ACA Council Meets to Endorse Several Proposed USAHA Resolutions (CWD TSE PRION DISEASE)
ACA Council Meets to Endorse Several Proposed USAHA Resolutions
October 17, 2013
Resolutions to Reform CWD Standards, TB,
Brucellosis, and ACA Seeking USAHA Board Seat
The American Cervid Alliance Leadership Council met
Thursday, October 17, 2013, to discuss possible opportunities at the upcoming
United States Animal Health Association (USAHA) Conference this weekend in San
Diego. The ACA’s Moderator, Eric Mohlman, wanted all the alliance council
members to have an understanding of the agenda effecting our industry at USAHA
prior to the leader’s arrival.
Several ideas were discussed including how to strengthen
the cervid industry’s voice within USAHA and ways to decrease on-farm testing
requirements and general regulations. Several of these ideas will be put
forth, during the conference, in the form of resolutions and presented to the
Brucellosis, Tuberculosis, and Captive Wildlife and Alternative Livestock
Committees.
Eric Mohlman advised the Alliance that he has had several
discussions with key USAHA leaders about the possibly of approving the ACA as an
allied organization and board member of USAHA. Currently, three of the national
cervid associations, which include the North American Elk Breeders Association,
North American Deer Farmers Association, and Exotic Wildlife Association,
serve on the board of directors. The possibility of the ACA having its own
seat, as an allied organization representing thirty cervid associations, would
increase the presence of the industry voice on the national level. A motion was
made by Kim Kafka of the North American Elk Breeders Association, seconded
by Brian Wagner of the Minnesota Elk Breeders Association, to approve the ACA
moving forward to seek the USAHA Board seat. The motion was adopted
unanimously, 22-0. The Alliance nominated two council members to serve as the
interim representative for the ACA, if approved. The nominees were Eric Mohlman
and Laurie Seale. After the roll call vote, Laurie Seale was approved by a vote
of 13-7. Next year the ACA will conduct a nominating vetting process for the
2014 USAHA Conference representative.
Charly Seale, of the Exotic Wildlife Association, stated
that many of the cervid industry’s issues have, in many instances, been
minimized and lost in the all encompassing Captive Wildlife and
Alternative Livestock Committee. Seale said that committee discusses everything
from wild animals to elephants and the cervid industry needs its own committee
to focus solely on cervid farming issues. Seale has been invited to speak with
the USAHA Executive Committee this weekend to propose a new and separate farmed
cervid committee, which, if approved would begin at the 2014 conference. A motion was made, by Tim Condict of the Deer
Breeders Corporation, for the ACA Council to endorse the resolution. Curt
Waldvogel of the Second Ark Foundation, seconded the motion. The motion was
adopted 22-0.
Two resolutions were presented to the council, which will
ask USAHA to urge the USDA to withdraw the current existing CWD standards
document versions 1 through 22 and also ask for a new CWD Standards Working
Group to be created with cervid industry, state agriculture, and USDA
representatives, without wildlife officials, to redraft a new standards
documents and remove the old Version 1 document from their policy. Seale
stated, “We tried improving the Chronic Wasting Disease program standards
document 22 times over a sixth month period and it is not even close. Version 1(
July 2012), which according to USDA/ APHIS, the industry is operating under needs to be completely withdrawn because some states
are already using it and even Dr Klein admits was not their best effort.” A motion was made by Todd Landt, of the Iowa
Whitetail Deer Association, to ask the ACA to endorse the two resolutions when
presented to the Captive Wildlife and Alternative Livestock Committee. Kim Kafka
of the North American Elk Breeders Association seconded the motion. The motion was adopted 18-0, with one
association passing.
Two additional resolutions will be presented in the
Brucellosis and Tuberculosis Committees to ask for longer testing intervals for
TB reaccreditation and Brucellosis recertification. Two separate motions were
offered by Michael Heiter of the Pennsylvania Deer Farmers Association and Joel
Espe of the Wisconsin Commercial Deer and Elk Farmers Association, respectively,
for the ACA endorsement. Both motions
were unanimously adopted for both resolutions.
ACA Moderator Eric Mohlman thanked the council for their
input on the proposed resolutions. Mohlman, said, “Last year at USAHA, the
association leaders didn’t know what the others within the
industry were planning. Now all the thirty associations participating with the
ACA are all up to date on industry proposals before we get to the convention. We
are heading to San Diego as a team and united as an industry.”
IT seems
the USAHA is going to cater to the cervid industry now, and I pray that it does
not wind up being a broken industry fed bunch like the OIE/USDA, but things have
been looking that way recently in regards with animal TSE prion disease. seems
nothing has been science based of late, in my opinion. heck, from the looks of
it ;
===================================
Two resolutions were presented to the council, which will
ask USAHA to urge the USDA to withdraw the current existing CWD standards
document versions 1 through 22 and also ask for a new CWD Standards Working
Group to be created with cervid industry, state agriculture, and USDA
representatives, without wildlife officials, to redraft a new standards
documents and remove the old Version 1 document from their policy. Seale
stated, “We tried improving the Chronic Wasting Disease program standards
document 22 times over a sixth month period and it is not even close. Version 1(
July 2012), which according to USDA/ APHIS, the industry is operating under needs to be completely withdrawn because some states
are already using it and even Dr Klein admits was not their best effort.” A motion was made by Todd Landt, of the Iowa
Whitetail Deer Association, to ask the ACA to endorse the two resolutions when
presented to the Captive Wildlife and Alternative Livestock Committee. Kim Kafka
of the North American Elk Breeders Association seconded the motion. The motion was adopted 18-0, with one
association passing.
...
================================
hell, the USAHA should
just step aside and let the shooting pens write their own rules, it’s all big ag
now.
seems with the USDA all
aboard now with the captives now being livestock, it’s all over but the crying,
the wild as we knew it, and wild cervids there from, do not have a chance now, a
sad day for hunters in my opinion. ...tss
USDA TO PGC ONCE CAPTIVES ESCAPE "it‘s no longer its business.”
Sunday, January 06, 2013
USDA TO PGC ONCE CAPTIVES ESCAPE "it‘s no longer its business.”
Indiana State to appeal high-fence deer hunting ruling
TAHC ADOPTS CWD RULE THAT the amendments ''remove'' the requirement for a specific fence height for captives Texas Animal Health Commission (TAHC) ANNOUNCEMENT
October 3, 2013
http://chronic-wasting-disease.blogspot.com/2013/10/tahc-adopts-cwd-rule-that-amendments.html
Wednesday, October 16, 2013
Pennsylvania Adjusts CWD Rules
PRION2013 CONGRESSIONAL ABSTRACTS CWD
Thursday, August 08, 2013
Characterization of the first case of
naturally occurring chronic wasting disease in a captive red deer (Cervus
elaphus) in North America
Sunday, September 01, 2013
hunting over gut piles and CWD TSE
prion disease
Wednesday, September 04, 2013
***cwd - cervid captive livestock
escapes, loose and on the run in the wild...
Monday, June 24, 2013
The Effects of Chronic Wasting
Disease on the Pennsylvania Cervid Industry Following its Discovery
Thursday, July 11, 2013
The New Hornographers: The Fight
Over the Future of Texas Deer, Captive shooting pens, and the CWD TSE prion
disease
Tuesday, July 02, 2013
National Rifle Association and the
Unified Sportsman of Florida support a Florida ban on the importation of captive
deer and cervids into Florida
Sunday, June 09, 2013
Missouri House forms 13-member
Interim Committee on the Cause and Spread of Chronic Wasting Disease CWD
Tuesday, April 16, 2013
Cervid Industry Unites To Set
Direction for CWD Reform and seem to ignore their ignorance and denial in their
role in spreading Chronic Wasting Disease
Monday, October 07, 2013
The importance of localized culling in
stabilizing chronic wasting disease prevalence in white-tailed deer populations
Saturday, February 04, 2012
Wisconsin 16 MONTH age limit on
testing dead deer Game Farm CWD Testing Protocol Needs To Be Revised
Tuesday, December 20, 2011
CHRONIC WASTING DISEASE CWD WISCONSIN
Almond Deer (Buckhorn Flats) Farm Update DECEMBER 2011
The CWD infection rate was nearly 80%,
the highest ever in a North American captive herd.
RECOMMENDATION: That the Board approve
the purchase of 80 acres of land for $465,000 for the Statewide Wildlife Habitat
Program in Portage County and approve the restrictions on public use of the
site.
SUMMARY:
recently, a report came out in the
U.K., about risk factors from entry of CWD from the USA. I think you might find
interest there ;
Friday, December 14, 2012
DEFRA U.K. What is the risk of Chronic
Wasting Disease CWD being introduced into Great Britain? A Qualitative Risk
Assessment October 2012
snip...
In the USA, under the Food and Drug
Administration’s BSE Feed Regulation (21 CFR 589.2000) most material (exceptions
include milk, tallow, and gelatin) from deer and elk is prohibited for use in
feed for ruminant animals. With regards to feed for non-ruminant animals, under
FDA law, CWD positive deer may not be used for any animal feed or feed
ingredients. For elk and deer considered at high risk for CWD, the FDA
recommends that these animals do not enter the animal feed system. However, this
recommendation is guidance and not a requirement by law.
Animals considered at high risk for
CWD include:
1) animals from areas declared to be
endemic for CWD and/or to be CWD eradication zones and
2) deer and elk that at some time
during the 60-month period prior to slaughter were in a captive herd that
contained a CWD-positive animal.
Therefore, in the USA, materials from
cervids other than CWD positive animals may be used in animal feed and feed
ingredients for non-ruminants.
The amount of animal PAP that is of
deer and/or elk origin imported from the USA to GB can not be determined,
however, as it is not specified in TRACES. It may constitute a small percentage
of the 8412 kilos of non-fish origin processed animal proteins that were
imported from US into GB in 2011.
Overall, therefore, it is considered
there is a __greater than negligible risk___ that (nonruminant) animal feed and
pet food containing deer and/or elk protein is imported into GB.
There is uncertainty associated with
this estimate given the lack of data on the amount of deer and/or elk protein
possibly being imported in these products.
snip...
36% in 2007 (Almberg et al., 2011). In
such areas, population declines of deer of up to 30 to 50% have been observed
(Almberg et al., 2011). In areas of Colorado, the prevalence can be as high as
30% (EFSA, 2011). The clinical signs of CWD in affected adults are weight loss
and behavioural changes that can span weeks or months (Williams, 2005). In
addition, signs might include excessive salivation, behavioural alterations
including a fixed stare and changes in interaction with other animals in the
herd, and an altered stance (Williams, 2005). These signs are indistinguishable
from cervids experimentally infected with bovine spongiform encephalopathy
(BSE). Given this, if CWD was to be introduced into countries with BSE such as
GB, for example, infected deer populations would need to be tested to
differentiate if they were infected with CWD or BSE to minimise the risk of BSE
entering the human food-chain via affected venison.
snip...
The rate of transmission of CWD has
been reported to be as high as 30% and can approach 100% among captive animals
in endemic areas (Safar et al., 2008).
snip...
In summary, in endemic areas, there is
a medium probability that the soil and surrounding environment is contaminated
with CWD prions and in a bioavailable form. In rural areas where CWD has not
been reported and deer are present, there is a greater than negligible risk the
soil is contaminated with CWD prion.
snip...
In summary, given the volume of
tourists, hunters and servicemen moving between GB and North America, the
probability of at least one person travelling to/from a CWD affected area and,
in doing so, contaminating their clothing, footwear and/or equipment prior to
arriving in GB is greater than negligible. For deer hunters, specifically, the
risk is likely to be greater given the increased contact with deer and their
environment. However, there is significant uncertainty associated with these
estimates.
snip...
Therefore, it is considered that
farmed and park deer may have a higher probability of exposure to CWD
transferred to the environment than wild deer given the restricted habitat range
and higher frequency of contact with tourists and returning GB residents.
snip...
SNIP...SEE ;
Friday, December 14, 2012
DEFRA U.K. What is the risk of Chronic
Wasting Disease CWD being introduced into Great Britain? A Qualitative Risk
Assessment October 2012
Wednesday, September 25, 2013
Inspections, Compliance, Enforcement,
and Criminal Investigations BSE TSE PRION 2013
DOCKET-- 03D-0186 -- FDA Issues Draft
Guidance on Use of Material From Deer and Elk in Animal Feed;
Availability
Date: Fri, 16 May 2003 11:47:37
–0500
EMC 1 Terry S. Singeltary Sr. Vol #:
1
PLEASE SEE FULL TEXT SUBMISSION
;
please see more on the potential of
the prion uptake from plants, and see what SEAC said about the one study that
showed prion uptake of the Tomato plant, although the test was never repeated
;
Furthermore, an unpublished study had
indicated low level absorption of PrP from soil by tomato plants although it
should be noted that this study had not been repeated. Details of this work
would be sent to the SEAC Secretary. Dr Matthews explained that most of the
manure from animals challenged with high doses of BSE had already been composted
and used for coppicing. Members agreed that the risks from disposal of residual
manure from experimental animals would be much less than historic risks of on
farm contamination from naturally infected animals at the height of the BSE
epidemic. ...SNIP...END
Uptake of Prions into Plants
Prion2013
Friday, August 09, 2013
***CWD TSE prion, plants, vegetables,
and the potential for environmental contamination
PRION2013 CONGRESSIONAL ABSTRACTS CWD
Sunday, August 25, 2013
HD.13: CWD infection in the spleen of humanized transgenic mice
Liuting Qing and Qingzhong Kong
Case Western Reserve University; Cleveland, OH USA
Chronic wasting disease (CWD) is a widespread prion disease in free-ranging
and captive cervid species in North America, and there is evidence suggesting
the existence of multiple CWD strains. The susceptibility of human CNS and
peripheral organs to the various CWD prion strains remains largely unclear.
Current literature suggests that the classical CWD strain is unlikely to infect
human brain, but the potential for peripheral infection by CWD in humans is
unknown. We detected protease-resistant PrpSc in the spleens of a few humanized
transgenic mice that were intracerebrally inoculated with natural CWD isolates,
but PrpSc was not detected in the brains of any of the CWD-inoculated mice. Our
ongoing bioassays in humanized Tg mice indicate that intracerebral challenge
with such PrpSc-positive humanized mouse spleen already led to prion disease in
most animals.
***These results indicate that the CWD prion may have the potential to
infect human peripheral lymphoid tissues.
Oral.15: Molecular barriers to zoonotic prion transmission: Comparison of
the ability of sheep, cattle and deer prion disease isolates to convert normal
human prion protein to its pathological isoform in a cell-free system
Marcelo A.Barria,1 Aru Balachandran,2 Masanori Morita,3 Tetsuyuki
Kitamoto,4 Rona Barron,5 Jean Manson,5 Richard Kniqht,1 James W. lronside1 and
Mark W. Head1
1National CJD Research and Surveillance Unit; Centre for Clinical Brain
Sciences; School of Clinical Sciences; The University of Edinburgh; Edinburgh,
UK; 2National and OIE Reference Laboratory for Scrapie and CWD; Canadian Food
Inspection Agency; Ottawa Laboratory; Fallowfield. ON Canada; 3Infectious
Pathogen Research Section; Central Research Laboratory; Japan Blood Products
Organization; Kobe, Japan; 4Department of Neurological Science; Tohoku
University Graduate School of Medicine; Sendai. Japan; 5Neurobiology Division;
The Roslin Institute and R(D)SVS; University of Edinburgh; Easter Bush;
Midlothian; Edinburgh, UK
Background. Bovine spongiform encephalopathy (BSE) is a known zoonotic
prion disease, resulting in variant Creurzfeldt- Jakob disease (vCJD) in humans.
In contrast, classical scrapie in sheep is thought to offer little or no danger
to human health. However, a widening range of prion diseases have been
recognized in cattle, sheep and deer. The risks posed by individual animal prion
diseases to human health cannot be determined a priori and are difficult to
assess empirically. The fundamemal event in prion disease pathogenesis is
thought to be the seeded conversion of normal prion protein (PrPC) to its
pathological isoform (PrPSc). Here we report the use of a rapid molecular
conversion assay to test whether brain specimens from different animal prion
diseases are capable of seeding the conversion of human PrPC ro PrPSc.
Material and Methods. Classical BSE (C-type BSE), H-type BSE, L-type BSE,
classical scrapie, atypical scrapie, chronic wasting disease and vCJD brain
homogenates were tested for their ability to seed conversion of human PrPC to
PrPSc in protein misfolding cyclic amplification (PMCA) reactions. Newly formed
human PrPSc was detected by protease digestion and western blotting using the
antibody 3F4.
Results. C-type BSE and vCJD were found to efficiently convert PrPC to
PrPSc. Scrapie failed to convert human PrPC to PrPSc. Of the other animal prion
diseases tested only chronic wasting disease appeared to have the capability ro
convert human PrPC to PrPSc. The results were consistent whether the human PrPC
came from human brain, humanised transgenic mouse brain or from cultured human
cells and the effect was more pronounced for PrPC with methionine at codon 129
compared with that with valine.
Conclusion. Our results show that none of the tested animal prion disease
isolates are as efficient as C-type BSE and vCJD in converting human prion
protein in this in vitro assay.
***However, they also show that there is no absolute barrier ro conversion
of human prion protein in the case of chronic wasting disease.
PRION2013 CONGRESSIONAL ABSTRACTS CWD
Sunday, August 25, 2013
***Chronic Wasting Disease CWD risk factors, humans, domestic cats, blood,
and mother to offspring transmission
Sunday, July 21, 2013
*** As Chronic Wasting Disease CWD rises in deer herd, what about risk for
humans?
Thursday, October 10, 2013
CJD REPORT 1994 increased risk for consumption of veal and venison and lamb
Tuesday, September 10, 2013
Review and Updates of the USDA-APHIS Veterinary Services (VS) National
Chronice Wasting Disease (CWD) Program 2012-2013
Creutzfeldt-Jakob Disease CJD cases rising North America updated report
August 2013
Creutzfeldt-Jakob Disease CJD cases rising North America with Canada seeing
an extreme increase of 48% between 2008 and 2010
Friday, August 16, 2013
*** Creutzfeldt-Jakob disease (CJD) biannual update August 2013 U.K. and
Contaminated blood products induce a highly atypical prion disease devoid of
PrPres in primates
Sunday, September 08, 2013
Iatrogenic Creutzfeldt-Jakob disease via surgical instruments and
decontamination possibilities for the TSE prion
Thursday, September 26, 2013
Minimise transmission risk of CJD and vCJD in healthcare settings Guidance
pens, pens, PENS ???
*** Spraker suggested an interesting explanation for the occurrence of CWD.
The deer pens at the Foot Hills Campus were built some 30-40 years ago by a Dr.
Bob Davis. At or abut that time, allegedly, some scrapie work was conducted at
this site. When deer were introduced to the pens they occupied ground that had
previously been occupied by sheep.
now, decades later ;
2012
PO-039: A comparison of scrapie and chronic wasting disease in white-tailed
deer
Justin Greenlee, Jodi Smith, Eric Nicholson US Dept. Agriculture;
Agricultural Research Service, National Animal Disease Center; Ames, IA USA
snip...
The results of this study suggest that there are many similarities in the
manifestation of CWD and scrapie in WTD after IC inoculation including early and
widespread presence of PrPSc in lymphoid tissues, clinical signs of depression
and weight loss progressing to wasting, and an incubation time of 21-23 months.
Moreover, western blots (WB) done on brain material from the obex region have a
molecular profile similar to CWD and distinct from tissues of the cerebrum or
the scrapie inoculum. However, results of microscopic and IHC examination
indicate that there are differences between the lesions expected in CWD and
those that occur in deer with scrapie: amyloid plaques were not noted in any
sections of brain examined from these deer and the pattern of immunoreactivity
by IHC was diffuse rather than plaque-like. After a natural route of exposure,
100% of WTD were susceptible to scrapie. Deer developed clinical signs of
wasting and mental depression and were necropsied from 28 to 33 months PI.
Tissues from these deer were positive for PrPSc by IHC and WB. Similar to IC
inoculated deer, samples from these deer exhibited two different molecular
profiles: samples from obex resembled CWD whereas those from cerebrum were
similar to the original scrapie inoculum. On further examination by WB using a
panel of antibodies, the tissues from deer with scrapie exhibit properties
differing from tissues either from sheep with scrapie or WTD with CWD. Samples
from WTD with CWD or sheep with scrapie are strongly immunoreactive when probed
with mAb P4, however, samples from WTD with scrapie are only weakly
immunoreactive. In contrast, when probed with mAb’s 6H4 or SAF 84, samples from
sheep with scrapie and WTD with CWD are weakly immunoreactive and samples from
WTD with scrapie are strongly positive. This work demonstrates that WTD are
highly susceptible to sheep scrapie, but on first passage, scrapie in WTD is
differentiable from CWD.
2011
*** After a natural route of exposure, 100% of white-tailed deer were
susceptible to scrapie.
Scrapie in Deer: Comparisons and Contrasts to Chronic Wasting Disease (CWD)
Justin J. Greenlee of the Virus and Prion Diseases Research Unit, National
Animal Disease Center, ARS, USDA, Ames, IA
snip...
This highlights the facts that 1) prior to the onset of clinical signs
PrPSc is widely distributed in the CNS and lymphoid tissues and 2) currently
used diagnostic methods are sufficient to detect PrPSc prior to the onset of
clinical signs. The results of this study suggest that there are many
similarities in the manifestation of CWD and scrapie in white-tailed deer after
IC inoculation including early and widespread presence of PrPSc in lymphoid
tissues, clinical signs of depression and weight loss progressing to wasting,
and an incubation time of 21-23 months. Moreover, western blots (WB) done on
brain material from the obex region have a molecular profile consistent with CWD
and distinct from tissues of the cerebrum or the scrapie inoculum. However,
results of microscopic and IHC examination indicate that there are differences
between the lesions expected in CWD and those that occur in deer with scrapie:
amyloid plaques were not noted in any sections of brain examined from these deer
and the pattern of immunoreactivity by IHC was diffuse rather than plaque-like.
After a natural route of exposure, 100% of white-tailed deer were susceptible to
scrapie. Deer developed clinical signs of wasting and mental depression and were
necropsied from 28 to 33 months PI. Tissues from these deer were positive for
scrapie by IHC and WB. Tissues with PrPSc immunoreactivity included brain,
tonsil, retropharyngeal and mesenteric lymph nodes, hemal node, Peyer’s patches,
and spleen. While two WB patterns have been detected in brain regions of deer
inoculated by the natural route, unlike the IC inoculated deer, the pattern
similar to the scrapie inoculum predominates.
2011 Annual Report
Research Project: TRANSMISSION, DIFFERENTIATION, AND PATHOBIOLOGY OF
TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHIES Location: Virus and Prion Research
Unit
2011 Annual Report
In Objective 1, Assess cross-species transmissibility of transmissible
spongiform encephalopathies (TSEs) in livestock and wildlife, numerous
experiments assessing the susceptibility of various TSEs in different host
species were conducted. Most notable is deer inoculated with scrapie, which
exhibits similarities to chronic wasting disease (CWD) in deer suggestive of
sheep scrapie as an origin of CWD.
snip...
4.Accomplishments 1. Deer inoculated with domestic isolates of sheep
scrapie. Scrapie-affected deer exhibit 2 different patterns of disease
associated prion protein. In some regions of the brain the pattern is much like
that observed for scrapie, while in others it is more like chronic wasting
disease (CWD), the transmissible spongiform encephalopathy typically associated
with deer. This work conducted by ARS scientists at the National Animal Disease
Center, Ames, IA suggests that an interspecies transmission of sheep scrapie to
deer may have been the origin of CWD. This is important for husbandry practices
with both captive deer, elk and sheep for farmers and ranchers attempting to
keep their herds and flocks free of CWD and scrapie.
White-tailed Deer are Susceptible to Scrapie by Natural Route of Infection
Jodi D. Smith, Justin J. Greenlee, and Robert A. Kunkle; Virus and Prion
Research Unit, National Animal Disease Center, USDA-ARS
snip...
This work demonstrates for the first time that white-tailed deer are
susceptible to sheep scrapie by potential natural routes of inoculation.
In-depth analysis of tissues will be done to determine similarities between
scrapie in deer after intracranial and oral/intranasal inoculation and chronic
wasting disease resulting from similar routes of inoculation.
see full text ;
SEE MORE USAHA REPORTS HERE, 2012 NOT PUBLISHED YET...TSS
Thursday, June 20, 2013
atypical, BSE, CWD, Scrapie, Captive Farmed shooting pens (livestock), Wild
Cervids, Rectal Mucosa Biopsy 2012 USAHA Proceedings, and CJD TSE prion Update
Tuesday, September 17, 2013
USAHA 116TH ANNUAL MEETING October 18 – 24, 2012 CWD, Scrapie, BSE, TSE
prion (September 17, 2013)
*** The potential impact of prion diseases on human health was greatly
magnified by the recognition that interspecies transfer of BSE to humans by beef
ingestion resulted in vCJD. While changes in animal feed constituents and
slaughter practices appear to have curtailed vCJD, there is concern that CWD of
free-ranging deer and elk in the U.S. might also cross the species barrier.
Thus, consuming venison could be a source of human prion disease. Whether BSE
and CWD represent interspecies scrapie transfer or are newly arisen prion
diseases is unknown. Therefore, the possibility of transmission of prion disease
through other food animals cannot be ruled out. There is evidence that vCJD can
be transmitted through blood transfusion. There is likely a pool of unknown size
of asymptomatic individuals infected with vCJD, and there may be asymptomatic
individuals infected with the CWD equivalent. These circumstances represent a
potential threat to blood, blood products, and plasma supplies.
IT is of my
opinion, that the OIE and the USDA et al, are the soul reason, and responsible
parties, for Transmissible Spongiform Encephalopathy TSE prion diseases,
including typical and atypical BSE, typical and atypical Scrapie, and all
strains of CWD, and human TSE there from, spreading around the globe.
I have lost all
confidence of this organization as a regulatory authority on animal disease, and
consider it nothing more than a National Trading Brokerage for all strains of
animal TSE, just to satisfy there commodity. AS i said before, OIE should hang
up there jock strap now, since it appears they will buckle every time a country
makes some political hay about trade protocol, commodities and futures. IF they
are not going to be science based, they should do everyone a favor and dissolve
there organization.
JUST because of low
documented human body count with nvCJD and the long incubation periods, the lack
of sound science being replaced by political and corporate science in relations
with the fact that science has now linked some sporadic CJD with atypical BSE
and atypical scrapie, and the very real threat of CWD being zoonosis, I believed
the O.I.E. has failed terribly and again, I call for this organization to be
dissolved. ...
IN A NUT SHELL ;
(Adopted by the
International Committee of the OIE on 23 May 2006)
11. Information
published by the OIE is derived from appropriate declarations made by the
official Veterinary Services of Member Countries. The OIE is not responsible for
inaccurate publication of country disease status based on inaccurate information
or changes in epidemiological status or other significant events that were not
promptly reported to the Central Bureau,
Thursday, May 30,
2013
World Organization
for Animal Health (OIE) has upgraded the United States' risk classification for
mad cow disease to "negligible" from "controlled", and risk further exposing the
globe to the TSE prion mad cow type disease
U.S. gets top
mad-cow rating from international group and risk further exposing the globe to
the TSE prion mad cow type disease
Tuesday, June 11,
2013
Weld County
Bi-Products dba Fort Morgan Pet Foods 6/1/12 significant deviations from
requirements in FDA regulations that are intended to reduce the risk of bovine
spongiform encephalopathy (BSE) within the United States
Thursday, June 6,
2013
BSE TSE PRION USDA
FDA MAD COW FEED COMPLIANCE REPORT and NAI, OAI, and VAI ratings as at June 5,
2013
Greetings,
since our fine
federal friends have decided not to give out any more reports on the USA
breaches of the feed ban and surveillance etc. for the BSE TSE prion mad cow
type disease in the USDA livestock, I thought I might attempt it. I swear, I
just don’t understand the logic of the SSS policy, and that includes all of it.
I assure you, it would be much easier, and probably better for the FDA and the
USDA INC., if they would simply put some kind of report out for Pete’s sake,
instead of me doing it after I get mad, because I am going to put it all out
there. the truth.
PLEASE BE ADVISED,
any breach of any of the above classifications OAI, VAI, RTS, CAN lead to
breaches into the feed BSE TSE prion protocols, and CAN lead to the eventual
suspect tainted feed reaching livestock. please, if any USDA official out there
disputes this, please explain then how they could not. paperwork errors can
eventually lead to breaches of the BSE TSE prion mad cow feed ban reaching
livestock, or contamination and exposure there from, as well.
I would sure like
to see the full reports of just these ;
4018 CHI-DO
3007091297 Rancho Cantera 2866 N Sunnyside Rd Kent IL 61044-9605 OPR FR, OF HP
11/26/2012 OAI Y
9367 3008575486
Rocky Ford Pet Foods 21693 Highway 50 East Rocky Ford CO 81067 OPR RE, TH HP
2/27/2013 OAI N
9446 DEN-DO 1713202
Weld County Bi Products, Inc. 1138 N 11th Ave Greeley CO 80631-9501 OPR RE, TH
HP 10/12/2012 OAI N
9447 DEN-DO
3002857110 Weld County Bi-Products dba Fort Morgan Pet Foods 13553 County Road
19 Fort Morgan CO 80701-7506 OPR RE HP 12/7/2011 OAI N
see full list of
the fda mad cow bse feed follies, toward the bottom, after a short brief update
on the mad cow bse follies, and our good friend Lester Crawford that was at the
FDA.
ALSO, I would
kindly like to comment on this FDA BSE/Ruminant Feed Inspections Firms Inventory
(excel format)4 format, for reporting these breaches of BSE TSE prion protocols,
from the extensive mad cow feed ban warning letters the fda use to put out for
each violations. simply put, this excel format sucks, and the FDA et al
intentionally made it this difficult to follow the usda fda mad cow follies.
this is an intentional format to make it as difficult as possible to follow
these breaches of the mad cow TSE prion safety feed protocols. to have
absolutely no chronological or numerical order, and to format such violations in
a way that they are almost impossible to find, says a lot about just how far the
FDA and our fine federal friends will go through to hide these continued
violations of the BSE TSE prion mad cow feed ban, and any breaches of protocols
there from. once again, the wolf guarding the henhouse $$$
NAI = NO ACTION
INDICATED
OAI = OFFICIAL
ACTION INDICATED
VAI = VOLUNTARY
ACTION INDICATED
RTS = REFERRED TO
STATE
Inspections
conducted by State and FDA investigators are classified to reflect the
compliance status at the time of the inspection, based upon whether
objectionable conditions were documented. Based on the conditions found,
inspection results are recorded in one of three classifications:
OAI (Official
Action Indicated) when inspectors find significant objectionable conditions or
practices and believe that regulatory sanctions are warranted to address the
establishment’s lack of compliance with the regulation. An example of an OAI
classification would be findings of manufacturing procedures insufficient to
ensure that ruminant feed is not contaminated with prohibited material.
Inspectors will promptly re-inspect facilities classified OAI after regulatory
sanctions have been applied to determine whether the corrective actions are
adequate to address the objectionable conditions.
VAI (Voluntary
Action Indicated) when inspectors find objectionable conditions or practices
that do not meet the threshold of regulatory significance, but warrant an
advisory to inform the establishment that inspectors found conditions or
practices that should be voluntarily corrected. VAI violations are typically
technical violations of the 1997 BSE Feed Rule. These violations include minor
recordkeeping lapses or conditions involving non-ruminant feeds.
NAI (No Action
Indicated) when inspectors find no objectionable conditions or practices or, if
they find objectionable conditions, those conditions are of a minor nature and
do not justify further actions.
when sound science
was bought off by junk science, in regards to the BSE TSE prion mad cow type
disease, by the USDA, CFIA, WHO, OIE, et al. $$$
when the infamous,
and fraudulently USDA, FSIS, APHIS, FDA, gold card was taken away that infamous
day in December of 2003, all cards were off the table, it was time to change the
science, and change they did. ...tss
snip. ...please see
full text ;
Thursday, June 6,
2013
BSE TSE PRION USDA
FDA MAD COW FEED COMPLIANCE REPORT and NAI, OAI, and VAI ratings as at June 5,
2013
Friday, July 19,
2013
PART 589 --
SUBSTANCES PROHIBITED FROM USE IN ANIMAL FOOD OR FEED Revised as of April 1,
2013 50# Regular Chicken Feed was found to contain mammalian protein label does
not contain the warning statement
From:
Terry S.
Singeltary Sr.
Sent: Sunday, July 21, 2013 1:44 PM
Cc: wag-en@mailuk.custhelp.com ; Carwyn.jones@wales.gsi.gov.uk ;
Correspondence.Alun.Davies@Wales.gsi.gov.uk ; Correspondence.Carl.Sargeant@Wales.gsi.gov.uk ; Correspondence.Edwina.Hart@Wales.gsi.gov.uk ; Correspondence.Huw.Lewis@Wales.gsi.gov.uk ; Correspondence.Jane.Hutt@Wales.gsi.gov.uk ; Correspondence.John.Griffiths@Wales.gsi.gov.uk ; Correspondence.Lesley.Griffiths@Wales.gsi.gov.uk ; Correspondence.Mark.Drakeford@Wales.gsi.gov.uk ; Correspondence.Jeff.Cuthbert@Wales.gsi.gov.uk ; Correspondence.Vaughan.Gething@wales.gsi.gov.uk ; Correspondence.Ken.Skates@wales.gsi.gov.uk ; Correspondence.Gwenda.Thomas@Wales.gsi.gov.uk
Subject: Welsh Government and Food Standards Agency Wales Joint
Public Consultation on the Proposed Transmissible Spongiform Encephalopathies
(Wales) Regulations 2013
July
21, 2013
Subject:
Welsh Government and Food Standards Agency Wales Joint Public Consultation on
the Proposed Transmissible Spongiform Encephalopathies (Wales) Regulations
2013
endemics@wales.gsi.gov.uk; fenris@caramail.com;
Greetings Welsh Government and Food Standards Agency Wales,
With great urgency, I would kindly like to comment on ;
Number: WG18417
Joint Public Consultation on the Proposed Transmissible
Spongiform Encephalopathies (Wales) Regulations
2013
The European Food Safety Authority (EFSA) and the European Centre for
Disease Prevention and Control jointly advised in 2011 that BSE is the only
animal TSE that has been shown to be a risk to human health and that there is no
epidemiological evidence to suggest that classical scrapie is a risk to human
health.
and
What are the main issues under consideration?
2.1 The main issues under consideration relate to changes in BSE testing
requirements; more proportionate measures for controlling classical scrapie in
sheep flocks and goat herds in which classical scrapie is confirmed; and more
proportionate controls on animal feed. The Welsh Government also wishes to
consult on proposed amendments to the BSE cattle compensation system in light of
identified anomalies in the current system, in addition to a variety of other
proposed technical and procedural amendments to the 2008 Regulations.
2.2 The key specific amendments are summarised and then considered in more
detail below. Other proposed technical amendments, which are considered to have
a negligible impact or have already been implemented administratively, are
listed at Annex A.
What are the main issues under consideration?
2.1
* The main issues under consideration relate to changes in BSE testing
requirements;
* more proportionate measures for controlling classical scrapie in sheep
flocks
* and goat herds in which classical scrapie is confirmed; and
* more proportionate controls on animal feed.
Greetings again Welsh Government and Food
Standards Agency Wales,
with another TSE prion medical blunder
happening just this past week ;
Friday, July 19, 2013
Beaumont Hospital in Dublin assessing patients for CJD http://creutzfeldt-jakob-disease.blogspot.com/2013/07/beaumont-hospital-in-dublin-assessing.html
I STRENUOUSLY urge you take my submission with the greatest urgency.
My submission and concerns as follows, and in part mixed in and throughout the WG18417 Proposed Transmissible Spongiform Encephalopathies (Wales) Regulations 2013.
First and foremost, I think it is very important for the Welsh Government and Food Standards Agency Wales, to take a full inventory of your imports from North America, and other Countries, especially your pet and fish foods, for reasons and scientific facts I have supplied below.
I think the attempt by Governments around the globe to do away with the Transmissible Spongiform Encephalopathy TSE prion disease, via weakening of the BSE TSE prion surveillance, lowering of age limits in testing, weakening the feed bans, caving in to industry, ignoring all the science of the past 3 decades, ignoring these different atypical TSE prion disease breaking out, making up new names for these TSE prion disease, and by the way, what ever happened to the IBNC BSE, and the pathology there, and what about testing there from?
The USDA, CFIA, with the help of the OIE, have made it there goal to extinguish all BSE TSE prion trade barriers, before all the science on the TSE prion disease is in, and are working hard to exempt all TSE prion in all species from any trade barrier, and the OIE is working right along with them. This happened December 2003, when the USA lost it’s OIE BSE Gold Card, when BSE was first documented in the USA, after a long hard fought battle trying to cover mad cow disease up. This proven time and time again by the OIG and the GAO of the USA. I will supply url links of submissions I have made to the USDA et al over the years, since the death of my Mother to the Heidenhain Variant of Creutzfeldt Jakob Disease i.e. hvCJD ‘confirmed’ 12/14/97. just another happenstance of bad luck they tell me, that it only happens in the UK, and that it’s a UK disease, this mad cow disease, that no other animal species TSE prion disease in the world, at no other location, can transmit a TSE prion disease to a human, and then basing all trading protocol from country to country, based on this junk science, destroys the past 3 decades of trying to eradicate the damn disease, and the OIE, USDA, CFIA, and all the other countries that know better, that just goes along with this due to trade purposes, just because it’s a long incubating disease, just because the science is still in it’s infancy, does not mean we should start ignoring what early science has taught us, and start weakening any safety protocols there from.
North America has the most documented TSE prion disease in the wild and in farmed livestock than any other country in the world, excluding the TSE prion disease documented in zoo animals. Chronic Wasting Disease CWD in cervids is running rampant, the USA and Canada can’t stop it, while Mexico has not a clue of any TSE prion disease. The shooting pens, and CWD there from in the USA, is a real risk factor, one the game farms refuse to admit they are a big problem with the spreading of the CWD TSE prion disease, fighting tooth and nail completely ignoring the evolving science on the CWD TSE prion disease, and how it’s spread, and these antler mills are multiplying from state to state in big numbers. There is ample evidence of CWD transmission to humans, to warrant a warning to the world, of the IATROGENIC potential for this TSE prion disease in cervids, via the multitude of potential routes of infection, via the medical, dental, surgical, blood, tissue. CWD has now mutated to multiple strains. The science is there to warrant this very real concern, it’s just the same as with what happened in the U.K., the industry and USDA inc., are stopping these concerns to be made public, with the same watered down junk science used in the beginning of the BSE blunder. you should all be very aware of this, if you come abroad to North America. DEFRA has put out a warning on CWD TSE prion disease in the USA and have put out a document I supplied with additional risk factors from North America, this is supplied below as well.
Another concern is with your assumptions that typical classical scrapie is not a risk factor for humans, when there _is_ evidence to show otherwise, that indeed typical scrapie is a risk factor for humans, as with atypical Scrapie.
The OIE, USDA inc, and the CFIA, have come to the conclusion that neither typical scrapie nor the atypical Nor-98 scrapie are neither a risk factor for humans, they have urged the OIE to conclude that atypical Nor-98 to be exempt from any trading protocols, and indeed have made the Nor-98 atypical scrapie EXEMPT, and made it legal to trade, and they are also in the works to make typical scrapie exempt.
typical scrapie consist of many different strains of scrapie, not just one. and the atypical Nor-98 has very similar features with human Gerstmann-Sträussler-Scheinker Disease GSS and Variably Protease-Sensitive Prionopathy VPSPr. I have not seen anywhere in the Bible, or the scrolls of the Dead Sea, where it was stipulated that indeed typical c-BSE is the only zoonosis Transmissible Spongiform Encephalopathy TSE prion disease. This is ludicrous in 2013 to still believe this junk science.
With the science to date of the 1st 10 nvCJD victims of Dr. Ironside et al, and the diagnostic criteria then to diagnose the nvCJD, compared to what Dr. Gambetti et al diagnosed in their 1st 10, of which young victims are being diagnosed, but yet changed the name to VPSPr type CJD human TSE, is not scientific in my opinion. I believe that the UKBSEnvCJD only theory is bogus, it is not scientific, and should be put to bed once and for all. you cannot have your cake and eat it too. either Ironside was wrong, or Gambetti is wrong. to continue this UKBSEnvCJD only myth, will only help continue spread the TSE prion agent long and far.
typical Scrapie has been studied for decades and decades, and has proven to be transmitted to non-human primates by their NON-FORCED oral consumption (Gibbs et al). when you write in absolute terms as this is fact that ‘’BSE is the only animal TSE that has been shown to be a risk to human health and that there is no epidemiological evidence to suggest that classical scrapie is a risk to human health’’ may be true in terms of documentation, but in terms of science to date, I think you are wishing. I kindly wish to submit the following in good faith ;
snip...
Singeltary Complete submission located in PDF attachment in this email, or in the link below...
Sunday, July 21, 2013
Welsh Government and Food Standards Agency Wales Joint Public Consultation
on the Proposed Transmissible Spongiform Encephalopathies (Wales) Regulations
2013 Singeltary Submission WG18417
TSS
posted by Terry S. Singeltary Sr. at
2:23 PM
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