Tuesday, June 11, 2013

CWD GONE WILD, More cervid escapees from more shooting pens on the loose in Pennsylvania

CWD GONE WILD, More cervid escapees from more shooting pens on the loose in Pennsylvania

Deer mystery yielding clues

Source of animal shot in county park here under investigation Intelligencer Journal Lancaster New Era Updated Jun 10, 2013 18:00


By AD CRABLE Staff Writer acrable@lnpnews.com

Pennsylvania Game Commission officials are investigating the source of an unusual deer that they ordered shot in Lancaster County Central Park.

The deer, with a black tail, black behind its ears and numbered tags in both ears, was seen regularly by park staff this past winter, traveling with the park's white-tailed deer herd.

Dennis Warfel, a PGC wildlife conservation officer in Lancaster County, thinks the deer could be a captive Sitka blacktailed deer native to parts of Alaska and British Columbia, or a white-tailed deer in a rare melanistic phase — a state of dark pigmentation.

Park rangers, knowing the current concerns about escaped deer from private deer farms possibly spreading the dreaded deer disease chronic wasting disease, notified the PGC.

The PGC watched the deer, which appeared healthy.

Agency policies dictate that time be given to allow the owner of an escaped deer to notify the state Department of Agriculture — where pen-raised deer must be registered — and for an owner to recover the animal, Warfel noted.

The PGC, after providing that time, ordered that the animal be shot.

"People are hypersensitive right now about it," Warfel said, referring to the CWD scare.

A city police officer shot the deer Feb. 21. A sample of its brain was sent to a state lab, where it tested negative for CWD.

Using the ear tags, the agency has been trying to determine the source of the deer.

The investigation is close to being concluded, but "it is not clear in whose possession the deer was when it escaped," Walt Cottrell, PGC wildlife veterinarian, said Monday.

"It's clear this is an escaped captive deer," Cottrell said. "All over the state, including in Lancaster County, the Game Commission is following up on these deer because they do represent a risk factor, and this is one example."

Warfel said an escaped deer is "more of a violation on the ag department's side because we don't control the deer. We want to make sure the containment area (where the deer came from) is suitable to keep the deer in."

Deer farms are a popular side industry in Lancaster County, especially among Plain sects. Deer are raised mainly to be sold to private hunting preserves.

Concerns over captive deer escalated last year when two deer with CWD were found on a deer farm in Adams County. The deer had also spent time at a York County deer farm. Another deer escaped from the Adams County site but it was later shot and found not to have CWD.

The PGC set up a disease-management area in York and Adams counties, and 2,000 deer shot there during hunting seasons were tested. None had CWD.

Also last fall, two wild deer shot during hunting seasons in Blair County tested positive for CWD, as well as one in Butler County.

Game officials think the arrival of CWD in western Pennsylvania may be from the migration of wild deer from nearby Maryland and West Virginia.

No connection has been found between the captive deer with CWD in Adams County and the CWD deer in the western part of the state.

Since the discovery of CWD in deer in both areas, the PGC has issued an executive order forbidding feeding and rehabilitation of deer in the areas, as well as other controls.

The agriculture department quarantined 29 deer farms in 16 counties associated with the Adams and York County captive deer. None was in Lancaster County.

CWD, which is always fatal to deer once they contract it, is now found in 23 states. In 13 of those states, the disease is found only in captive deer herds.

There are 1,100 registered captive deer farms in Pennsylvania, with about 23,000 deer fenced in.

Tuesday, October 23, 2012

PA Captive deer from CWD-positive farm roaming free

Tuesday, May 28, 2013

Chronic Wasting Disease CWD quarantine Louisiana via CWD index herd Pennsylvania Update May 28, 2013

6 doe from Pennsylvania CWD index herd still on the loose in Louisiana, quarantine began on October 18, 2012, still ongoing, Lake Charles premises.

Wednesday, November 14, 2012


Monday, April 15, 2013

Deer farmers in the state of Louisiana are under a quarantine due to Chronic Wasting Disease CWD

Tuesday, October 23, 2012

PA Captive deer from CWD-positive farm roaming free

> Ag is one of the agencies cooperating in the response plan because it has responsibility for regulating captive deer and deer farms, of which there are estimated to be more 23,000 on 1,100 Pennsylvania properties.

Tuesday, November 06, 2012

PA Department of Agriculture investigating possible 2nd case of chronic wasting disease

Thursday, November 01, 2012


Friday, October 26, 2012


Monday, October 15, 2012


Commissioner Strain Sir, I believe you can see the history here, where the state of Pennsylvania DNR forum banned me for speaking about CWD back in 2005 ;

Thursday, October 11, 2012

Pennsylvania Confirms First Case CWD Adams County Captive Deer Tests Positive

Pennsylvania CWD number of deer exposed and farms there from much greater than first thought

Published: Wednesday, October 17, 2012, 10:44 PM Updated: Wednesday, October 17, 2012, 11:33 PM

Sunday, June 09, 2013

Missouri House forms 13-member Interim Committee on the Cause and Spread of Chronic Wasting Disease CWD


I write you about the interim House committee that has been appointed to investigate Chronic Wasting Disease CWD, and the 13-member Interim Committee on the Cause and Spread of Chronic Wasting Disease.

Chronic Wasting Disease CWD, and other TSE prion disease, these TSE prions know no borders.

these TSE prions know no age restrictions.

The TSE prion disease survives ashing to 600 degrees celsius, that’s around 1112 degrees farenheit.

you cannot cook the TSE prion disease out of meat.

you can take the ash and mix it with saline and inject that ash into a mouse, and the mouse will go down with TSE.

Prion Infected Meat-and-Bone Meal Is Still Infectious after Biodiesel Production as well.

the TSE prion agent also survives Simulated Wastewater Treatment Processes.

IN fact, you should also know that the TSE Prion agent will survive in the environment for years, if not decades.

you can bury it and it will not go away.

The TSE agent is capable of infected your water table i.e. Detection of protease-resistant cervid prion protein in water from a CWD-endemic area.

it’s not your ordinary pathogen you can just cook it out and be done with. that’s what’s so worrisome about Iatrogenic mode of transmission, a simple autoclave will not kill this TSE prion agent.

I go from state to state trying to warn of the CWD and other TSE prion disease in other species, I just made a promise to mom. back then, there was no information.

so, I submit this to you all in good faith, and hope that you take the time to read my research of the _sound_, peer review science, not the junk science that goes with the politics $$$

right or left or teaparty or independent, you cannot escape the TSE prion disease.

there is a lot of science here to digest, but better digesting this _sound_ science, instead of the junk political science you will hear from the shooting pen industry.

snip...see full text of my submission here ;

Tuesday, April 16, 2013

Cervid Industry Unites To Set Direction for CWD Reform and seem to ignore their ignorance and denial in their role in spreading Chronic Wasting Disease

pens, pens, PENS ???

*** Spraker suggested an interesting explanation for the occurrence of CWD. The deer pens at the Foot Hills Campus were built some 30-40 years ago by a Dr. Bob Davis. At or abut that time, allegedly, some scrapie work was conducted at this site. When deer were introduced to the pens they occupied ground that had previously been occupied by sheep.

now, decades later ;


PO-039: A comparison of scrapie and chronic wasting disease in white-tailed deer


After a natural route of exposure, 100% of WTD were susceptible to scrapie. Deer developed clinical signs of wasting and mental depression and were necropsied from 28 to 33 months PI. Tissues from these deer were positive for PrPSc by IHC and WB. Similar to IC inoculated deer, samples from these deer exhibited two different molecular profiles: samples from obex resembled CWD whereas those from cerebrum were similar to the original scrapie inoculum. On further examination by WB using a panel of antibodies, the tissues from deer with scrapie exhibit properties differing from tissues either from sheep with scrapie or WTD with CWD. Samples from WTD with CWD or sheep with scrapie are strongly immunoreactive when probed with mAb P4, however, samples from WTD with scrapie are only weakly immunoreactive. In contrast, when probed with mAb’s 6H4 or SAF 84, samples from sheep with scrapie and WTD with CWD are weakly immunoreactive and samples from WTD with scrapie are strongly positive. This work demonstrates that WTD are highly susceptible to sheep scrapie, but on first passage, scrapie in WTD is differentiable from CWD.


*** After a natural route of exposure, 100% of white-tailed deer were susceptible to scrapie.

Scrapie in Deer: Comparisons and Contrasts to Chronic Wasting Disease (CWD)

Justin J. Greenlee of the Virus and Prion Diseases Research Unit, National Animal Disease Center, ARS, USDA, Ames, IA provided a presentation on scrapie and CWD in inoculated deer. Interspecies transmission studies afford the opportunity

After a natural route of exposure, 100% of white-tailed deer were susceptible to scrapie. Deer developed clinical signs of wasting and mental depression and were necropsied from 28 to 33 months PI. Tissues from these deer were positive for scrapie by IHC and WB. Tissues with PrPSc immunoreactivity included brain, tonsil, retropharyngeal and mesenteric lymph nodes, hemal node, Peyer’s patches, and spleen. While two WB patterns have been detected in brain regions of deer inoculated by the natural route, unlike the IC inoculated deer, the pattern similar to the scrapie inoculum predominates.

2011 Annual Report


In Objective 1, Assess cross-species transmissibility of transmissible spongiform encephalopathies (TSEs) in livestock and wildlife, numerous experiments assessing the susceptibility of various TSEs in different host species were conducted. Most notable is deer inoculated with scrapie, which exhibits similarities to chronic wasting disease (CWD) in deer suggestive of sheep scrapie as an origin of CWD.


4.Accomplishments 1. Deer inoculated with domestic isolates of sheep scrapie. Scrapie-affected deer exhibit 2 different patterns of disease associated prion protein. In some regions of the brain the pattern is much like that observed for scrapie, while in others it is more like chronic wasting disease (CWD), the transmissible spongiform encephalopathy typically associated with deer. This work conducted by ARS scientists at the National Animal Disease Center, Ames, IA suggests that an interspecies transmission of sheep scrapie to deer may have been the origin of CWD. This is important for husbandry practices with both captive deer, elk and sheep for farmers and ranchers attempting to keep their herds and flocks free of CWD and scrapie.

White-tailed Deer are Susceptible to Scrapie by Natural Route of Infection


This work demonstrates for the first time that white-tailed deer are susceptible to sheep scrapie by potential natural routes of inoculation. In-depth analysis of tissues will be done to determine similarities between scrapie in deer after intracranial and oral/intranasal inoculation and chronic wasting disease resulting from similar routes of inoculation.

see full text ;

*** The potential impact of prion diseases on human health was greatly magnified by the recognition that interspecies transfer of BSE to humans by beef ingestion resulted in vCJD. While changes in animal feed constituents and slaughter practices appear to have curtailed vCJD, there is concern that CWD of free-ranging deer and elk in the U.S. might also cross the species barrier. Thus, consuming venison could be a source of human prion disease. Whether BSE and CWD represent interspecies scrapie transfer or are newly arisen prion diseases is unknown. Therefore, the possibility of transmission of prion disease through other food animals cannot be ruled out. There is evidence that vCJD can be transmitted through blood transfusion. There is likely a pool of unknown size of asymptomatic individuals infected with vCJD, and there may be asymptomatic individuals infected with the CWD equivalent. These circumstances represent a potential threat to blood, blood products, and plasma supplies.

Friday, November 09, 2012

*** Chronic Wasting Disease CWD in cervidae and transmission to other species

Sunday, November 11, 2012

*** Susceptibilities of Nonhuman Primates to Chronic Wasting Disease November 2012

Friday, December 14, 2012

Susceptibility Chronic Wasting Disease (CWD) in wild cervids to Humans 2005 - December 14, 2012

Saturday, March 09, 2013

Chronic Wasting Disease in Bank Voles: Characterisation of the Shortest Incubation Time Model for Prion Diseases



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