Friday, March 10, 2017

Nebraska Tests confirm spread of CWD to Lancaster County

Subject: Nebraska Tests confirm spread of CWD to Lancaster County

Tests confirm spread of CWD to Lancaster County

LINCOLN, Neb. – A sick deer in Lancaster County tested positive for chronic wasting disease (CWD), a fatal neurological disease, in early March. This is the first known case of CWD in Lancaster County. 

CWD is a degenerative disease that attacks the brain of an infected deer or elk, eventually causing emaciation, listlessness and death. It is generally thought that CWD is transmitted from animal to animal through the exchange of bodily fluids, but other modes of transmission may exist. CWD has been found in 35 Nebraska counties, but no population declines attributable to the disease have yet occurred.

Help limit the spread of CWD by refraining from feeding deer and elk. This practice is not recommended and increases the risk of disease transmission.

Hunters are advised to dress animals at the place of kill, avoid spreading spinal cord or brain tissue to meat, and dispose of the head (brain), spinal column and other bones at a licensed landfill.

Learn more about CWD at Please direct any reports of sick deer to the nearest Game and Parks office.


Wednesday, January 11, 2017 

Nebraska Four positives for CWD found in recent testing of deer 



The TSE prion disease survives ashing to 600 degrees celsius, that’s around 1112 degrees farenheit.
you cannot cook the TSE prion disease out of meat. 

you can take the ash and mix it with saline and inject that ash into a mouse, and the mouse will go down with TSE.
Prion Infected Meat-and-Bone Meal Is Still Infectious after Biodiesel Production as well.
the TSE prion agent also survives Simulated Wastewater Treatment Processes.
IN fact, you should also know that the TSE Prion agent will survive in the environment for years, if not decades.
you can bury it and it will not go away.
The TSE agent is capable of infected your water table i.e. Detection of protease-resistant cervid prion protein in water from a CWD-endemic area.
it’s not your ordinary pathogen you can just cook it out and be done with.
that’s what’s so worrisome about Iatrogenic mode of transmission, a simple autoclave will not kill this TSE prion agent.

Title: Pathological features of chronic wasting disease in reindeer and demonstration of horizontal transmission
Using in vitro prion replication for high sensitive detection of prions and prionlike proteins and for understanding mechanisms of transmission.
Claudio Soto
Mitchell Center for Alzheimer's diseases and related Brain disorders, Department of Neurology, University of Texas Medical School at Houston.
***Recently, we have been using PMCA to study the role of environmental prion contamination on the horizontal spreading of TSEs. These experiments have focused on the study of the interaction of prions with plants and environmentally relevant surfaces. Our results show that plants (both leaves and roots) bind tightly to prions present in brain extracts and excreta (urine and feces) and retain even small quantities of PrPSc for long periods of time. Strikingly, ingestion of prioncontaminated leaves and roots produced disease with a 100% attack rate and an incubation period not substantially longer than feeding animals directly with scrapie brain homogenate. Furthermore, plants can uptake prions from contaminated soil and transport them to different parts of the plant tissue (stem and leaves). Similarly, prions bind tightly to a variety of environmentally relevant surfaces, including stones, wood, metals, plastic, glass, cement, etc. Prion contaminated surfaces efficiently transmit prion disease when these materials were directly injected into the brain of animals and strikingly when the contaminated surfaces were just placed in the animal cage. These findings demonstrate that environmental materials can efficiently bind infectious prions and act as carriers of infectivity, suggesting that they may play an important role in the horizontal transmission of the disease.

In summary, given the volume of tourists, hunters and servicemen moving between GB and North America, the probability of at least one person travelling to/from a CWD affected area and, in doing so, contaminating their clothing, footwear and/or equipment prior to arriving in GB is greater than negligible. For deer hunters, specifically, the risk is likely to be greater given the increased contact with deer and their environment. However, there is significant uncertainty associated with these estimates.


Therefore, it is considered that farmed and park deer may have a higher probability of exposure to CWD transferred to the environment than wild deer given the restricted habitat range and higher frequency of contact with tourists and returning GB residents.


What is the risk of chronic wasting disease being introduced into Great Britain? A Qualitative Risk Assessment October 2012

Thursday, April 07, 2016

What is the risk of chronic wasting disease being introduced into Great Britain? An updated Qualitative Risk Assessment March 2016

Subject: DEFRA What is the risk of a cervid TSE being introduced from Norway into Great Britain? Qualitative Risk Assessment September 2016

Friday, September 30, 2016

DEFRA What is the risk of a cervid TSE being introduced from Norway into Great Britain? Qualitative Risk Assessment September 2016

Scientific Opinion

Chronic wasting disease (CWD) in cervids


EFSA Panel on Biological Hazards (BIOHAZ),

First published: 18 January 2017Full publication history
DOI: 10.2903/j.efsa.2017.4667View/save citation

Prions in Skeletal Muscles of Deer with Chronic Wasting Disease
Rachel C. Angers1,*, Shawn R. Browning1,*,†, Tanya S. Seward2, Christina J. Sigurdson4,‡, Michael W. Miller5, Edward A. Hoover4, Glenn C. Telling1,2,3,§ snip...
Abstract The emergence of chronic wasting disease (CWD) in deer and elk in an increasingly wide geographic area, as well as the interspecies transmission of bovine spongiform encephalopathy to humans in the form of variant Creutzfeldt Jakob disease, have raised concerns about the zoonotic potential of CWD. Because meat consumption is the most likely means of exposure, it is important to determine whether skeletal muscle of diseased cervids contains prion infectivity.
***Here bioassays in transgenic mice expressing cervid prion protein revealed the presence of infectious prions in skeletal muscles of CWD-infected deer, demonstrating that humans consuming or handling meat from CWD-infected deer are at risk to prion exposure.
Cervid to human prion transmission
Kong, Qingzhong
Case Western Reserve University, Cleveland, OH, United States
Prion disease is transmissible and invariably fatal. Chronic wasting disease (CWD) is the prion disease affecting deer, elk and moose, and it is a widespread and expanding epidemic affecting 22 US States and 2 Canadian provinces so far.
*** CWD poses the most serious zoonotic prion transmission risks in North America because of huge venison consumption (>6 million deer/elk hunted and consumed annually in the USA alone), significant prion infectivity in muscles and other tissues/fluids from CWD-affected cervids, and usually high levels of individual exposure to CWD resulting from consumption of the affected animal among often just family and friends. However, we still do not know whether CWD prions can infect humans in the brain or peripheral tissues or whether clinical/asymptomatic CWD zoonosis has already occurred, and we have no essays to reliably detect CWD infection in humans. We hypothesize that:
(1) The classic CWD prion strain can infect humans at low levels in the brain and peripheral lymphoid tissues;
(2) The cervid-to-human transmission barrier is dependent on the cervid prion strain and influenced by the host (human) prion protein (PrP) primary sequence;
(3) Reliable essays can be established to detect CWD infection in humans;and
(4) CWD transmission to humans has already occurred. We will test these hypotheses in 4 Aims using transgenic (Tg) mouse models and complementary in vitro approaches.
Aim 1 will prove that the classical CWD strain may infect humans in brain or peripheral lymphoid tissues at low levels by conducting systemic bioassays in a set of "humanized" Tg mouse lines expressing common human PrP variants using a number of CWD isolates at varying doses and routes. Experimental "human CWD" samples will also be generated for Aim 3.
Aim 2 will test the hypothesis that the cervid-to-human prion transmission barrier is dependent on prion strain and influenced by the host (human) PrP sequence by examining and comparing the transmission efficiency and phenotypes of several atypical/unusual CWD isolates/strains as well as a few prion strains from other species that have adapted to cervid PrP sequence, utilizing the same panel of humanized Tg mouse lines as in Aim 1.
Aim 3 will establish reliable essays for detection and surveillance of CWD infection in humans by examining in details the clinical, pathological, biochemical and in vitro seeding properties of existing and future experimental "human CWD" samples generated from Aims 1-2 and compare them with those of common sporadic human Creutzfeldt-Jakob disease (sCJD) prions.
Aim 4 will attempt to detect clinical CWD-affected human cases by examining a significant number of brain samples from prion-affected human subjects in the USA and Canada who have consumed venison from CWD-endemic areas utilizing the criteria and essays established in Aim 3. The findings from this proposal will greatly advance our understandings on the potential and characteristics of cervid prion transmission in humans, establish reliable essays for CWD zoonosis and potentially discover the first case(s) of CWD infection in humans.
Public Health Relevance There are significant and increasing human exposure to cervid prions because chronic wasting disease (CWD, a widespread and highly infectious prion disease among deer and elk in North America) continues spreading and consumption of venison remains popular, but our understanding on cervid-to-human prion transmission is still very limited, raising public health concerns. This proposal aims to define the zoonotic risks of cervid prions and set up and apply essays to detect CWD zoonosis using mouse models and in vitro methods. The findings will greatly expand our knowledge on the potentials and characteristics of cervid prion transmission in humans, establish reliable essays for such infections and may discover the first case(s) of CWD infection in humans.
Funding Agency Agency National Institute of Health (NIH)
Institute National Institute of Neurological Disorders and Stroke (NINDS)
Pathological Prion Protein (PrPTSE) in Skeletal Muscles of Farmed and Free Ranging White-Tailed Deer Infected with Chronic Wasting Disease
***The presence and seeding activity of PrPTSE in skeletal muscle from CWD-infected cervids suggests prevention of such tissue in the human diet as a precautionary measure for food safety, pending on further clarification of whether CWD may be transmissible to humans.
***In contrast, cattle are highly susceptible to white-tailed deer CWD and mule deer CWD in experimental conditions but no natural CWD infections in cattle have been reported (Sigurdson, 2008; Hamir et al., 2006). It is not known how susceptible humans are to CWD but given that the prion can be present in muscle, it is likely that humans have been exposed to the agent via consumption of venison (Sigurdson, 2008). Initial experimental research, however, suggests that human susceptibility to CWD is low and there may be a robust species barrier for CWD transmission to humans (Sigurdson, 2008). It is apparent, though, that CWD is affecting wild and farmed cervid populations in endemic areas with some deer populations decreasing as a result.
Technical Abstract:
***Cattle could be exposed to the agent of chronic wasting disease (CWD) through contact with infected farmed or free-ranging cervids or exposure to contaminated premises. The purpose of this study was to assess the potential for CWD derived from elk to transmit to cattle after intracranial inoculation. Calves (n=14) were inoculated with brain homogenate derived from elk with CWD to determine the potential for transmission and define the clinicopathologic features of disease.
Cattle were necropsied if clinical signs occurred or at the termination of experiment (49 months post-inoculation (MPI)).
Clinical signs of poor appetite, weight loss, circling, and bruxism occurred in two cattle (14%) at 16 and 17 MPI, respectively.
Accumulation of abnormal prion protein (PrP**Sc) in these cattle was confined to the central nervous system with the most prominent immunoreactivity in midbrain, brainstem, and hippocampus with lesser immunoreactivity in the cervical spinal cord.
*** The rate of transmission was lower than in cattle inoculated with CWD derived from mule deer (38%) or white-tailed deer (86%).
Additional studies are required to fully assess the potential for cattle to develop CWD through a more natural route of exposure, but a low rate of transmission after intracranial inoculation suggests that risk of transmission through other routes is low.
***A critical finding here is that if CWD did transmit to exposed cattle, currently used diagnostic techniques would detect and differentiate it from other prion diseases in cattle based on absence of spongiform change, distinct pattern of PrP**Sc deposition, and unique molecular profile.
Monday, April 04, 2016
*** Limited amplification of chronic wasting disease prions in the peripheral tissues of intracerebrally inoculated cattle ***
Monday, May 02, 2016
*** Zoonotic Potential of CWD Prions: An Update Prion 2016 Tokyo ***
Exotic Meats USA Announces Urgent Statewide Recall of Elk Tenderloin Because It May Contain Meat Derived From An Elk Confirmed To Have Chronic Wasting Disease
Contact: Exotic Meats USA 1-800-680-4375
FOR IMMEDIATE RELEASE -- February 9, 2009 -- Exotic Meats USA of San Antonio, TX is initiating a voluntary recall of Elk Tenderloin because it may contain meat derived from an elk confirmed to have Chronic Wasting Disease (CWD). The meat with production dates of December 29, 30 and 31, 2008 was purchased from Sierra Meat Company in Reno, NV. The infected elk came from Elk Farm LLC in Pine Island, MN and was among animals slaughtered and processed at USDA facility Noah’s Ark Processors LLC.
Chronic Wasting Disease (CWD) is a fatal brain and nervous system disease found in elk and deer. The disease is caused by an abnormally shaped protein called a prion, which can damage the brain and nerves of animals in the deer family. Currently, it is believed that the prion responsible for causing CWD in deer and elk is not capable of infecting humans who eat deer or elk contaminated with the prion, but the observation of animal-to-human transmission of other prion-mediated diseases, such as bovine spongiform encephalopathy (BSE), has raised a theoretical concern regarding the transmission of CWD from deer or elk to humans. At the present time, FDA believes the risk of becoming ill from eating CWD-positive elk or deer meat is remote. However, FDA strongly advises consumers to return the product to the place of purchase, rather than disposing of it themselves, due to environmental concerns.
Exotic Meats USA purchased 1 case of Elk Tenderloins weighing 16.9 lbs. The Elk Tenderloin was sold from January 16 – 27, 2009. The Elk Tenderloins was packaged in individual vacuum packs weighing approximately 3 pounds each. A total of six packs of the Elk Tenderloins were sold to the public at the Exotic Meats USA retail store. Consumers who still have the Elk Tenderloins should return the product to Exotic Meats USA at 1003 NE Loop 410, San Antonio, TX 78209. Customers with concerns or questions about the Voluntary Elk Recall can call 1-800-680-4375. The safety of our customer has always been and always will be our number one priority.
Exotic Meats USA requests that for those customers who have products with the production dates in question, do not consume or sell them and return them to the point of purchase. Customers should return the product to the vendor. The vendor should return it to the distributor and the distributor should work with the state to decide upon how best to dispose. If the consumer is disposing of the product he/she should consult with the local state EPA office.
*** These results would seem to suggest that CWD does indeed have zoonotic potential, at least as judged by the compatibility of CWD prions and their human PrPC target. Furthermore, extrapolation from this simple in vitro assay suggests that if zoonotic CWD occurred, it would most likely effect those of the PRNP codon 129-MM genotype and that the PrPres type would be similar to that found in the most common subtype of sCJD (MM1).***
*** We recently observed the direct transmission of a natural classical scrapie isolate to macaque after a 10-year silent incubation period,
***with features similar to some reported for human cases of sporadic CJD, albeit requiring fourfold long incubation than BSE. Scrapie, as recently evoked in humanized mice (Cassard, 2014),
***is the third potentially zoonotic PD (with BSE and L-type BSE),
***thus questioning the origin of human sporadic cases. We will present an updated panorama of our different transmission studies and discuss the implications of such extended incubation periods on risk assessment of animal PD for human health.
***thus questioning the origin of human sporadic cases***
***our findings suggest that possible transmission risk of H-type BSE to sheep and human. Bioassay will be required to determine whether the PMCA products are infectious to these animals.
*** In complement to the recent demonstration that humanized mice are susceptible to scrapie, we report here the first observation of direct transmission of a natural classical scrapie isolate to a macaque after a 10-year incubation period. Neuropathologic examination revealed all of the features of a prion disease: spongiform change, neuronal loss, and accumulation of PrPres throughout the CNS.
*** This observation strengthens the questioning of the harmlessness of scrapie to humans, at a time when protective measures for human and animal health are being dismantled and reduced as c-BSE is considered controlled and being eradicated.
*** Our results underscore the importance of precautionary and protective measures and the necessity for long-term experimental transmission studies to assess the zoonotic potential of other animal prion strains.
why do we not want to do TSE transmission studies on chimpanzees $
5. A positive result from a chimpanzee challenged severly would likely create alarm in some circles even if the result could not be interpreted for man. I have a view that all these agents could be transmitted provided a large enough dose by appropriate routes was given and the animals kept long enough. Until the mechanisms of the species barrier are more clearly understood it might be best to retain that hypothesis.
Transmission Studies
Mule deer transmissions of CWD were by intracerebral inoculation and compared with natural cases {the following was written but with a single line marked through it ''first passage (by this route)}...TSS
resulted in a more rapidly progressive clinical disease with repeated episodes of synocopy ending in coma. One control animal became affected, it is believed through contamination of inoculum (?saline). Further CWD transmissions were carried out by Dick Marsh into ferret, mink and squirrel monkey. Transmission occurred in ALL of these species with the shortest incubation period in the ferret.
Spongiform Encephalopathy in Captive Wild ZOO BSE INQUIRY
”The occurrence of CWD must be viewed against the contest of the locations in which it occurred. It was an incidental and unwelcome complication of the respective wildlife research programmes. Despite it’s subsequent recognition as a new disease of cervids, therefore justifying direct investigation, no specific research funding was forthcoming. The USDA veiwed it as a wildlife problem and consequently not their province!” page 26.
We inoculated cynomolgus macaques with serial dilutions of BSE-infected material. High dose-inoculated animals developed typical clinical disease with all the pathognomonic hallmarks, and incubation periods ranging from 3–8 years. Among low-dosed animals, some developed clinical signs with atypical patterns after extensive incubation periods, exhibiting lesion and biochemical profiles that differed sharply from the typical disease picture. Despite the presence of neurological signs and neuronal lesions, classical lesions of spongiosis and presence of cerebral PrPres were inconstant, or even absent. These observations suggest that low-dose exposure, which would have been the most frequent occurrence during the period of risk, could induce a non-typical pathology that may not be recognized as "prion disease."
link url not available, please see PRION 2011 ;
*** WDA 2016 NEW YORK ***
 We found that CWD adapts to a new host more readily than BSE and that human PrP was unexpectedly prone to misfolding by CWD prions. In addition, we investigated the role of specific regions of the bovine, deer and human PrP protein in resistance to conversion by prions from another species. We have concluded that the human protein has a region that confers unusual susceptibility to conversion by CWD prions.
 Student Presentations Session 2
 The species barriers and public health threat of CWD and BSE prions
 Ms. Kristen Davenport1, Dr. Davin Henderson1, Dr. Candace Mathiason1, Dr. Edward Hoover1 1Colorado State University
 Chronic wasting disease (CWD) is spreading rapidly through cervid populations in the USA. Bovine spongiform encephalopathy (BSE, mad cow disease) arose in the 1980s because cattle were fed recycled animal protein. These and other prion diseases are caused by abnormal folding of the normal prion protein (PrP) into a disease causing form (PrPd), which is pathogenic to nervous system cells and can cause subsequent PrP to misfold. CWD spreads among cervids very efficiently, but it has not yet infected humans. On the other hand, BSE was spread only when cattle consumed infected bovine or ovine tissue, but did infect humans and other species. The objective of this research is to understand the role of PrP structure in cross-species infection by CWD and BSE. To study the propensity of each species’ PrP to be induced to misfold by the presence of PrPd from verious species, we have used an in vitro system that permits detection of PrPd in real-time. We measured the conversion efficiency of various combinations of PrPd seeds and PrP substrate combinations. We observed the cross-species behavior of CWD and BSE, in addition to feline-adapted CWD and BSE. We found that CWD adapts to a new host more readily than BSE and that human PrP was unexpectedly prone to misfolding by CWD prions. In addition, we investigated the role of specific regions of the bovine, deer and human PrP protein in resistance to conversion by prions from another species. We have concluded that the human protein has a region that confers unusual susceptibility to conversion by CWD prions. CWD is unique among prion diseases in its rapid spread in natural populations. BSE prions are essentially unaltered upon passage to a new species, while CWD adapts to the new species. This adaptation has consequences for surveillance of humans exposed to CWD.
Wildlife Disease Risk Communication Research Contributes to Wildlife Trust Administration Exploring perceptions about chronic wasting disease risks among wildlife and agriculture professionals and stakeholders
Axis Deer and CWD ?
Many of the regulations or proposed regulations cover only species known to be susceptible to CWD; however, it is not known whether some farmed exotic cervid species in North America, such as fallow deer (Dama dama), sika deer (Cervus nippon), or axis deer (Axis axis), are susceptible to CWD.

Wednesday, December 21, 2016

Tuesday, August 30, 2016
Friday, January 29, 2016
NEBRASKA Three Positives for CWD Found in Recent Testing of Deer
Wednesday, January 25, 2012
*** Nebraska Fish and Game Association Censors Singeltary from speaking about Chronic Wasting Disease (CWD) again ***
*** Nebraska Fish and Game Association Censors Singeltary from speaking about Chronic Wasting Disease (CWD) again, what is Nebraska hiding ? ***
*** Elk and Deer Use of Mineral Licks: Implications for Disease Transmission  ***
01-19-2012, 06:58 PM Elk and Deer Use of Mineral Licks: Implications for Disease Transmission
Wednesday, January 04, 2012
Tuesday, April 24, 2012
There were 1,565 lymph node samples collected from deer taken during the 2011 November firearm deer season, with 26 samples testing positive for CWD. In addition, samples were taken from 37 culled deer that showed clinical symptoms for CWD, with one male mule deer from Garden County testing positive. Those symptoms include a rough, emaciated appearance and a lack of fear of humans.
There were a record 51 positives from 3,645 samples in Nebraska in 2010. However, the surveillance effort was reduced in 2011 due to a lack of funds. The 2011 effort focused on central Nebraska, the leading edge of the disease as it spreads from west to east.
Wednesday, January 25, 2012
Nebraska Fish and Game Association Censors Singeltary from speaking about Chronic Wasting Disease (CWD) again
NOW, let me be perfectly clear. this time, it was the Nebraska Fish and Game Association that allowed me back on board, to post about CWD, after I had asked them to do so. what happened was, I got to speaking the truth about game farms, and CWD spreading there from, and a certain few complained, and kept complaining, they did not want anymore information (valid scientific peer review journals) that might hurt their industry. SO, I thank NFGC again for giving me a chance to try and educate hunters on CWD and the TSE prion disease. I think I supplied enough information to help educate, the ones that wanted to be educated, however, it’s the other folks I am concerned about. the ones that don’t want to be educated on this CWD, the ones that don’t want to speak about it, or learn about, and they don’t want others to either. these few folks are the ones that will help continue the spread of CWD. these folks caused the surpressing of CWD TSE prion information. to be good stewards of the woods and hunt, you cannot stick your head in the sand. these few folks did, and in doing so, they want everyone else’s head in the sand. and that’s been the problem all along. ...good luck!
so much for freedom of speech. can’t say I did not try. ... TSS
Wednesday, March 02, 2011
Wednesday, February 04, 2009
Nebraska reports 22 cases of CWD in deer
Tuesday, December 18, 2007
NEBRASKA CWD tested 3,400 deer, with 17 testing positive 2007
Singeltary permanently banned from NEFGA for speaking about Chronic Wasting Disease CWD TSE Prion...see;
Monday, March 17, 2014
Greetings again Nebraska CWD officials et al,
I thought I should update you a bit on CWD TSE Prion. I hope you don’t mind. it’s been a while, so I thought it was time. lot going on.
I am still banned from posting on
you can see a bit of history here, and in the full history of me being banned in the link below in the post, there is another link.
Although the Game and Parks Commission's wildlife management areas and U.S. Forest Service pastures in the Bordeaux and Hat Creek units provide some opportunities for elk hunters, most elk taken in Nebraska are killed on private land. Obtaining private land access to hunt elk is difficult, but not impossible, and a growing number of Nebraska landowners charge fees for hunting privileges.
snip...see full text ;
The director and the moderators of this forum came together and voted to ban you from this forum. We had a lot of members complain about the way you wet about posting your threads.
I personally would like to say thanks for helping some of our members realize the importance of CWD and the affects. Thank you for your time.
Best regards, xxxx
First off, I would like to apologize for the harsh manner in which you have been greeted on this site. As you said, I'm sure you are used to it but that is no excuse. I know there have been problems in the past of people registering on this forum to simply blow their own horn and promote their own cause. One guy was trying to convince people common carp were the best game fish and threatened to stock them into every public body of water he could reach! Notwithstanding, the greeting you received was unnecessarily harsh and a poor representation of the majority of people on this site.
Regarding CWD, I am not too familiar with the disease but I do try to keep up on the current state it. I too am puzzled why the people with the largest interest in deer are so resistant to learn more about this major issue. It certainly seems like you know what you are talking about and as you have said you have spent many years learning about and researching this topic.
One small piece of advice I may offer you is to introduce yourself and give some background information about yourself. Where are you from? Do you hunt or fish? Why are you interested in CWD/TSE? Do you work professionally with this topic? Just some ideas. I understand you are trying to provide a large amount of information and are unable to post links to articles, but the large blocks of text pasted in your posts comes off as impersonal and abrasive to some, especially from a new member.
I hope you stick around, I am always eager to learn especially when the issue is something as large as this.
Regards xxxxxxx
I was waaaay too far down the food chain to know him! The only vets I knew were the IIC's (inspector in charge) if whatever establishment I was working at. With the game and parks pushing increased white tail doe harvest in our state as a result of politically motivated legislation (Senator Lautenbaugh's deer depredation bill) passed 2 years ago, your information and "sounding the alarm" is falling on deaf ears with those who have the clout to do anything about it. It's your right to do what you're doing, but in my opinion it's a waste of time. Where are you from, and do you eat the deer you kill?
You're proceeding on the wrong assumption. A little background info. would be in order. I'm a retired Federal Meat and Poultry Inspector for the USDA. I've educated myself on the CWD issue, and have made my decision in regards to the consumption (or lack thereof) of venison. You have obviously educated yourself on this issue, and are free to divulge your information in this forum. I honestly feel the vast majority of the members of this forum have already made their decision in regards to this matter; and it is my personal opinion you are, for lack of a better description, "beating a dead horse". Out of curiosity, have you had any contact with our states game and parks commission with your concerns, and if so, what has been their response? xxxx
*************** nobody's trolling here. i asked to come here to speak about CWD. CWD in Nebraska is mounting, it's spreading, and humans are being exposed to the CWD agent. if it was just the people that eat meat, were the only ones that were exposed, that is one thing. but this is about your cervids and environment, and, when you become exposed with the CWD agent, or any other TSE, and then have medical or dental or donate blood, you then expose others. so, it's just not about consumption. i knew there would be some that do not want to hear it, that's o.k., your entitled to your opinion, but others here may want to hear some of this, and try and do something to prevent the further exposure and spreading of the CWD TSE agent. i assure you i am here to promote awareness about CWD, to promote discussion and debate, nothing else. i am vested in nothing but the truth, and have been all along. i don't advertise on any of my blogs i post this CWD TSE science to. it's there for educational purposes. you need to educate yourself folks, with all the science. and yes, i am anti, i am anti stupid. i am anti corporate and political science. and the cut and paste are just facts, they have links. when i can post the links, the posts will be much shorter. but if it will make you feel better, i will go jump in a lake too, if you just become more aware to what you are dealing with i.e. CWD TSE prion disease. i had chicken last night, and a steak last week. oh, and yes somebody does just show up out of no where, from another state, and talk about CWD. i did, i do, and i have done it for years, from state to state as they fall with CWD, or any other TSE in a species. i was asked to be moderator of one State that fell, after being there for years, and i said the same thing, i can't moderate a state that i don't live in. but they freely accept the science and discussion and debate that comes from it. there are a few states that i have had trouble with, simply because they did not want to hear it, and did not want there hunters to hear it, because of the financial fallout. one of those states has recently let me back in to speak and share science about CWD. look, this is about more than money here, it's about your cervids, and your people. i have been in the pits for years doing this, debated folks and scientist all over the world, there is nothing you can say that will hurt my feelings. i am just the messenger folks, you can hate me, throw all the stones you wish, but read the science and educate yourself all you can. CWD and TSEs are here. they have even now linked the TSE agent to Alzheimer's, ALS, Parkinson's disease. and there is science now showing that Alzheimer's is transmissible. course there was science showing that a decade ago i.e. transmission studies. what does all this mean? i cannot answer that. i can tell you this, continue to ignore the CWD TSE agent and you risk your herd and people. shoot the messenger if you must, (i am full of holes), but don't ignore the science. ...
kind regards,



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