Arkansas AGFC Confirms 4 More CWD TSE Prion in WTD NW Counties
CWD confirmed in three NW Arkansas counties
Jan. 9, 2018
Keith Stephens Chief of Communications
LITTLE ROCK – Chronic wasting disease, detected in Arkansas almost two years ago, has been found in three more counties. Four white-tailed deer in Benton, Washington and Sebastian counties recently tested positive for the deadly disease, according to the Arkansas Game and Fish Commission.
The deer in Benton County were a 2½-year-old doe near Decatur and a 5½-year-old doe near Springtown. The Sebastian County deer was an adult buck near Lavaca, and the one from Washington County was a 1½-year-old buck near Prairie Grove. All four were harvested by hunters during the 2017-18 deer season, and confirmed as CWD-positive by the Wisconsin Veterinary Diagnostic Laboratory in Madison.
Test results have not been received for all samples that have been collected; it’s possible more deer and elk could test positive for the disease. Since these positive samples were detected outside the current CWD Management Zone, the AGFC will continue their review to ensure all information is accurate.
“Although CWD is a serious threat to Arkansas’s elk and white-tailed deer, we are not the first to deal with the disease,” AGFC Director Pat Fitts said. “Our staff is prepared and, with help from the public, will respond with effective measures. We have learned from the experiences of 23 other states.”
CWD was first detected in Arkansas Feb. 23, 2016, when a hunter-harvested elk in Newton County tested positive. The first Arkansas deer with CWD was verified March 3, 2016, also in Newton County.
Public meetings in the area will be scheduled as forums to discuss plans and to answer questions.
CWD was first documented among captive mule deer in Colorado in 1967, and has been detected in 24 states and two Canadian provinces. It’s been found in the wild in 20 states and among captive cervids in 15 states.
The Commission has taken several steps to prevent the disease, which strikes cervids (deer, elk and moose), from entering the state. A moratorium on live cervid importation began in 2002, and the importation of cervid carcasses was banned in 2005. Moratoriums on permits for commercial hunting resorts and breeder/dealer permits for cervid facilities were put in place in 2006. Capturing white-tailed deer by hand was banned in 2012.
According to the CWD Alliance, the disease was discovered among captive mule deer in Colorado in 1967, and has been detected in 24 other states and two Canadian provinces. Biologists believe a protein particle called a prion is transmitted through feces, urine and saliva, and can survive for years in soil and plants. CWD can have an incubation period of at least 16 months, which means infected animals may not show symptoms immediately.
CWD affects an animal’s nervous system. Prions transform normal cellular proteins into abnormal shapes that accumulate until neural cells cease to function. Infected animals begin to lose weight, lose their appetite and develop an insatiable thirst. They tend to separate from their herds, walk in repetitive patterns, carry their head low, salivate, urinate frequently and grind their teeth.
Visit ArkansasCWD.com for more information.
WEDNESDAY, DECEMBER 06, 2017
DEFRA U.K. What is the risk of Chronic Wasting Disease CWD being introduced into Great Britain? A Qualitative Risk Assessment October 2012
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In the USA, under the Food and Drug Administration's BSE Feed Regulation (21 CFR 589.2000) most material (exceptions include milk, tallow, and gelatin) from deer and elk is prohibited for use in feed for ruminant animals. With regards to feed for non-ruminant animals, under FDA law, CWD positive deer may not be used for any animal feed or feed ingredients. For elk and deer considered at high risk for CWD, the FDA recommends that these animals do not enter the animal feed system. However, this recommendation is guidance and not a requirement by law.
Animals considered at high risk for CWD include:
1) animals from areas declared to be endemic for CWD and/or to be CWD eradication zones and
2) deer and elk that at some time during the 60-month period prior to slaughter were in a captive herd that contained a CWD-positive animal.
Therefore, in the USA, materials from cervids other than CWD positive animals may be used in animal feed and feed ingredients for non-ruminants.
The amount of animal PAP that is of deer and/or elk origin imported from the USA to GB can not be determined, however, as it is not specified in TRACES. It may constitute a small percentage of the 8412 kilos of non-fish origin processed animal proteins that were imported from US into GB in 2011.
Overall, therefore, it is considered there is a __greater than negligible risk___ that (nonruminant) animal feed and pet food containing deer and/or elk protein is imported into GB.
There is uncertainty associated with this estimate given the lack of data on the amount of deer and/or elk protein possibly being imported in these products.
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36% in 2007 (Almberg et al., 2011). In such areas, population declines of deer of up to 30 to 50% have been observed (Almberg et al., 2011). In areas of Colorado, the prevalence can be as high as 30% (EFSA, 2011).
The clinical signs of CWD in affected adults are weight loss and behavioural changes that can span weeks or months (Williams, 2005). In addition, signs might include excessive salivation, behavioural alterations including a fixed stare and changes in interaction with other animals in the herd, and an altered stance (Williams, 2005). These signs are indistinguishable from cervids experimentally infected with bovine spongiform encephalopathy (BSE).
Given this, if CWD was to be introduced into countries with BSE such as GB, for example, infected deer populations would need to be tested to differentiate if they were infected with CWD or BSE to minimise the risk of BSE entering the human food-chain via affected venison.
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The rate of transmission of CWD has been reported to be as high as 30% and can approach 100% among captive animals in endemic areas (Safar et al., 2008).
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In summary, in endemic areas, there is a medium probability that the soil and surrounding environment is contaminated with CWD prions and in a bioavailable form. In rural areas where CWD has not been reported and deer are present, there is a greater than negligible risk the soil is contaminated with CWD prion.
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In summary, given the volume of tourists, hunters and servicemen moving between GB and North America, the probability of at least one person travelling to/from a CWD affected area and, in doing so, contaminating their clothing, footwear and/or equipment prior to arriving in GB is greater than negligible. For deer hunters, specifically, the risk is likely to be greater given the increased contact with deer and their environment. However, there is significant uncertainty associated with these estimates.
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Therefore, it is considered that farmed and park deer may have a higher probability of exposure to CWD transferred to the environment than wild deer given the restricted habitat range and higher frequency of contact with tourists and returning GB residents.
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http://www.defra.gov.uk/animal-diseases/files/qra_chronic-wasting-disease-121029.pdf
TSS
Singeltary submission ;
Program Standards: Chronic Wasting Disease Herd Certification Program and Interstate Movement of Farmed or Captive Deer, Elk, and Moose
*** DOCUMENT ID: APHIS-2006-0118-0411
http://www.regulations.gov/#!documentDetail;D=APHIS-2006-0118-0411
http://chronic-wasting-disease.blogspot.com/2014/03/docket-no-00-108-10-chronic-wasting.html
Some unofficial information from a source on the inside looking out -
Confidential!!!!
As early as 1992-3 there had been long studies conducted on small pastures containing scrapie infected sheep at the sheep research station associated with the Neuropathogenesis Unit in Edinburgh, Scotland. Whether these are documented...I don't know. But personal recounts both heard and recorded in a daily journal indicate that leaving the pastures free and replacing the topsoil completely at least 2 feet of thickness each year for SEVEN years....and then when very clean (proven scrapie free) sheep were placed on these small pastures.... the new sheep also broke out with scrapie and passed it to offspring. I am not sure that TSE contaminated ground could ever be free of the agent!! A very frightening revelation!!!
---end personal email---end...tss
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