Pre-congress Workshop: TSEs in animals and their environment 5
Friday, May 13, 2011
Chronic Wasting Disease (CWD) outbreaks and surveillance program in the Republic of Korea
Tuesday, July 01, 2014
Thursday, July 03, 2014
*** How Chronic Wasting Disease is affecting deer population and what’s the risk to humans and pets? ***
Conclusions. To our knowledge, this is the first established experimental model of CWD in TgSB3985. We found evidence for co-existence or divergence of two CWD strains adapted to Tga20 mice and their replication in TgSB3985 mice. Finally, we observed phenotypic differences between cervid-derived CWD and CWD/Tg20 strains upon propagation in TgSB3985 mice. Further studies are underway to characterize these strains.
We conclude that TSE infectivity is likely to survive burial for long time periods with minimal loss of infectivity and limited movement from the original burial site. However PMCA results have shown that there is the potential for rainwater to elute TSE related material from soil which could lead to the contamination of a wider area. These experiments reinforce the importance of risk assessment when disposing of TSE risk materials.
The results show that even highly diluted PrPSc can bind efficiently to polypropylene, stainless steel, glass, wood and stone and propagate the conversion of normal prion protein. For in vivo experiments, hamsters were ic injected with implants incubated in 1% 263K-infected brain homogenate. Hamsters, inoculated with 263K-contaminated implants of all groups, developed typical signs of prion disease, whereas control animals inoculated with non-contaminated materials did not.
Our data establish that meadow voles are permissive to CWD via peripheral exposure route, suggesting they could serve as an environmental reservoir for CWD. Additionally, our data are consistent with the hypothesis that at least two strains of CWD circulate in naturally-infected cervid populations and provide evidence that meadow voles are a useful tool for CWD strain typing.
Conclusion. CWD prions are shed in saliva and urine of infected deer as early as 3 months post infection and throughout the subsequent >1.5 year course of infection. In current work we are examining the relationship of prionemia to excretion and the impact of excreted prion binding to surfaces and particulates in the environment.
Conclusion. CWD prions (as inferred by prion seeding activity by RT-QuIC) are shed in urine of infected deer as early as 6 months post inoculation and throughout the subsequent disease course. Further studies are in progress refining the real-time urinary prion assay sensitivity and we are examining more closely the excretion time frame, magnitude, and sample variables in relationship to inoculation route and prionemia in naturally and experimentally CWD-infected cervids.
Conclusions. Our results suggested that the odds of infection for CWD is likely controlled by areas that congregate deer thus increasing direct transmission (deer-to-deer interactions) or indirect transmission (deer-to-environment) by sharing or depositing infectious prion proteins in these preferred habitats. Epidemiology of CWD in the eastern U.S. is likely controlled by separate factors than found in the Midwestern and endemic areas for CWD and can assist in performing more efficient surveillance efforts for the region.
Conclusions. During the pre-symptomatic stage of CWD infection and throughout the course of disease deer may be shedding multiple LD50 doses per day in their saliva. CWD prion shedding through saliva and excreta may account for the unprecedented spread of this prion disease in nature.
P.28: Modeling prion species barriers and the new host effect using RT-QuIC
Kristen A Davenport, Davin M Henderson, Candace K Mathiason, and Edward A Hoover Prion Research Center; Colorado State University; Fort Collins, CO USA
The propensity for trans-species prion transmission is related to the structural characteristics of the enciphering and heterologous PrP, but the exact mechanism remains mostly mysterious.
Studies of the effects of primary or tertiary prion protein Prion 37 structures on trans-species prion transmission have relied upon animal bioassays, making the influence of prion protein structure vs. host co-factors (e.g. cellular constituents, trafficking, and innate immune interactions) difficult to dissect.
As an alternative strategy, we are using real-time quaking-induced conversion (RT-QuIC) to investigate the propensity for and the kinetics of trans-species prion conversion. RT-QuIC has the advantage of providing more defined conditions of seeded conversion to study the specific role of native PrP:PrPRES interactions as a component of the species barrier.
We are comparing chronic wasting disease (CWD) and bovine spongiform encephalopathy (BSE) prions by seeding each prion into its native host recPrP (full-length bovine recPrP, or white tail deer recPrP) vs. into the heterologous species.
Upon establishing the characteristics of intra-species and inter-species prion seeding for CWD and BSE prions, we will evaluate the seeding kinetics and cross-species seeding efficiencies of BSE and CWD passaged into a common new host—feline—shown to be a permissive host for both CWD and BSE.
*** We hypothesize that both BSE prions and CWD prions passaged through felines will seed human recPrP more efficiently than BSE or CWD from the original hosts, evidence that the new host will dampen the species barrier between humans and BSE or CWD. The new host effect is particularly relevant as we investigate potential means of trans-species transmission of prion disease.
Monday, June 23, 2014
Monday, June 23, 2014
Sunday, June 29, 2014
Transmissible Spongiform Encephalopathy TSE Prion Disease North America 2014
how many states have $465,000., and can quarantine and purchase there from, each cwd said infected farm, but how many states can afford this for all the cwd infected cervid game ranch type farms ???
Tuesday, December 20, 2011
*** The CWD infection rate was nearly 80%, the highest ever in a North American captive herd.
*** RECOMMENDATION: That the Board approve the purchase of 80 acres of land for $465,000 for the Statewide Wildlife Habitat Program in Portage County and approve the restrictions on public use of the site.
*** supercalifragilisticexpialidocious or superovulationcwdtsepriondocious ? ***
*** After a natural route of exposure, 100% of WTD were susceptible to scrapie.
Deer developed clinical signs of wasting and mental depression and were necropsied from 28 to 33 months PI. Tissues from these deer were positive for PrPSc by IHC and WB. Similar to IC inoculated deer, samples from these deer exhibited two different molecular profiles: samples from obex resembled CWD whereas those from cerebrum were similar to the original scrapie inoculum. On further examination by WB using a panel of antibodies, the tissues from deer with scrapie exhibit properties differing from tissues either from sheep with scrapie or WTD with CWD. Samples from WTD with CWD or sheep with scrapie are strongly immunoreactive when probed with mAb P4, however, samples from WTD with scrapie are only weakly immunoreactive. In contrast, when probed with mAb’s 6H4 or SAF 84, samples from sheep with scrapie and WTD with CWD are weakly immunoreactive and samples from WTD with scrapie are strongly positive. This work demonstrates that WTD are highly susceptible to sheep scrapie, but on first passage, scrapie in WTD is differentiable from CWD.
*** After a natural route of exposure, 100% of white-tailed deer were susceptible to scrapie.
(It was noted with concern that hormone extracts could be manufactured by a veterinary surgeon for administration to animals under his care without any Medicines Act Control.)
This was used to help cows super ovulate.
*** This tissue was considered to be of greatest risk of containing BSE and consequently transmitting the disease. ***
Considered to be of great risk.
snip...see full text ;
Thursday, July 10, 2014
*** supercalifragilisticexpialidocious or superovulationcwdtsepriondocious ? ***
Monday, July 07, 2014
Governor Nixon Hosting Press Conference to “Act” on Captive Deer Legislation in Missouri
Tuesday, July 08, 2014
Missouri Gov. Nixon vetoes two bills defining captive deer as livestock
Tuesday, May 27, 2014
New Missouri CWD regulations... You know where we stand... What are your thoughts?
Singeltary submission ;
Program Standards: Chronic Wasting Disease Herd Certification Program and Interstate Movement of Farmed or Captive Deer, Elk, and Moose
*** DOCUMENT ID: APHIS-2006-0118-0411
Thursday, May 01, 2014
Missouri DNR CWD prevention and captive cervid farming Update
Tuesday, April 29, 2014
Missouri SB964 Immediate Action Required: Captive Cervids Transfer is Still Alive in Senate
Terry S. Singeltary Sr. Bacliff, Texas USA 77518
Contact the Governor's Office Your message has been successfully submitted.
Thank you for contacting the office of Missouri Governor Jay Nixon.
Thursday, May 15, 2014
Missouri Stripping MDC regulatory authority of deer farms SB 506 HOW THEY VOTED Singeltary letter to Governor Nixon
Sunday, June 22, 2014
Governor Nixon Missouri Urged to VETO Legislation turning over captive shooting pens to USDA
October 11, 2013
Protecting Missouri's White-Tailed Deer fill out the questionnaire ;
Friday, September 20, 2013
Missouri State records show gaps in oversight of captive deer farms, ranches
Sunday, June 09, 2013
Missouri House forms 13-member Interim Committee on the Cause and Spread of Chronic Wasting Disease CWD
Tuesday, March 26, 2013
CWD Missouri remains confined to Linn-Macon-County Core Area with four new cases
Wednesday, January 23, 2013
Missouri sixth case CWD documented northwest Macon County
Tuesday, January 24, 2012
CWD found in two free-ranging deer from Macon County Missouri
Friday, February 26, 2010
Chronic wasting disease found in Missouri deer
Sunday, March 25, 2012
Three more cases of CWD found in free-ranging deer in Macon County
From: Terry S. Singeltary Sr.
Sent: Thursday, March 29, 2012 6:26 PM
Cc: ; ; ; ; ; ; ; ; ;
Subject: re-Missouri officials seek states' advice on chronic wasting disease in deer
Thursday, May 31, 2012
Missouri MDC staff will provide information on five recently found cases of CWD in free-ranging deer in northwest Macon County June 2, 2012
Wednesday, September 05, 2012
Missouri MDC seeks hunters’ help when processing harvested deer and preventing CWD
Thursday, December 20, 2012
MISSOURI Initial CWD sampling test results available online from MDC so far one adult buck has tested positive for the disease
Friday, October 21, 2011
Chronic Wasting Disease Found in Captive Deer Missouri October 20, 2011
Chronic Wasting Disease Found in Captive Deer
The Missouri departments of Agriculture, Conservation and Health and Senior Services and the U.S. Department of Agriculture announced that a captive white-tailed deer in Macon County, Missouri has tested positive for Chronic Wasting Disease (CWD). CWD is a neurological disease found in deer, elk and moose.
The animal that tested positive for CWD was a captive white-tailed deer inspected as part of the State's CWD surveillance and testing program. Preliminary tests were conducted by the USDA National Veterinary Services Laboratory in Ames, Iowa.
In February 2010 a case of CWD was confirmed in Linn County on a captive hunting preserve operated by the same entity, Heartland Wildlife Ranches, LLC. The Linn County facility was depopulated and no further infection was identified at that facility. The current case was identified through increased surveillance required by the management plan implemented from the previous CWD incident.
Friday, October 21, 2011
Chronic Wasting Disease Found in Captive Deer Missouri
The Missouri Department of Agriculture discovers the state's first case of CWD in a captive white-tailed deer.
Friday, February 26, 2010
Chronic wasting disease found in Missouri deer February 25, 2010
Chronic Wasting Disease Found in Captive Deer
The Missouri Departments of Agriculture, Conservation and Health and Senior Services and the U.S. Department of Agriculture announced today that a captive white-tailed deer in Linn County, Missouri has tested positive for Chronic Wasting Disease (CWD). CWD is a neurological disease found in deer, elk and moose.
"There is no evidence that CWD poses a risk to domestic animals or humans," said State Veterinarian Dr. Taylor Woods. "We have protocols in place to quickly and effectively handle these situations."
The animal that tested positive for CWD was a white-tailed deer inspected as part of the State's CWD surveillance and testing program. Preliminary tests were conducted by the USDA National Veterinary Services Laboratory in Ames, Iowa.
Upon receiving the confirmed CWD positive, Missouri's departments of Agriculture, Conservation and Health and Senior Services initiated their CWD Contingency Plan. The plan was developed in 2002 by the Cervid Health Committee, a task force comprised of veterinarians, animal health officers and conservation officers from USDA, MDA, MDC and DHSS working together to mitigate challenges associated with CWD.
CWD is transmitted by live animal to animal contact or soil to animal contact. The disease was first recognized in 1967 in captive mule deer in the Colorado Division of Wildlife captive wildlife research facility in Fort Collins, Colorado. CWD has been documented in deer and/or elk in Colorado, Illinois, Kansas, Michigan, Minnesota, Montana, Nebraska, New Mexico, New York, Oklahoma, South Dakota, Utah, Virginia, West Virginia, Wisconsin, and the Canadian Provinces of Alberta and Saskatchewan. There has been no evidence that the disease can be transmitted to humans.
"Missouri's proactive steps to put a testing protocol in place and create a contingency plan years ago is proving beneficial. We are in a solid position to follow pre-established steps to ensure Missouri's valuable whitetail deer resource remains healthy and strong," said Jason Sumners Missouri's Deer Biologist.
For more information regarding CWD, please contact Dr. Taylor Woods at (573) 751-3377.
Thursday, May 01, 2014
Missouri DNR CWD prevention and captive cervid farming Update
kind regards, terry


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