Indiana State to appeal high-fence deer hunting ruling
State to appeal high-fence deer hunting ruling
Attorney General Greg Zoeller’s office filed notice of
appeal today, two weeks after Harrison County Circuit Court Judge John Evans
issued a ruling in favor of Rodney Bruce, who operates Whitetail Bluff, a
preserve near Corydon.
In 2005 Bruce sued after the DNR tried to shut down his
operation and the 11 other high-fence hunting preserves in the state at the
time.
Owens ruling says that the DNR overstepped its
authority, and that deer at facilities such as Bruce’s “are privately owned and
not the property of the people of the state of Indiana,” so they’re not subject
to the DNR’s oversight.
Evans’ ruling troubled animal rights activists and
hunting groups who say they’re worried that it could severely restrict the
ability of state wildlife officials to enforce state hunting laws inside the
fenced enclosures, which they disparagingly call “canned hunts.” They say Evans
ruling also challenged the notion that wildlife is owned by the public and only
in special, tightly regulated circumstances can private individuals possess and
kill the animals.
While the actual legal arguments behind the attorney
general’s appeal haven’t yet been disclosed, both preserve operators and their
critics hope the state appellate court clarifies the matter. Two judges have
recently issued conflicting rulings on hunting preserves.
The Harrison County decision came 10 months after a
judge in Owen County threw out a similar challenge by a preserve
owner.
Call Star reporter
Ryan Sabalow at (317) 444-6179. Follow him on Twitter:
@twitter.com/ryansabalow.
http://www.indystar.com/article/20131015/NEWS/310150039/State-appeal-high-fence-deer-hunting-ruling?nclick_check=1
Saturday, March 23,
2013
Rep. Matt Ubelhor of
Bloomfield is going to amend Senate Bill 487 to include the legalization of
“canned” deer hunting operations in Indiana ?
Tuesday, February 14,
2012
Oppose Indiana House
Bill 1265 game farming cervids
Sunday, January 27, 2013
Indiana 6 deer missing from farm pose health risk to
state herds
INDIANA
1/22/2013 11:11:00 AM
6 deer
missing from farm pose health risk to state herds
Aubrey
Woods, Tribune
Indiana
Department of Natural Resourcescontinues to search for six ear-tagged deer in
Jackson County and neighboring Bartholomew, Jennings and Scott counties. Those
deer are among the 20 that escaped from a captive cervid facility — or deer farm
— in 2012, a spokesman with the state Department of Natural Resources said
Friday.
“Of the six,
one is a buck, which had a yellow ear tag with the No. 47,” Phil Bloom
said.
Bloom said
the buck is the only one of the six missing deer imported from a site in
Pennsylvania where deer have tested positive for chronic wasting
disease.
“He has been
exposed to it, but that doesn’t mean he has chronic wasting disease,” Bloom
said. “There is no live test for chronic wasting disease. That can’t be
determined until the deer is dead and tissue has been
tested.”
Bloom said
the other five deer that remain free are classified as exposed to chronic
wasting disease through their association with the buck.
Of
particular interest are any deer with a yellow ear tag bearing the prefix “IN
764” followed by another four numbers or any deer with a yellow ear tag and two
numbers on it.
Natural
Resources staff will assist in taking the deer carcass to Purdue University for
testing at the Indiana Animal Disease Diagnostic Laboratory.
Of the
remaining 20 that escaped from the deer farm, the owner recaptured 11 shortly
after the escape.
The location
and ownership of the deer farm have not been released, based on guidelines of
the Indiana Board of Animal Health, but the owner is cooperating, Bloom
said.
The escape
occurred after a tree fell on a fence at the deer farm, Bloom
said.
Of the
remaining nine deer that escaped, one was struck and killed by a vehicle, a
second was shot and killed by a bow hunter during archery season and a third was
killed during a special deer hunting session the first weekend of January, Bloom
said.
The owner of
the farm continues to help search for the remaining six and has been cooperative
with the state, Bloom said.
State
officials believe the buck is still alive because people have reported seeing
it, Bloom said.
“Although it
is conceivable it may have been killed,” he said.
Hunters also
may have killed some of the five missing deer and just not reported their deaths
to the state as required, Bloom said.
Bloom said
there are no special hunting seasons planned to find the remaining
deer.
“We’re
making an all-out effort to find the deer,” Bloom said.
Local hunter
Chuck Brenner of Freetown said the state is being too complacent about an issue
that could have long-range effects on the deer population,
however.
“I’m not
just a hunter,” Brenner said. “I’m a conservationist. We need to manage our
herds.”
Brenner said
chronic wasting disease could wipe out herds and cost the state millions of
dollars down the road.
“We should
hunt them down and find them whether it’s deer season or not,” Brenner
said.
He said
there hasn’t been much publicity about the missing deer and that local hunters
could help with the search.
“They need
to ask the local hunters for help,” Brenner said. “They could put out trail
cameras. If nothing else, the state could put a bounty on
them.”
Brenner said
the deer are clearly marked and there could be a special hunt just for the six
deer that are ear-tagged.
Bloom said
the state wants to make sure that the deer are tracked down, tested and
determined if they’ve been exposed to chronic wasting
disease.
“It could
have some far-reaching ramifications,” he said.
So far the
disease has never been found in Indiana, which has been testing for the disease
since 2002. More than 10,000 deer killed during hunts or by vehicles have been
tested and the disease has not been detected, Bloom said.
The tags on
those missing deer are expected to tell officials whether the animals have any
possible connection with a captive facility in Pennsylvania where chronic
wasting disease was confirmed this winter, Bloom said. That report made
Pennsylvania the 23rd state in which it has been found.
Cervid or
wild game farms in Indiana can raise deer, elk and moose for several reasons,
Bloom said. The animals can be processed at a slaughterhouse and sold as meat,
sold for pets or sold to high-fenced hunting reserves.
Copyright ©
2013 The Tribune
Editor, John
C. DePrez Jr.; Executive Editor, Carol Rogers; Publishers: IBRC and
IAR
Long kills
controversial fenced hunting bill
February 1,
2012 | Filed under: Issues,Top stories | Posted by: Lesley Weidenbener
By Lesley
Weidenbener
The
Statehouse File
INDIANAPOLIS
– An effort to legalize fenced deer hunting is dead for this session.
...
snip...
see full text ;
Sunday, January 27, 2013
Indiana 6 deer missing from farm
pose health risk to state herds INDIANA
how many
states have $465,000., and can quarantine and purchase there from, each cwd said
infected farm, but how many states can afford this for all the cwd infected
cervid game ranch type farms ???
Tuesday, December 20, 2011
CHRONIC WASTING DISEASE CWD WISCONSIN Almond
Deer (Buckhorn Flats) Farm Update DECEMBER 2011
The CWD infection rate was nearly 80%, the
highest ever in a North American captive herd.
RECOMMENDATION: That the Board approve the
purchase of 80 acres of land for $465,000 for the Statewide Wildlife Habitat
Program in Portage County and approve the restrictions on public use of the
site.
SUMMARY:
recently, a report came out in the U.K.,
about risk factors from entry of CWD from the USA. I think you might find
interest there ;
Friday, December 14, 2012
DEFRA U.K. What is the risk of Chronic
Wasting Disease CWD being introduced into Great Britain? A Qualitative Risk
Assessment October 2012
snip...
In the USA, under the Food and Drug
Administration’s BSE Feed Regulation (21 CFR 589.2000) most material (exceptions
include milk, tallow, and gelatin) from deer and elk is prohibited for use in
feed for ruminant animals. With regards to feed for non-ruminant animals, under
FDA law, CWD positive deer may not be used for any animal feed or feed
ingredients. For elk and deer considered at high risk for CWD, the FDA
recommends that these animals do not enter the animal feed system. However, this
recommendation is guidance and not a requirement by law.
Animals considered at high risk for CWD
include:
1) animals from areas declared to be endemic
for CWD and/or to be CWD eradication zones and
2) deer and elk that at some time during the
60-month period prior to slaughter were in a captive herd that contained a
CWD-positive animal.
Therefore, in the USA, materials from cervids
other than CWD positive animals may be used in animal feed and feed ingredients
for non-ruminants.
The amount of animal PAP that is of deer
and/or elk origin imported from the USA to GB can not be determined, however, as
it is not specified in TRACES. It may constitute a small percentage of the 8412
kilos of non-fish origin processed animal proteins that were imported from US
into GB in 2011.
Overall, therefore, it is considered there is
a __greater than negligible risk___ that (nonruminant) animal feed and pet food
containing deer and/or elk protein is imported into GB.
There is uncertainty associated with this
estimate given the lack of data on the amount of deer and/or elk protein
possibly being imported in these products.
snip...
36% in 2007 (Almberg et al., 2011). In such
areas, population declines of deer of up to 30 to 50% have been observed
(Almberg et al., 2011). In areas of Colorado, the prevalence can be as high as
30% (EFSA, 2011). The clinical signs of CWD in affected adults are weight loss
and behavioural changes that can span weeks or months (Williams, 2005). In
addition, signs might include excessive salivation, behavioural alterations
including a fixed stare and changes in interaction with other animals in the
herd, and an altered stance (Williams, 2005). These signs are indistinguishable
from cervids experimentally infected with bovine spongiform encephalopathy
(BSE). Given this, if CWD was to be introduced into countries with BSE such as
GB, for example, infected deer populations would need to be tested to
differentiate if they were infected with CWD or BSE to minimise the risk of BSE
entering the human food-chain via affected venison.
snip...
The rate of transmission of CWD has been
reported to be as high as 30% and can approach 100% among captive animals in
endemic areas (Safar et al., 2008).
snip...
In summary, in endemic areas, there is a
medium probability that the soil and surrounding environment is contaminated
with CWD prions and in a bioavailable form. In rural areas where CWD has not
been reported and deer are present, there is a greater than negligible risk the
soil is contaminated with CWD prion.
snip...
In summary, given the volume of tourists,
hunters and servicemen moving between GB and North America, the probability of
at least one person travelling to/from a CWD affected area and, in doing so,
contaminating their clothing, footwear and/or equipment prior to arriving in GB
is greater than negligible. For deer hunters, specifically, the risk is likely
to be greater given the increased contact with deer and their environment.
However, there is significant uncertainty associated with these estimates.
snip...
Therefore, it is considered that farmed and
park deer may have a higher probability of exposure to CWD transferred to the
environment than wild deer given the restricted habitat range and higher
frequency of contact with tourists and returning GB residents.
snip...
SNIP...SEE ;
Friday, December 14, 2012
DEFRA U.K. What is the risk of Chronic
Wasting Disease CWD being introduced into Great Britain? A Qualitative Risk
Assessment October 2012
Wednesday, September 25, 2013
Inspections, Compliance, Enforcement, and
Criminal Investigations BSE TSE PRION 2013
DOCKET-- 03D-0186 -- FDA Issues Draft
Guidance on Use of Material From Deer and Elk in Animal Feed;
Availability
Date: Fri, 16 May 2003 11:47:37
–0500
EMC 1 Terry S. Singeltary Sr. Vol #:
1
PLEASE SEE FULL TEXT SUBMISSION
;
please see more on the potential of the prion
uptake from plants, and see what SEAC said about the one study that showed prion
uptake of the Tomato plant, although the test was never repeated ;
Furthermore, an unpublished study had
indicated low level absorption of PrP from soil by tomato plants although it
should be noted that this study had not been repeated. Details of this work
would be sent to the SEAC Secretary. Dr Matthews explained that most of the
manure from animals challenged with high doses of BSE had already been composted
and used for coppicing. Members agreed that the risks from disposal of residual
manure from experimental animals would be much less than historic risks of on
farm contamination from naturally infected animals at the height of the BSE
epidemic. ...SNIP...END
Uptake of Prions into Plants
Prion2013
Friday, August 09, 2013
***CWD TSE prion, plants, vegetables, and the
potential for environmental contamination
PRION2013 CONGRESSIONAL ABSTRACTS CWD
Thursday, August 08, 2013
Characterization of the first case of
naturally occurring chronic wasting disease in a captive red deer (Cervus
elaphus) in North America
Sunday, September 01, 2013
hunting over gut piles and CWD TSE prion
disease
Wednesday, September 04, 2013
***cwd - cervid captive livestock escapes,
loose and on the run in the wild...
Saturday, June 29, 2013
PENNSYLVANIA CAPTIVE CWD INDEX HERD MATE
YELLOW *47 STILL RUNNING LOOSE IN INDIANA, YELLOW NUMBER 2 STILL MISSING, AND
OTHERS ON THE RUN STILL IN LOUISIANA
Monday, June 24, 2013
The Effects of Chronic Wasting Disease on the
Pennsylvania Cervid Industry Following its Discovery
Tuesday, June 11, 2013
CWD GONE WILD, More cervid escapees from more
shooting pens on the loose in Pennsylvania
Tuesday, May 28, 2013
Chronic Wasting Disease CWD quarantine
Louisiana via CWD index herd Pennsylvania Update May 28, 2013
6 doe from Pennsylvania CWD index herd still
on the loose in Louisiana, quarantine began on October 18, 2012, still ongoing,
Lake Charles premises.
Prion Disease Detection,
PMCA Kinetics, and IgG in Urine from Sheep Naturally/Experimentally Infected
with Scrapie and Deer with Preclinical/Clinical Chronic Wasting Disease▿
- Richard
Rubenstein1,*,
- Binggong
Chang1,
- Perry
Gray2,
- Martin
Piltch2,
- Marie S.
Bulgin3,
- Sharon
Sorensen-Melson3 and
- Michael W.
Miller4
+ Author Affiliations
ABSTRACT
Prion
diseases, also known as transmissible spongiform encephalopathies, are fatal
neurodegenerative disorders. Low levels of infectious agent and limited,
infrequent success of disease transmissibility and PrPSc detection
have been reported with urine from experimentally infected clinical cervids and
rodents. We report the detection of prion disease-associated seeding activity
(PASA) in urine from naturally and orally infected sheep with clinical scrapie
agent and orally infected preclinical and infected white-tailed deer with
clinical chronic wasting disease (CWD). This is the first report on PASA
detection of PrPSc from the urine of naturally or preclinical
prion-diseased ovine or cervids. Detection was achieved by using the surround
optical fiber immunoassay (SOFIA) to measure the products of limited serial
protein misfolding cyclic amplification (sPMCA). Conversion of PrPC
to PrPSc was not influenced by the presence of poly(A) during sPMCA
or by the homogeneity of the PrP genotypes between the PrPC source
and urine donor animals. Analysis of the sPMCA-SOFIA data resembled a linear,
rather than an exponential, course. Compared to uninfected animals, there was a
2- to 4-log increase of proteinase K-sensitive, light chain immunoglobulin G
(IgG) fragments in scrapie-infected sheep but not in infected CWD-infected deer.
The higher-than-normal range of IgG levels found in the naturally and
experimentally infected clinical scrapie-infected sheep were independent of
their genotypes. Although analysis of urine samples throughout the course of
infection would be necessary to determine the usefulness of altered IgG levels
as a disease biomarker, detection of PrPSc from PASA in urine points
to its potential value for antemortem diagnosis of prion diseases.
FOOTNOTES
- Received 13
May 2011.
- Accepted 14
June 2011.
- ↵*Corresponding author. Mailing address: Downstate Medical Center, Departments of Neurology and
Physiology/Pharmacology, Box 1213, 450 Clarkson Avenue, Brooklyn, NY
11203. Phone: (718) 270-2019. Fax: (718) 270-2459. E-mail: richard.rubenstein@downstate.edu.
-
↵▿ Published ahead of print on 29 June 2011.
- Copyright © 2011, American Society for Microbiology. All
Rights Reserved.
Thursday, June 09, 2011
Detection of CWD prions in salivary, urinary, and
intestinal tissues of deer: potential mechanisms of prion shedding and
transmission
Wednesday, March 18, 2009
Detection of CWD Prions in Urine and Saliva of Deer
by Transgenic Mouse Bioassay
doi:10.1016/j.febslet.2008.08.003 Copyright © 2008
Published by Elsevier B.V.
Detection of infectious prions in
urine
Dennisse Gonzalez-Romeroa, Marcelo A. Barriaa,
Patricia Leona, Rodrigo Moralesa and Claudio Soto, a,
Subject: MAD DEER/ELK DISEASE AND POTENTIAL
SOURCES
Date: Sat, 20 Jul 2002
09:43:10 -0700
From: "Terry S.
Singeltary Sr."
Subject: MAD
DEER/ELK DISEASE AND POTENTIAL SOURCES
Date: Sat, 25 May 2002 18:41:46
-0700
From: "Terry S. Singeltary
Sr."
Reply-To: Bovine Spongiform
Encephalopathy To: BSE-L@uni-karlsruhe.de
######## Bovine Spongiform Encephalopathy
#########
MAD DEER/ELK DISEASE
AND POTENTIAL SOURCES
snip...
now, what about those 'deer scents' of 100% urine',
and the prion that is found in urine, why not just pass the prion with the urine
to other deer...
Mrs.
Doe Pee Doe in Estrus Model FDE1 Mrs. Doe Pee's Doe in Estrus is made from
Estrus urine collected at the peak of the rut, blended with Fresh Doe Urine for
an extremely effective buck enticer. Use pre-rut before the does come into heat.
Use during full rut when bucks are most active. Use during post-rut when bucks
are still actively looking for does. 1 oz.
http://www.gamecalls.net/huntingproducts/deerlures.html
ELK SCENT/SPRAY BOTTLE
*
Works anytime of the year *
100 % Cow Elk-in-Heat urine (2oz.)
*
Economical - mix with water in
spray mist bottle *
Use wind to
your advantage
Product Code
WP-ESB $9.95
http://www.elkinc.com/Scent.asp
prions in urine?
[PDF] A URINE TEST FOR THE IN-VIVO DIAGNOSIS OF
PRION DISEASES
http://www.sigov.si/vurs/PDF/diagnoastika-bse-urin.pdf
Monday, June 17, 2013
Early detection of chronic
wasting disease prions in urine of pre-symptomatic deer by real-time
quaking-induced conversion assay
http://chronic-wasting-disease.blogspot.com/2013/06/early-detection-of-chronic-wasting.html
Tuesday, September 10, 2013
Review and Updates of the USDA-APHIS
Veterinary Services (VS) National Chronice Wasting Disease (CWD) Program
2012-2013
Sunday, August 11, 2013
Creutzfeldt-Jakob Disease CJD cases rising
North America updated report August 2013
Creutzfeldt-Jakob Disease CJD cases rising
North America with Canada seeing an extreme increase of 48% between 2008 and
2010
CWD transmission to humans.
NEVER ???
never say never with the TSE prion.
PRION2013 CONGRESSIONAL ABSTRACTS CWD
Sunday, August 25, 2013
HD.13: CWD infection in the spleen of
humanized transgenic mice
Liuting Qing and Qingzhong Kong
Case Western Reserve University; Cleveland,
OH USA
Chronic wasting disease (CWD) is a widespread
prion disease in free-ranging and captive cervid species in North America, and
there is evidence suggesting the existence of multiple CWD strains. The
susceptibility of human CNS and peripheral organs to the various CWD prion
strains remains largely unclear. Current literature suggests that the classical
CWD strain is unlikely to infect human brain, but the potential for peripheral
infection by CWD in humans is unknown. We detected protease-resistant PrpSc in
the spleens of a few humanized transgenic mice that were intracerebrally
inoculated with natural CWD isolates, but PrpSc was not detected in the brains
of any of the CWD-inoculated mice. Our ongoing bioassays in humanized Tg mice
indicate that intracerebral challenge with such PrpSc-positive humanized mouse
spleen already led to prion disease in most animals. ***These results indicate
that the CWD prion may have the potential to infect human peripheral lymphoid
tissues.
Oral.15: Molecular barriers to zoonotic prion
transmission: Comparison of the ability of sheep, cattle and deer prion disease
isolates to convert normal human prion protein to its pathological isoform in a
cell-free system
Marcelo A.Barria,1 Aru Balachandran,2
Masanori Morita,3 Tetsuyuki Kitamoto,4 Rona Barron,5 Jean Manson,5 Richard
Kniqht,1 James W. lronside1 and Mark W. Head1
1National CJD Research and Surveillance Unit;
Centre for Clinical Brain Sciences; School of Clinical Sciences; The University
of Edinburgh; Edinburgh, UK; 2National and OIE Reference Laboratory for Scrapie
and CWD; Canadian Food Inspection Agency; Ottawa Laboratory; Fallowfield. ON
Canada; 3Infectious Pathogen Research Section; Central Research Laboratory;
Japan Blood Products Organization; Kobe, Japan; 4Department of Neurological
Science; Tohoku University Graduate School of Medicine; Sendai. Japan;
5Neurobiology Division; The Roslin Institute and R(D)SVS; University of
Edinburgh; Easter Bush; Midlothian; Edinburgh, UK
Background. Bovine spongiform encephalopathy
(BSE) is a known zoonotic prion disease, resulting in variant Creurzfeldt- Jakob
disease (vCJD) in humans. In contrast, classical scrapie in sheep is thought to
offer little or no danger to human health. However, a widening range of prion
diseases have been recognized in cattle, sheep and deer. The risks posed by
individual animal prion diseases to human health cannot be determined a priori
and are difficult to assess empirically. The fundamemal event in prion disease
pathogenesis is thought to be the seeded conversion of normal prion protein
(PrPC) to its pathological isoform (PrPSc). Here we report the use of a rapid
molecular conversion assay to test whether brain specimens from different animal
prion diseases are capable of seeding the conversion of human PrPC ro PrPSc.
Material and Methods. Classical BSE (C-type
BSE), H-type BSE, L-type BSE, classical scrapie, atypical scrapie, chronic
wasting disease and vCJD brain homogenates were tested for their ability to seed
conversion of human PrPC to PrPSc in protein misfolding cyclic amplification
(PMCA) reactions. Newly formed human PrPSc was detected by protease digestion
and western blotting using the antibody 3F4.
Results. C-type BSE and vCJD were found to
efficiently convert PrPC to PrPSc. Scrapie failed to convert human PrPC to
PrPSc. Of the other animal prion diseases tested only chronic wasting disease
appeared to have the capability ro convert human PrPC to PrPSc. The results were
consistent whether the human PrPC came from human brain, humanised transgenic
mouse brain or from cultured human cells and the effect was more pronounced for
PrPC with methionine at codon 129 compared with that with valine.
Conclusion. Our results show that none of the
tested animal prion disease isolates are as efficient as C-type BSE and vCJD in
converting human prion protein in this in vitro assay. ***However, they also
show that there is no absolute barrier ro conversion of human prion protein in
the case of chronic wasting disease.
PRION2013 CONGRESSIONAL ABSTRACTS CWD
Sunday, August 25, 2013
***Chronic Wasting Disease CWD risk factors,
humans, domestic cats, blood, and mother to offspring transmission
Sunday, July 21, 2013
*** As Chronic Wasting Disease CWD rises in
deer herd, what about risk for humans?
> sCJDMM1-2 should be considered as a
separate entity at this time.
> All of the Heidenhain variants were of
the methionine/ methionine type 1 molecular subtype.
*** The potential impact of prion diseases on
human health was greatly magnified by the recognition that interspecies transfer
of BSE to humans by beef ingestion resulted in vCJD. While changes in animal
feed constituents and slaughter practices appear to have curtailed vCJD, there
is concern that CWD of free-ranging deer and elk in the U.S. might also cross
the species barrier. Thus, consuming venison could be a source of human prion
disease. Whether BSE and CWD represent interspecies scrapie transfer or are
newly arisen prion diseases is unknown. Therefore, the possibility of
transmission of prion disease through other food animals cannot be ruled out.
There is evidence that vCJD can be transmitted through blood transfusion. There
is likely a pool of unknown size of asymptomatic individuals infected with vCJD,
and there may be asymptomatic individuals infected with the CWD equivalent.
These circumstances represent a potential threat to blood, blood products, and
plasma supplies.
now, let’s see what the authors said about
this casual link, personal communications years ago. see where it is stated NO
STRONG evidence. so, does this mean there IS casual evidence ????
“Our conclusion stating that we found no
strong evidence of CWD transmission to humans”
From: TSS (216-119-163-189.ipset45.wt.net)
Subject: CWD aka MAD DEER/ELK TO HUMANS ???
Date: September 30, 2002 at 7:06 am PST
From: "Belay, Ermias"
To:
Cc: "Race, Richard (NIH)" ; ; "Belay, Ermias"
Sent: Monday, September 30, 2002 9:22 AM
Subject: RE: TO CDC AND NIH - PUB MED- 3 MORE
DEATHS - CWD - YOUNG HUNTERS
Dear Sir/Madam,
In the Archives of Neurology you quoted (the
abstract of which was attached to your email), we did not say CWD in humans will
present like variant CJD.
That assumption would be wrong. I encourage
you to read the whole article and call me if you have questions or need more
clarification (phone: 404-639-3091). Also, we do not claim that "no-one has ever
been infected with prion disease from eating venison." Our conclusion stating
that we found no strong evidence of CWD transmission to humans in the article
you quoted or in any other forum is limited to the patients we investigated.
Ermias Belay, M.D. Centers for Disease
Control and Prevention
-----Original Message-----
From:
Sent: Sunday, September 29, 2002 10:15 AM
To: rr26k@nih.gov; rrace@niaid.nih.gov;
ebb8@CDC.GOV
Subject: TO CDC AND NIH - PUB MED- 3 MORE
DEATHS - CWD - YOUNG HUNTERS
Sunday, November 10, 2002 6:26 PM
......snip........end..............TSS
Thursday, April 03, 2008
A prion disease of cervids: Chronic wasting
disease
2008 1: Vet Res. 2008 Apr 3;39(4):41
A prion disease of cervids: Chronic wasting
disease
Sigurdson CJ.
snip...
*** twenty-seven CJD patients who regularly
consumed venison were reported to the Surveillance Center***,
snip...
full text ;
CJD REPORT 1994 increased risk for
consumption of veal and venison and lamb
CREUTZFELDT JAKOB DISEASE SURVEILLANCE IN THE
UNITED KINGDOM THIRD ANNUAL REPORT AUGUST 1994
Consumption of venison and veal was much less
widespread among both cases and controls. For both of these meats there was
evidence of a trend with increasing frequency of consumption being associated
with increasing risk of CJD. (not nvCJD, but sporadic CJD...tss)
These associations were largely unchanged
when attention was restricted to pairs with data obtained from relatives. ...
Table 9 presents the results of an analysis
of these data.
There is STRONG evidence of an association
between ‘’regular’’ veal eating and risk of CJD (p = .0.01).
Individuals reported to eat veal on average
at least once a year appear to be at 13 TIMES THE RISK of individuals who have
never eaten veal.
There is, however, a very wide confidence
interval around this estimate. There is no strong evidence that eating veal less
than once per year is associated with increased risk of CJD (p = 0.51).
The association between venison eating and
risk of CJD shows similar pattern, with regular venison eating associated with a
9 FOLD INCREASE IN RISK OF CJD (p = 0.04).
There is some evidence that risk of CJD
INCREASES WITH INCREASING FREQUENCY OF LAMB EATING (p = 0.02).
The evidence for such an association between
beef eating and CJD is weaker (p = 0.14). When only controls for whom a relative
was interviewed are included, this evidence becomes a little STRONGER (p =
0.08).
snip...
It was found that when veal was included in
the model with another exposure, the association between veal and CJD remained
statistically significant (p = < 0.05 for all exposures), while the other
exposures ceased to be statistically significant (p = > 0.05).
snip...
In conclusion, an analysis of dietary
histories revealed statistical associations between various meats/animal
products and INCREASED RISK OF CJD. When some account was taken of possible
confounding, the association between VEAL EATING AND RISK OF CJD EMERGED AS THE
STRONGEST OF THESE ASSOCIATIONS STATISTICALLY. ...
snip...
In the study in the USA, a range of
foodstuffs were associated with an increased risk of CJD, including liver
consumption which was associated with an apparent SIX-FOLD INCREASE IN THE RISK
OF CJD. By comparing the data from 3 studies in relation to this particular
dietary factor, the risk of liver consumption became non-significant with an
odds ratio of 1.2 (PERSONAL COMMUNICATION, PROFESSOR A. HOFMAN. ERASMUS
UNIVERSITY, ROTTERDAM). (???...TSS)
snip...see full report ;
Thursday, October 10, 2013
CJD REPORT 1994 increased risk for
consumption of veal and venison and lamb
CJD9/10022
October 1994
Mr R.N. Elmhirst Chairman British Deer
Farmers Association Holly Lodge Spencers Lane BerksWell Coventry CV7
7BZ
Dear Mr Elmhirst,
CREUTZFELDT-JAKOB DISEASE (CJD) SURVEILLANCE
UNIT REPORT
Thank you for your recent letter concerning
the publication of the third annual report from the CJD Surveillance Unit. I am
sorry that you are dissatisfied with the way in which this report was
published.
The Surveillance Unit is a completely
independant outside body and the Department of Health is committed to publishing
their reports as soon as they become available. In the circumstances it is not
the practice to circulate the report for comment since the findings of the
report would not be amended. In future we can ensure that the British Deer
Farmers Association receives a copy of the report in advance of
publication.
The Chief Medical Officer has undertaken to
keep the public fully informed of the results of any research in respect of CJD.
This report was entirely the work of the unit and was produced completely
independantly of the the Department.
The statistical results reqarding the
consumption of venison was put into perspective in the body of the report and
was not mentioned at all in the press release. Media attention regarding this
report was low key but gave a realistic presentation of the statistical findings
of the Unit. This approach to publication was successful in that consumption of
venison was highlighted only once by the media ie. in the News at one television
proqramme.
I believe that a further statement about the
report, or indeed statistical links between CJD and consumption of venison,
would increase, and quite possibly give damaging credence, to the whole issue.
From the low key media reports of which I am aware it seems unlikely that
venison consumption will suffer adversely, if at all.
http://web.archive.org/web/20030511010117/http://www.bseinquiry.gov.uk/files/yb/1994/10/00003001.pdf
Tuesday, October 15, 2013
AD.47: Polymorphic variations
in cervid prion protein affect CWD agent properties
Friday, April 26, 2013
INDIANA Republican State
Sen. Travis Holdman Senate Bill 373 ag-gag WILL PUT HUMANS AT RISK
http://madcowusda.blogspot.com/2013/04/indiana-republican-state-sen-travis.html
Saturday, September 21, 2013
Westland/Hallmark: 2008 Beef Recall A Case Study by The
Food Industry Center January 2010 THE FLIM-FLAM REPORT
FOR 4 YEARS, THE USDA NSLP fed our children all across
the USA, from school to school, county to county, state to state, the most high
rish cattle for mad cow type disease i.e. the TSE prion disease, they fed our
children dead stock downer cows. did your children consume this, and will you
have to wait 5 decades to see if they become exposed and go clinical with CJD
TSE prion disease ???
Monday, September 02, 2013
Atypical BSE: role of the E211K prion
polymorphism
Research Project: TRANSMISSION,
DIFFERENTIATION, AND PATHOBIOLOGY OF TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHIES
Location: Virus and Prion Research Unit
Sunday, September 1, 2013
Evaluation of the Zoonotic Potential of
Transmissible Mink Encephalopathy
We previously described the biochemical
similarities between PrPres derived from L-BSE infected macaque and cortical MM2
sporadic CJD: those observations suggest a link between these two uncommon prion
phenotypes in a primate model (it is to note that such a link has not been
observed in other models less relevant from the human situation as hamsters or
transgenic mice overexpressing ovine PrP [28]). We speculate that a group of
related animal prion strains (L-BSE, c-BSE and TME) would have a zoonotic
potential and lead to prion diseases in humans with a type 2 PrPres molecular
signature (and more specifically type 2B for vCJD)
snip...
Together with previous experiments performed
in ovinized and bovinized transgenic mice and hamsters [8,9] indicating
similarities between TME and L-BSE, the data support the hypothesis that L-BSE
could be the origin of the TME outbreaks in North America and Europe during the
mid-1900s.
Sunday, August 11, 2013
Creutzfeldt-Jakob
Disease CJD cases rising North America updated report August 2013
***
Creutzfeldt-Jakob Disease CJD cases rising North America with Canada seeing an
extreme increase of 48% between 2008 and 2010
http://creutzfeldt-jakob-disease.blogspot.com/2013/08/creutzfeldt-jakob-disease-cjd-cases.html
Sunday, October 13, 2013
CJD TSE Prion Disease
Cases in Texas by Year, 2003-2012
http://creutzfeldt-jakob-disease.blogspot.com/2013/10/cjd-tse-prion-disease-cases-in-texas-by.html
Sunday, October 13, 2013
Prion Disease Cases in
Texas by Year, 2003-2012
http://cjdtexas.blogspot.com/2013/10/prion-disease-cases-in-texas-by-year_13.html
Wednesday, October 09, 2013
WHY THE UKBSEnvCJD
ONLY THEORY IS SO POPULAR IN IT'S FALLACY, £41,078,281 in compensation
REVISED
http://creutzfeldt-jakob-disease.blogspot.com/2013/10/why-ukbsenvcjd-only-theory-is-so.html
Tuesday, September 24, 2013
NORDION (US), INC., AND BIOAXONE BIOSCIENCES,
INC., Settles $90M Mad Cow TSE prion Contamination Suit Cethrin(R)
Case 0:12-cv-60739-RNS Document 1 Entered on
FLSD Docket 04/26/2012 Page 1 of 15
TSS
Indiana’s attorney
general is going to appeal a recent court ruling that appears to open the door
to more high-fence deer hunting in the state, and restrict the Department of
Natural Resources’s authority to enforce state hunting laws on the private
deer-hunting preserves.
Attorney General Greg Zoeller’s office filed notice of appeal today, two weeks after Harrison County Circuit Court Judge John Evans issued a ruling in favor of Rodney Bruce, who operates Whitetail Bluff, a preserve near Corydon.
In 2005 Bruce sued after the DNR tried to shut down his operation and the 11 other high-fence hunting preserves in the state at the time.
Owens ruling says that the DNR overstepped its authority, and that deer at facilities such as Bruce’s “are privately owned and not the property of the people of the state of Indiana,” so they’re not subject to the DNR’s oversight.
Evans’ ruling troubled animal rights activists and hunting groups who say they’re worried that it could severely restrict the ability of state wildlife officials to enforce state hunting laws inside the fenced enclosures, which they disparagingly call “canned hunts.” They say Evans ruling also challenged the notion that wildlife is owned by the public and only in special, tightly regulated circumstances can private individuals possess and kill the animals.
While the actual legal arguments behind the attorney general’s appeal haven’t yet been disclosed, both preserve operators and their critics hope the state appellate court clarifies the matter. Two judges have recently issued conflicting rulings on hunting preserves.
The Harrison County decision came 10 months after a judge in Owen County threw out a similar challenge by a preserve owner.
Attorney General Greg Zoeller’s office filed notice of appeal today, two weeks after Harrison County Circuit Court Judge John Evans issued a ruling in favor of Rodney Bruce, who operates Whitetail Bluff, a preserve near Corydon.
In 2005 Bruce sued after the DNR tried to shut down his operation and the 11 other high-fence hunting preserves in the state at the time.
Owens ruling says that the DNR overstepped its authority, and that deer at facilities such as Bruce’s “are privately owned and not the property of the people of the state of Indiana,” so they’re not subject to the DNR’s oversight.
Evans’ ruling troubled animal rights activists and hunting groups who say they’re worried that it could severely restrict the ability of state wildlife officials to enforce state hunting laws inside the fenced enclosures, which they disparagingly call “canned hunts.” They say Evans ruling also challenged the notion that wildlife is owned by the public and only in special, tightly regulated circumstances can private individuals possess and kill the animals.
While the actual legal arguments behind the attorney general’s appeal haven’t yet been disclosed, both preserve operators and their critics hope the state appellate court clarifies the matter. Two judges have recently issued conflicting rulings on hunting preserves.
The Harrison County decision came 10 months after a judge in Owen County threw out a similar challenge by a preserve owner.
INDIANA
1/22/2013 11:11:00 AM
6 deer
missing from farm pose health risk to state herds
Aubrey
Woods, Tribune
Indiana
Department of Natural Resourcescontinues to search for six ear-tagged deer in
Jackson County and neighboring Bartholomew, Jennings and Scott counties. Those
deer are among the 20 that escaped from a captive cervid facility — or deer farm
— in 2012, a spokesman with the state Department of Natural Resources said
Friday.
“Of the six,
one is a buck, which had a yellow ear tag with the No. 47,” Phil Bloom
said.
Bloom said
the buck is the only one of the six missing deer imported from a site in
Pennsylvania where deer have tested positive for chronic wasting
disease.
“He has been
exposed to it, but that doesn’t mean he has chronic wasting disease,” Bloom
said. “There is no live test for chronic wasting disease. That can’t be
determined until the deer is dead and tissue has been
tested.”
Bloom said
the other five deer that remain free are classified as exposed to chronic
wasting disease through their association with the buck.
Of
particular interest are any deer with a yellow ear tag bearing the prefix “IN
764” followed by another four numbers or any deer with a yellow ear tag and two
numbers on it.
Natural
Resources staff will assist in taking the deer carcass to Purdue University for
testing at the Indiana Animal Disease Diagnostic Laboratory.
Of the
remaining 20 that escaped from the deer farm, the owner recaptured 11 shortly
after the escape.
The location
and ownership of the deer farm have not been released, based on guidelines of
the Indiana Board of Animal Health, but the owner is cooperating, Bloom
said.
The escape
occurred after a tree fell on a fence at the deer farm, Bloom
said.
Of the
remaining nine deer that escaped, one was struck and killed by a vehicle, a
second was shot and killed by a bow hunter during archery season and a third was
killed during a special deer hunting session the first weekend of January, Bloom
said.
The owner of
the farm continues to help search for the remaining six and has been cooperative
with the state, Bloom said.
State
officials believe the buck is still alive because people have reported seeing
it, Bloom said.
“Although it
is conceivable it may have been killed,” he said.
Hunters also
may have killed some of the five missing deer and just not reported their deaths
to the state as required, Bloom said.
Bloom said
there are no special hunting seasons planned to find the remaining
deer.
“We’re
making an all-out effort to find the deer,” Bloom said.
Local hunter
Chuck Brenner of Freetown said the state is being too complacent about an issue
that could have long-range effects on the deer population,
however.
“I’m not
just a hunter,” Brenner said. “I’m a conservationist. We need to manage our
herds.”
Brenner said
chronic wasting disease could wipe out herds and cost the state millions of
dollars down the road.
“We should
hunt them down and find them whether it’s deer season or not,” Brenner
said.
He said
there hasn’t been much publicity about the missing deer and that local hunters
could help with the search.
“They need
to ask the local hunters for help,” Brenner said. “They could put out trail
cameras. If nothing else, the state could put a bounty on
them.”
Brenner said
the deer are clearly marked and there could be a special hunt just for the six
deer that are ear-tagged.
Bloom said
the state wants to make sure that the deer are tracked down, tested and
determined if they’ve been exposed to chronic wasting
disease.
“It could
have some far-reaching ramifications,” he said.
So far the
disease has never been found in Indiana, which has been testing for the disease
since 2002. More than 10,000 deer killed during hunts or by vehicles have been
tested and the disease has not been detected, Bloom said.
The tags on
those missing deer are expected to tell officials whether the animals have any
possible connection with a captive facility in Pennsylvania where chronic
wasting disease was confirmed this winter, Bloom said. That report made
Pennsylvania the 23rd state in which it has been found.
Cervid or
wild game farms in Indiana can raise deer, elk and moose for several reasons,
Bloom said. The animals can be processed at a slaughterhouse and sold as meat,
sold for pets or sold to high-fenced hunting reserves.
Copyright ©
2013 The Tribune
Editor, John
C. DePrez Jr.; Executive Editor, Carol Rogers; Publishers: IBRC and
IAR
Long kills
controversial fenced hunting bill
February 1,
2012 | Filed under: Issues,Top stories | Posted by: Lesley Weidenbener
By Lesley
Weidenbener
The
Statehouse File
INDIANAPOLIS
– An effort to legalize fenced deer hunting is dead for this session.
...
snip...
see full text ;
Sunday, January 27, 2013
Indiana 6 deer missing from farm
pose health risk to state herds INDIANA
- Richard Rubenstein1,*,
- Binggong Chang1,
- Perry Gray2,
- Martin Piltch2,
- Marie S. Bulgin3,
- Sharon Sorensen-Melson3 and
- Michael W. Miller4
+ Author Affiliations
ABSTRACT
Prion
diseases, also known as transmissible spongiform encephalopathies, are fatal
neurodegenerative disorders. Low levels of infectious agent and limited,
infrequent success of disease transmissibility and PrPSc detection
have been reported with urine from experimentally infected clinical cervids and
rodents. We report the detection of prion disease-associated seeding activity
(PASA) in urine from naturally and orally infected sheep with clinical scrapie
agent and orally infected preclinical and infected white-tailed deer with
clinical chronic wasting disease (CWD). This is the first report on PASA
detection of PrPSc from the urine of naturally or preclinical
prion-diseased ovine or cervids. Detection was achieved by using the surround
optical fiber immunoassay (SOFIA) to measure the products of limited serial
protein misfolding cyclic amplification (sPMCA). Conversion of PrPC
to PrPSc was not influenced by the presence of poly(A) during sPMCA
or by the homogeneity of the PrP genotypes between the PrPC source
and urine donor animals. Analysis of the sPMCA-SOFIA data resembled a linear,
rather than an exponential, course. Compared to uninfected animals, there was a
2- to 4-log increase of proteinase K-sensitive, light chain immunoglobulin G
(IgG) fragments in scrapie-infected sheep but not in infected CWD-infected deer.
The higher-than-normal range of IgG levels found in the naturally and
experimentally infected clinical scrapie-infected sheep were independent of
their genotypes. Although analysis of urine samples throughout the course of
infection would be necessary to determine the usefulness of altered IgG levels
as a disease biomarker, detection of PrPSc from PASA in urine points
to its potential value for antemortem diagnosis of prion diseases.
FOOTNOTES
- Received 13 May 2011.
- Accepted 14 June 2011.
- ↵*Corresponding author. Mailing address: Downstate Medical Center, Departments of Neurology and Physiology/Pharmacology, Box 1213, 450 Clarkson Avenue, Brooklyn, NY 11203. Phone: (718) 270-2019. Fax: (718) 270-2459. E-mail: richard.rubenstein@downstate.edu.
-
↵▿ Published ahead of print on 29 June 2011.
Date: Sat, 20 Jul 2002 09:43:10 -0700
From: "Terry S. Singeltary Sr."
Subject: MAD DEER/ELK DISEASE AND POTENTIAL SOURCES
Date: Sat, 25 May 2002 18:41:46 -0700
From: "Terry S. Singeltary Sr."
Reply-To: Bovine Spongiform Encephalopathy To: BSE-L@uni-karlsruhe.de
######## Bovine Spongiform Encephalopathy #########
MAD DEER/ELK DISEASE AND POTENTIAL SOURCES
INDIANA Republican State Sen. Travis Holdman Senate Bill 373 ag-gag WILL PUT HUMANS AT RISK
http://madcowusda.blogspot.com/2013/04/indiana-republican-state-sen-travis.html
Saturday, September 21, 2013
Westland/Hallmark: 2008 Beef Recall A Case Study by The
Food Industry Center January 2010 THE FLIM-FLAM REPORT
FOR 4 YEARS, THE USDA NSLP fed our children all across
the USA, from school to school, county to county, state to state, the most high
rish cattle for mad cow type disease i.e. the TSE prion disease, they fed our
children dead stock downer cows. did your children consume this, and will you
have to wait 5 decades to see if they become exposed and go clinical with CJD
TSE prion disease ???
Sunday, August 11, 2013
Creutzfeldt-Jakob Disease CJD cases rising North America updated report August 2013
*** Creutzfeldt-Jakob Disease CJD cases rising North America with Canada seeing an extreme increase of 48% between 2008 and 2010
http://creutzfeldt-jakob-disease.blogspot.com/2013/08/creutzfeldt-jakob-disease-cjd-cases.html
Sunday, October 13, 2013
CJD TSE Prion Disease Cases in Texas by Year, 2003-2012
http://creutzfeldt-jakob-disease.blogspot.com/2013/10/cjd-tse-prion-disease-cases-in-texas-by.html
Sunday, October 13, 2013
Prion Disease Cases in Texas by Year, 2003-2012
http://cjdtexas.blogspot.com/2013/10/prion-disease-cases-in-texas-by-year_13.html
Wednesday, October 09, 2013
WHY THE UKBSEnvCJD ONLY THEORY IS SO POPULAR IN IT'S FALLACY, £41,078,281 in compensation REVISED
http://creutzfeldt-jakob-disease.blogspot.com/2013/10/why-ukbsenvcjd-only-theory-is-so.html
posted by Terry S. Singeltary Sr. at
9:51 PM
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