Thursday, December 13, 2012

HUNTERS FEELING THE HEAT Houston Chronicle December 13, 2012 OUTDOORS not talking about CWD in Texas


Houston Chronicle

December 13, 2012


A break in the unusually warm, dry weather could bolster late deer-season prospects

by Shannon Tompkins

Texas deer hunters welcomed the for-real cold front that swept across the state earlier this week bringing with it two crucial items that have been in short supply this deer season: cold temperatures and rain. ...

full page article print @

HUNTERS FEELING THE HEAT Houston Chronicle December 13, 2012 OUTDOORS



Greetings Texas Hunters, Friends, and Neighbors, Houston Chronicle et al,

Once again, our prolific sports writer for the Houston Chronicle, Shannon Tompkins, writes another wonderful FULL PAGE piece on Texas hunting and Texas deer, without so much as whispering a hint of what’s going on about Chronic Wasting Disease CWD in the cervid population in Texas. so I guess I have to do it for them. for years and years, Shannon was writing about CWD from state to state, writing how Texas had escaped this deadly pathogen, until that fateful day in 2012 in Texas, when NEW MEXICO FORCED or FINALLY HUMILIATED TAHD into CWD testing, where I told them to start testing a decade (10 years ago). nope, Shannon has become silent on this topic, since his one short article about it. so, who is paying Shannon, the Chronicle, or the captive shooting pen deer farms and or 98% of the private hunting land owners in Texas, with big game private hunting trips, or what, as to NOT to write about CWD? CWD waltzing across the border from New Mexico for a decade and nobody is talking much about it. captive shooting pens in Texas rampant, CWD in captive shooting pens all over the U.S. are going down with CWD, and in Texas ??? age restrictions on testing for CWD? we know CWD can develop in 4 to 5 month old cervids. only testing in captive shooting pens when a dead deer is found? that is if you don’t use the SSS policy the USDA is so fond of using covering up mad cow disease BSE in the USA. _voluntary_ CWD surveillance, testing, and monitoring ??? how did that work out for us with the infamous August 4, 1997 partial and voluntary mad cow feed ban? I will tell you, one decade (10 YEARS) post partial and voluntary mad cow feed ban, 2007, in one week of fda recall alone, 10 MILLION POUNDS OF BANNED BLOOD LACED MEAT AND BONE MEAL WENT OUT INTO COMMERCE TO BE FED OUT. 2006 was a banner year as well. since 2007, these type documents on this type violations are not available anymore to the public, with the new reporting system put in. in my opinion, the Chronicle and Mr. Tompkins are doing a disservice to it’s readers and hunters in the state of Texas. very sad and disturbing, as to how Shannon can write for years and years how bad CWD is, until it is documented in Texas, and then see how silent Shannon became on CWD.
very disturbing to me. opinion, thank you. ...terry

Wednesday, November 07, 2012

Chronic Wasting Disease CWD, Texas, Houston Chronicle Shannon Tomkins 1998 - 2012 what happened ???


Tompkins: There are a lot of reasons to be concerned about CWD

SHANNON TOMPKINS, Copyright 2002 Houston Chronicle Published 5:30 a.m., Thursday, March 14, 2002

Today, most Texas deer hunters probably yawn at the mention of Chronic Wasting Disease. After all, the number of wild deer documented as killed, nationwide, by the unusual malady probably is less than annually are crushed by tractor-trailer rigs scorching Interstate 10 between Kerrville and Fort Stockton.

And, so far, no cases of the fatal, incurable, communicable, brain-destroying cervid disease have been documented in Texas.

What's so bad about a little-understood disease responsible for the death of scattered pockets of deer in a handful of Rocky Mountain states?

If Texas' deer herd survived screwworms and can thrive despite endemic bluetongue and anthrax and even the constant gnawing away of habitat, then why worry about a little Chronic Wasting Disease?

There is abundant reason to be concerned.

CWD carries potential for incredible impacts on Texas' 4 million deer, its half-million deer hunters, the hunting-based economy of rural areas and private landowners and even the future of the state agency responsible for overseeing those deer and all other natural resources.

Just how seriously many Texas wildlife managers and those with economic or other interest in deer take the CWD threat was manifestly evident over the past week.

In the wake of news that Wisconsin officials had discovered CWD in three of 26 wild deer taken by hunters in a small area of that state, Katharine Armstrong Idsal, presiding officer of the Texas Parks and Wildlife Commission, called an emergency meeting of the TPW Commission to address the issue of deer importation into Texas.

A proposal to suspend all imports of deer into Texas was, and is, on the TPW Commission's agenda for its scheduled April 4 meeting, with the recommendation having been triggered by discovery over the past few months of CWD in wild deer in Nebraska and South Dakota.

The emergency TPW Commission meeting was arranged Friday, the day the Texas Wildlife Association, a politically active, landowner-based organization, sent to the governor, members of the Legislature and the TPW Commission a resolution calling for sealing the state's borders to deer imports because of the chance some might be carrying CWD.

At the TPW Commission's hastily called Monday meeting, the group approved and adopted an emergency rule prohibiting importation of white-tailed and mule deer into Texas.

That emergency rule, which is effective for 45 days, took effect Tuesday. It is the first time the TPW Commission has used its emergency rule-making authority.

Justifications for the emergency action were laid out in the preamble to the regulation change. CWD, the document states, "constitutes a direct threat to wild deer populations in Texas and therefore to the multi-billion dollar hunting industry, as well as a potential threat to human health, safety and welfare."

To understand the threat to deer and, perhaps, public health and the subsequent potentially devastating impact on Texas' deer-based economy, it's necessary to understand CWD.

CWD is one of a group of transmissible spongiform encephalopathies (TSE) diseases that destroy brain cells. Triggering the destruction is a prion, an abnormal form of protein. The prion mutates normal cellular protein into the abnormal form.

This "eats away" at the brain and damages an infected animal's ability to maintain normal functions such as converting food and body fat to energy.

Animals suffering from CWD begin wasting away as their body tries to convert protein to energy, a very inefficient process.

Eventually, the animal loses motor control and even goes blind, giving rise to the pitiful "blind staggers" seen in livestock suffering from CWD's close relative, Bovine Spongiform Encephalopathy, better known as "Mad Cow Disease."

Death is inevitable and horrible.

Scientists know relatively little about CWD.

"We don't really know what triggers it. Does the prion create the disease or does the disease create the prion?" said Jerry Cooke, game mammal branch chief of the Texas Parks and Wildlife Department's wildlife division. "What we do know is that it is transmissible to other cervids."

First documented in the 1960s in penned herds in Colorado, CWD "jumped" into the wild cervid population there, being confirmed in wild deer and elk in the 1980s.

A common suspicion is that CWD is a mutated form of "scrapie," a TSE long confined to sheep.

There is some evidence that the cervids in the Colorado pen where CWD was first documented were fed protein feeds containing sheep parts and that those parts could have contained brain material infected with scrapie.

One of the scrapie-triggering prions might have mutated just enough to break the molecular barrier of a deer's brain cell, and the disease was off and running.

Scientists are convinced CWD is spread by close contact between uninfected and infected animals. That can happen between animals in a pen or behind a fence, or by nose-to-nose contact between deer or elk inside the fence and those outside the enclosure.

From Colorado, CWD spread throughout the northwest corner of the state into wild herds in Wyoming and Nebraska.

Its spread was accelerated over the past decade by a burgeoning market in deer and elk triggered by elk farming and deer ranching.

Thousands of deer and elk are bought and transported each year, most to penned facilities where they are either raised for food or, in the case of white-tailed deer, used in an effort to produce bucks with large antlers to feed a market in trophy hunting.

To test for CWD, brain tissue is needed. And such tissue samples can be obtained only if the animal is dead.

Plus, getting rid of the disease has proved difficult, if not impossible, even in penned facilities.

In at least one case, a penned facility holding CWD-infected deer was "depopulated" (the animals slaughtered and destroyed) and the site left with no animals for three years.

When uninfected deer were placed in the pens, they contracted CWD.

As deer and elk from areas with CWD have been traded and transported across the nation, they have brought the disease with them

Currently, CWD-infected, free-ranging deer have been confirmed in Colorado, Nebraska, Wyoming, South Dakota and Wisconsin, plus the Canadian province of Saskatchewan.

CWD has been found in captive herds in Saskatchewan, Colorado, South Dakota, Nebraska, Kansas, Oklahoma and Montana.

Texas has been a big player in the deer trade over the past decade, as hundreds of deer-breeding facilities have sprung up in the state to feed the interest in building bucks with bigger antlers.

Today, more than 450 individuals in Texas hold a TPWD-issued "scientific breeder permit" allowing them to manipulate deer. Some of these breeders and other landowners over the past four years have imported 2,107 deer from outside Texas.

Because deer can be traded so often -- a deer may be sold as a fawn in Nebraska to a broker in Missouri who sells it to a breeder in Pennsylvania who sells it to a landowner in Texas -- it often is nearly impossible to determine the provenance of individual animals.

Whether any of the thousands of deer imported into Texas over the past decade carried CWD remains an unsettling question.

Texas has no CWD-testing program for wild deer and only a voluntary program for elk and other animals under the jurisdiction of the Texas Animal Health Commission.

"Ten years ago, elk and deer (imported into Texas) were not regulated at all," said Dr. Ken Waldrup, an epidemiologist with the Texas Animal Health Commission and one of the agency's point men on CWD. "If Texas doesn't already have CWD, then I say that proves that God is a Texan.

"For everyone's sake, I sure hope He is."

CWD has not been proved to be transmissible to any animal other than deer and elk.

But that was the original thought with BSE, which did "jump" into humans who ate BSE-infected meat in Europe and contracted Creutzfeldt-Jakob Disease (CJD), the human form of TSE. CJD, like CWD and BSE, is fatal, incurable and untreatable. It is blamed for at least 80 deaths in Europe.

While there is no proof CWD can jump to humans, there is no absolute proof it can't if given enough opportunities.

And that issue scares wildlife managers.

If CWD shows up in a deer herd and the deer-hunting public gets spooked about the possibility -- no matter how tiny -- that by cleaning or eating a deer they will contract CJD and face a certain and horrible death, they could, en masse, abandon deer hunting.

This could destroy the $2 billion-plus deer hunting economy in Texas.

Also, if deer hunters abandon their recreation, natural resource agencies such as TPWD, which depend almost entirely on hunting license fees to fund their diverse wildlife programs, would be maimed, perhaps mortally.

"It's not the immediate impact on the deer herds that (is) the most frightening thing about CWD," Waldrup said. "It's the secondary impacts that are really scary.

"People better just pray it doesn't show up here. If it does, things could get very ugly."

Shannon Tompkins covers the outdoors for the Chronicle. His column appears Thursdays, Fridays and Sundays.

now, don’t get me wrong Mr. Tompkins, you have written some great articles on the wild and on the fisheries, and on CWD in the past. see ;

Sunday, November 30, 2008

Commentary: Crimes hurt essence of hunting

Commentary on Houston Chronicle article [below] by Dr. Thomas Pringle

Date: Fri, 10 May 2002 11:03:29 –0800

Subject: nice cwd reporting Shannon,

My compliments on these superb CWD Houston Chronical articles: not mincing words, they display an excellent -- and most rare -- journalistic understanding of the origin and continuing spread of CWD. (A couple of technical points were not quite on target, see bottom.)

It is really refreshing to see in print the probable origin as sheep scrapie-to-penned cervids in 1967 at Foothills Research Station, after decades of relentless PR out of Colorado DOW seeking to distance itself from responsibility (and liability). Facility workers at Colorado Dept of Wildlife commented on the similarity to scrapie already in 1967 but never autopsied any of their many dead research animals until 1979, discovering immediately an obvious spongiform encephalopathy.

By that time of course, release to the wild and transfer of surplus animals to zoos, game farms, and sister facilities had seeded widespread dissemination of the disease. This was subsequently aggravated by the explosive growth in game farms and intra- and inter-state cervid shipping, which at industry insistence was in essence unregulated (eg regulated by state ag dept boosters). It is not just the shoot-deer-in-a-barrel industry --elk velvet nutriceutical was never tested by anyone for abnormal prions despite its troublesome composition (the market collapsed from live CWD exported to Korea).

DOW itself did nothing to change its practises or control the disease until very recently. Only last year, in the face of published evidence [below] that the disease is expected to transfer to humans at the same low efficency as BSE (129 human deaths to date), did they back off from encouraging human consumption of venison from the endemic area. Nebraska fish and game even offered a deer-neck stew recipe on its web site, even though spinal cord was long known to have high infectious titres.

State fish and game depts are basically unfenced game farms. They have a commercial concession that allows them earn a salary from sale of antler tags. This motivates them to set up winter feeding stations, watering holes, salt blocks, control predators, fight CWD testing, anything and everything that increases numbers and leads to more or continued sales. Unfortunately, practises leading to high cervid concentrations and testing avoidance are highly conducive to the spread of CWD.

States such as Montana require testing of every game farm cervid dead for any reason and an accounting of each animal's provenance and disposition; other states adopt a "don't look, don't find" policy of testing avoidance with no monitoring whatsoever of facilities. Absence of evidence is not evidence of absence when it comes to TSEs. This disease just does not go away on its own, be it kuru in New Guinea or scrapie in the US.

Given the numbers of Texas game farms, massive importation statistics, and the high likelihood of trace-backs to affected facilities, it would be most surprising if CWD were not already entrenched in Texas along the lines of Wisconsin. It really questionable if stonewalling really is in the industry's best interest -- who is going to hunt in a state that fears to test? The longer infectious foci are allowed to operate, the greater the probability of multiple introductions into wild deer. To ban imports (only after everyone has finished importing all they want) just locks the barn door after the horse is long gone.

Half-measures on prion diseases are worse than no measures because they put off the day of reckoning while exacerbating it immensely. Wisconsin's hasty policy of culling 15,000 wild deer, yet business as usual (no testing, no trace-backs, no inspection, no recordkeeping, no culls) at its sacrosanct 535 game farms. will result in CWD in perpetuity. The focus is on temporary abatement for purposes of hunter reassurance. Dr. Charles Southwick is a good source of scientific information on cwd control strategies.

A few technical notes. First, the word mutation is reserved for genetic change affecting DNA. It is not applicable to mere protein conformational changes and fibril formation seen in amyloid diseases such as Alzheimer and CWD. Mutation has been ruled out in CWD amplification. The prion gene of hundreds of CWD and non-CWD animals have been sequenced by Dr. O'Rourke at Pullman. There is no counterpart to the mutations that cause 15% of human CJD, much to the disappointment of DOW.

No TSE has ever been seen in natural populations of any wild animal anywhere in the world, making Colorado's story of a natural pocket (by coincidence located adjacent to Foothills and Sybille research stations) a bit far-fetched. Now by golly another natural pocket has flared up next to a game farm in Wisconsin. How about the supposed natural pocket adjacent to the massively infected game farm in the Black Hills -- despite its import history, the industry PR firm in Ketchum turned this around 180 degrees -- now it's the wild animals infecting innocent game farms!?! There has invariably been a nexus to intensive livestock operations, be that cows fed rendered cows, mink farms fed downer cows or deer quartered in a scrapie research facility.

Second, the "best available scientific evidence" upon which public policy is normally based (more studies are needed, they always are, but something must be used for the interim) is that published by Byron Caughey's group at Rocky Mtn labs (after two years of delay by co-author Mike Miller of DOW who controlled sample access). A proxy test was used since human volunteers cannot be considered. Transmission efficiencies to human were similar to BSE -- low, but hardly reassuring given England's experience.

Third, CWD has already been experimentally transferred to 6-7 species including rodents, primates, and bovids, as published in peer-reviewed scientific journals. The first round of transmission can be inefficient in TSEs; after that, no species barrier. It is really the human-to-human second round (plasma donation, childhood vaccines, cornea transplants) that has cause the greatest consternation in England. A Ft. Collins hunter/blood donor with preclinical cwd-induced CJD would have no idea he is ill.

It is currently impossible to test humans for cwd-induced CJD because there is no known signature. Rises in baseline CJD cannot be monitored, contrary to CDC, because of very large numbers of missed diagnoses, swings in ascertainment effort, and diagnostic changes.

Best wishes and keep up the good work! Tom

Dr. Thomas Pringle Sperling Biomedical Foundation 3295 Kincaid St. Eugene, OR 97405

CWD archives

Wisconsin latest to be hit by deer brain disease

May 10, 2002

The Houston Chronicle by Shannon Tompkins

Wisconsin drew the black bean in the continent's expanding war with chronic wasting disease, and that simple twist of fate promises to be expensive and painful for the state's deer and human populations. It also serves as a sobering study for Texas in what can happen when the poorly understood but invariably fatal brain disease shows up in a state's wild deer herd.

Just three months after CWD was documented in a handful of white-tailed deer taken by hunters in southwestern Wisconsin, the state is preparing to kill thousands of deer; Gov. Scott McCallum is calling for a special session of the state Legislature to address the issue; politicians are asking for millions of dollars to fight CWD spread; and the hunting-based economies of the region are preparing to take a stunning blow.

Add to that the uncertainty many of Wisconsin's 700,000 deer hunters are expressing about the safety of eating venison, and you have the future of that state's deer and deer hunting hanging in the balance. CWD is a recently discovered transmissible spongiform encephalopathy that affects deer and elk. It is similar to the TSE that causes "mad cow disease" in livestock, and which in Europe "jumped" from infected livestock to humans as a variation of the TSE Creutzfeldt-Jakob disease in humans.

The disease manifests itself via prions, or mutant proteins, which cause deterioration of brain cells. The effects include loss of weight and muscle control, blindness and dementia. There is no treatment and the disease is fatal.

CWD has been proved transmissible between deer and elk, but it has not been shown to be transmittable to humans. But neither has it been proved non-transmittable. The possibility, however minuscule, exists that a human could contract the fatal disease.

Since it was discovered in 1967 in wild deer in the northeast corner of Colorado, CWD has been a mystery. How it came to exist remains a question, but the most accepted theory is that it is a mutation of a TSE called "scrapie" found in sheep. A Colorado research facility that housed sheep, deer and elk in close contact is assumed to have been the genesis of CWD.

The disease for most of the past three decades seems to have remained localized in a small area of Colorado.

Interstate trade in "farmed" live elk and deer, some of which were infected with CWD, is assumed to have begun the diseases' spread to other states.

CWD has been identified in a half-dozen states and a couple of Canadian provinces, almost always associated with penned elk or deer.

The discovery of CWD in three wild deer in Wisconsin during a routine sampling of hunter-taken animals stunned most wildlife scientists and managers.

The disease never had been documented east of the Mississippi River, and never in an area where deer densities are as high as they are in Wisconsin.

The closest CWD cases were more than 900 miles from Wisconsin.

The discovery triggered a rush of states closing their borders to importation of deer and elk.

Texas, which has for years been one of the major players in live deer and elk traffic, shut its borders to all importation of deer and elk within a couple of weeks of the Wisconsin discovery.

Wisconsin officials began addressing the issue by killing and testing 516 deer in the area that produced CWD-infected animals. (There is no certified live-animal test for CWD; animals must be killed and brain or brain stem tissue analyzed to document infection.)

When 11 of those 516 deer proved infected with CWD, the state's Department of Natural Resources and politicians knew they had a severe problem.

In an effort to prevent the spread of CWD, Wisconsin wildlife officials are proposing to kill every deer in a 287-square-mile (about 184,000 acres) area where the infected deer have been found.

That will involve killing 14,000-15,000 deer, officials estimate.

Just how that will be accomplished remains a question. But the slaughter almost certainly will begin next month.

CWD has become a white-hot political issue in the state, where fingers are being pointed at agriculture officials who disregarded warnings about the possibility of CWD-infected deer being brought into the state.

McCallum said this week he will call a special session of the state's Legislature to address CWD-related issues such as regulation of feeding wild deer, a practice that crowds deer together and is suspected of making it easier for CWD to spread.

The Wisconsin Legislature has approved spending $ 4.4 million this year to fight CWD. Officials say they need at least $ 22.5 million over the next three years to contain CWD.

McCallum is asking the federal government for $ 18.5 million.

At least Wisconsin knows it has a CWD problem, and is addressing it. Other states, including Texas, probably have CWD-infected deer within their borders.

But because they do no testing for the disease, they have no evidence of its presence.

Other states are beginning to fashion CWD testing programs, though.

Iowa, which abuts the southwest corner of Wisconsin where the CWD-infected deer have been found, this week announced it will begin collecting brain tissue samples from road-killed deer and submitting them for CWD testing.

Iowa officials said they hope to collect 100-200 road-killed deer for sampling each month.

Texas has no CWD testing program.

But the Texas Deer Association, a trade group representing many of the state's 400-plus state-permitted deer and elk ranches, this past month promised to put together a voluntary CWD monitoring program in cooperation with the Texas Parks and Wildlife Department and Texas Animal Health Commission.

If the voluntary program is not accepted by TPWD and TAHC, the agencies could issue regulations for mandatory CWD testing.

The issue will be discussed at May 29-30 TPW Commission meetings in Austin.


Mr. Thomkins, and Houston Chronicle, I think your disregard for concern NOW, at least the same concern now, than you had back when the CWD TSE prion disease was not on the other foot, I think your silence is deafening now about Chronic Wasting Disease CWD, and very disturbing, and is doing an injustice to your readers.

I wasted 10 years or so, trying to get them to test, where New Mexico forced them to test, i.e. White Sands Missle range side of Texas, and there about. course, I did the same with mad cow disease too. to no avail. $$$

IT would be nice for you and the chronicle, to at least give the same concern for CWD as you did in the past when CWD was in other states, but not TEXAS. you can see here what one deer and one captive shooting pen can cost a state ;

THE states are going to have to regulate how many farms that are allowed, or every state in the USA will wind up being just one big private fenced in game farm. kind of like they did with the shrimping industry in the bays, when there got to be too many shrimp boats, you stop issuing permits, and then lower the exist number of permits, by not renewing them, due to reduced permits issued.

how many states have $465,000., and can quarantine and purchase there from, each cwd said infected farm, but how many states can afford this for all the cwd infected cervid game ranch type farms ???

11,000 game farms X $465,000., do all these game farms have insurance to pay for this risk of infected the wild cervid herds, in each state ???

Tuesday, December 20, 2011


The CWD infection rate was nearly 80%, the highest ever in a North American captive herd.

RECOMMENDATION: That the Board approve the purchase of 80 acres of land for $465,000 for the Statewide Wildlife Habitat Program in Portage County and approve the restrictions on public use of the site.


SUBJECT: Information Item: Almond Deer Farm Update



Monday, January 16, 2012


see full text and more here ;

Thursday, February 09, 2012


Volume 18, Number 3—March 2012

Samuel E. Saunders1, Shannon L. Bartelt-Hunt, and Jason C. Bartz

Author affiliations: University of Nebraska-Lincoln, Omaha, Nebraska, USA (S.E. Saunders, S.L. Bartelt-Hunt); Creighton University, Omaha (J.C. Bartz)


Occurrence, Transmission, and Zoonotic Potential of Chronic Wasting Disease

Originally recognized only in southeastern Wyoming and northeastern Colorado, USA, CWD was reported in Canada in 1996 and Wisconsin in 2001 and continues to be identified in new geographic locations (Figure 1, panel A).

CWD has been identified in free-ranging cervids in 15 US states and 2 Canadian provinces and in ≈100 captive herds in 15 states and provinces and in South Korea (Figure 1, panel B). Except in South Korea, CWD has not been detected outside North America. In most locations reporting CWD cases in free-ranging animals, the disease continues to emerge in wider geographic areas, and prevalence appears to be increasing in many disease-endemic areas.

Areas of Wyoming now have an apparent CWD prevalence of near 50% in mule deer, and prevalence in areas of Colorado and Wisconsin is <15 deer.="deer." div="div" in="in">

However, prevalence in many areas remains between 0% and 5% according to reports and data obtained from state and provincial wildlife agencies.

Prevalence in elk is lower than in deer but reaches 10% in parts of Wyoming.

Known risk factors for CWD include sex and age, and adult male deer show the highest prevalence (5).

Polymorphisms in the PrP (PRNP) gene appear to influence susceptibility in deer and elk (2,6,7), but remain less understood than the strong genetic influences for scrapie.


Most epidemiologic studies and experimental work have suggested that the potential for CWD transmission to humans is low, and such transmission has not been documented through ongoing surveillance (2,3). In vitro prion replication assays report a relatively low efficiency of CWD PrPSc-directed conversion of human PrPc to PrPSc (30), and transgenic mice overexpressing human PrPc are resistant to CWD infection (31); these findings indicate low zoonotic potential. However, squirrel monkeys are susceptible to CWD by intracerebral and oral inoculation (32). Cynomolgus macaques, which are evolutionarily closer to humans than squirrel monkeys, are resistant to CWD infection (32). Regardless, the finding that a primate is orally susceptible to CWD is of concern...


Reasons for Caution There are several reasons for caution with respect to zoonotic and interspecies CWD transmission. First, there is strong evidence that distinct CWD strains exist (36). Prion strains are distinguished by varied incubation periods, clinical symptoms, PrPSc conformations, and CNS PrPSc depositions (3,32). Strains have been identified in other natural prion diseases, including scrapie, BSE, and CJD (3). Intraspecies and interspecies transmission of prions from CWD-positive deer and elk isolates resulted in identification of >2 strains of CWD in rodent models (36), indicating that CWD strains likely exist in cervids. However, nothing is currently known about natural distribution and prevalence of CWD strains. Currently, host range and pathogenicity vary with prion strain (28,37). Therefore, zoonotic potential of CWD may also vary with CWD strain. In addition, diversity in host (cervid) and target (e.g., human) genotypes further complicates definitive findings of zoonotic and interspecies transmission potentials of CWD.

Intraspecies and interspecies passage of the CWD agent may also increase the risk for zoonotic CWD transmission. The CWD prion agent is undergoing serial passage naturally as the disease continues to emerge. In vitro and in vivo intraspecies transmission of the CWD agent yields PrPSc with an increased capacity to convert human PrPc to PrPSc (30). Interspecies prion transmission can alter CWD host range (38) and yield multiple novel prion strains (3,28). The potential for interspecies CWD transmission (by cohabitating mammals) will only increase as the disease spreads and CWD prions continue to be shed into the environment. This environmental passage itself may alter CWD prions or exert selective pressures on CWD strain mixtures by interactions with soil, which are known to vary with prion strain (25), or exposure to environmental or gut degradation.

Given that prion disease in humans can be difficult to diagnose and the asymptomatic incubation period can last decades, continued research, epidemiologic surveillance, and caution in handling risky material remain prudent as CWD continues to spread and the opportunity for interspecies transmission increases. Otherwise, similar to what occurred in the United Kingdom after detection of variant CJD and its subsequent link to BSE, years of prevention could be lost if zoonotic transmission of CWD is subsequently identified,...


Thursday, July 12, 2012


Subject: TEXAS MANDATORY CWD CHECK STATION LOCATIONS Nov. 23 – Dec. 10; 9 a.m. to 9 p.m.


(Nov. 23 – Dec. 10; 9 a.m. to 9 p.m.)

Monday, November 19, 2012

HUNTING: New protocols for mule deer hunting Texas Parks and Wildlife Department due to CWD

Friday, October 12, 2012

Texas Animal Health Commission (TAHC) is Now Accepting Comments on Rule Proposals for “Chronic Wasting Disease (CWD)”


Texas Animal Health Commission (TAHC)

Wednesday, October 03, 2012

TAHC Chronic Wasting Disease Rule What you need to know

Wednesday, September 26, 2012

TPWD Gearing Up for CWD Response during Deer Season

Monday, September 17, 2012


Friday, September 07, 2012

Texas Wildlife Officials Considering New Deer Movement Rules in Response to CWD

Thursday, July 12, 2012


Wednesday, July 11, 2012 Brain-eating disease found in Texas deer

Tuesday, July 10, 2012

Chronic Wasting Disease Detected in Far West Texas

Saturday, July 07, 2012

TEXAS Animal Health Commission Accepting Comments on Chronic Wasting Disease Rule Proposal

Considering the seemingly high CWD prevalence rate in the Sacramento and Hueco Mountains of New Mexico, CWD may be well established in the population and in the environment in Texas at this time.

Wednesday, June 13, 2012

TAHC Modifies Entry Requirements Effective Immediately for Cervids DUE TO CWD


Saturday, June 09, 2012

USDA Establishes a Herd Certification Program for Chronic Wasting Disease in the United States

Friday, September 07, 2012

Texas Wildlife Officials Considering New Deer Movement Rules in Response to CWD

Friday, June 01, 2012


Tuesday, May 01, 2012 Texas deer breeder gets fine, probation illegally importing live whitetail deer three times in 2007

Monday, March 26, 2012

Texas Prepares for Chronic Wasting Disease CWD Possibility in Far West Texas

Monday, March 26, 2012


Sunday, October 04, 2009


Subject: CWD NEW MEXICO RECORDS IT'S 19 CASE (near Texas border again)

From: "Terry S. Singeltary Sr." flounder9@VERIZON.NET

Reply-To: Bovine Spongiform Encephalopathy BSE-L@LISTS.AEGEE.ORG

Date: Wed, 29 Aug 2007 21:13:08 -0500 Content-Type: text/plain Parts/Attachments: text/plain (146 lines) Reply

Subject: CWD NEW MEXICO RECORDS IT'S 19 CASE (near Texas border again) Date: August 29, 2007 at 6:39 pm PST


LAS CRUCES ? New Mexico recorded its 19th case of chronic wasting disease in deer in a sick animal found in the Bishop's Cap area of the Organ Mountains .

Officer Richard McDonald investigated a report of an emaciated deer July 12. The animal was unaware of human presence, chronically thirsty, urinating often, and staying in and near a water source. Officer McDonald followed the state's protocol for disease surveillance by killing the animal and sending it to the Veterinary Diagnostic Laboratory in Albuquerque for testing.

Based on the symptoms and the area from which the deer came, the laboratory was instructed that chronic wasting disease (CWD) was highly probable. Laboratory diagnostic testing confirmed presence of CWD in this deer. This is the 19th deer with confirmed CWD found since it was first detected in New Mexico in 2002. Two elk have also been found with CWD.

This deer was in Game Management Unit 19, where special CWD restrictions already exist for hunters.

Anyone who finds a deer or elk that appears unaware of human presence and displays symptoms including droopy ears, emaciation, chronic thirst, frequent urination, and reluctance to leave water, should report their observations to the Department of Game and Fish, Wildlife Management Division, (505) 476-8127.

----- Original Message -----

From: "Terry S. Singeltary Sr."

Sent: Saturday, December 23, 2006 1:47 PM

Subject: CWD in New Mexico 35 MILES FROM TEXAS BORDER and low testing sampling figures -- what gives TAHC ???

----- Original Message -----

From: "Terry S. Singeltary Sr." flounder9@VERIZON.NET


Sent: Saturday, December 23, 2006 1:47 PM

Subject: CWD in New Mexico 35 MILES FROM TEXAS BORDER and low testing sampling figures -- what gives TAHC ???

Subject: CWD in New Mexico 35 MILES FROM TEXAS BORDER and low testing sampling figures -- what gives TAHC ???

Date: December 23, 2006 at 11:25 am PST

Greetings BSE-L members,

i never know if i am going crazy or just more of the same BSe. several years ago i brought up the fact to the TAHC that CWD was literally at the Texas borders and that the sample size for cwd testing was no where near enough in the location of that zone bordering NM. well, i just wrote them another letter questioning this again on Dec. 14, 2006 (see below) and showed them two different pdf maps, one referencing this url, which both worked just fine then. since then, i have NOT received a letter from them answering my question, and the url for the map i used as reference is no longer working? i had reference this map several times from the hunter-kill cwd sampling as of 31 August 2005 pdf which NO longer works now??? but here are those figures for that zone bordering NM, for those that were questioning the url. the testing samples elsewhere across Texas where much much more than that figure in the zone bordering NM where CWD has been documented bordering TEXAS, near the White Sands Missile Range. SO, why was the Texas hunter-kill cwd sampling as of 31 August 2005 document removed from the internet??? you know, this reminds me of the infamous TEXAS MAD COW that i documented some 7 or 8 months before USDA et al documented it, when the TAHC accidentally started ramping up for the announcement on there web site, then removed it (see history at bottom). i am not screaming conspiracy here, but confusious is confused again on the ciphering there using for geographical distribution of cwd tissue sample size survey, IF they are serious about finding CWD in TEXAS. common sense would tell you if cwd is 35 miles from the border, you would not run across state and have your larger samples there, and least samples 35 miles from where is what found..........daaa..........TSS

THEN NOTICE CWD sample along that border in TEXAS, Three Year Summary of Hunter-Kill CWD sampling as of 31 August 2005 of only 191 samples, then compare to the other sample locations ;

TPWD has been conducting surveys of hunter-kill animals since 2002 and has collected more than 7300 samples (as of 31 August 2005). In total, there have been over 9400 samples, both hunter-kill and private samples, tested in Texas to date, and no positives have been found.

SO, out of a total of 9,400 samples taken for CWD surveillance in TEXAS since 2002 of both hunter-kill and private kill, ONLY 191 samples have been taken in the most likely place one would find CWD i.e. the border where CWD has been documented at TEXAS and New Mexico

latest map NM cwd old data

CWD in New Mexico ;

What is the Department doing to prevent the spread of CWD?

Chronic wasting disease (CWD) was recently

detected in a mule deer from

Unit 34. Until 2005, CWD had only been found

in Unit 19. With this discovery, the Department

will increase its surveillance of deer and elk

harvested in Units 29, 30 and 34.

Lymph nodes and/or brain stems from every

harvested deer and brain stems from all elk

taken in Unit 34 will be sampled.




A geographically-focused free-ranging cervid Monitoring Program was implemented during the fall 2002 deer-hunting season. Brain stem samples from hunter-killed deer will be obtained from TPWD Wildlife Management Areas (WMA), State Parks, and where otherwise available with hunter and/or landowner permission, from deer taken on private land. Volume 1, Sixth Edition of United States Department of Agriculture, Animal and Plant Health Inspection Service, Veterinary Services, Regulatory Statistics (Appendix D1) indicates that 148 samples is sufficient to detect disease at two per-cent prevalence, regardless of the population size. Therefore the goal is to acquire 148 samples from each of the State's ten ecoregions provided adequate sampling distribution is achieved across each ecoregion. The five year 2002 -2006, goal is to cumulatively collect 459 samples from each of the ten ecoregions. The cumulative sample would be used statistically to detect CWD at one per-cent prevalence level with 99 per-cent confidence. However, funding from APHIS/USDA could provide the necessary funds for sampling at the one per-cent prevalence level each year. TAHC conducted a risk assessment of counties where deer and elk have been imported and where high densities of free-ranging deer occur. The assessment was conducted for USDA funding consideration. The risk assessment was based on limited number of criteria. Since CWD could potentially occur anywhere in Texas, monitoring efforts would be focused to achieve a stratified sampling scheme across each ecoregion of the State.

Confidentiality laws restrict the type of data TPWD personnel can collect as it relates to a specific parcel of land. Therefore, personnel will ensure that no property specific information is collected (i.e. ranch name or exact location) without the landowner's written permission. The following are guidelines for data and sample collection distributed to TPWD personnel prior to sample collection:

A Texas Veterinary Medical Diagnostic Laboratory (TVMDL) Accession Form must be submitted with brain stem samples. The most important items to be filled out are the TPWD employee name, address and phone number, and "Patient/Deer ID". County of Kill can be recorded on the bottom of the form, but DO NOT report any information that identifies the specific parcel of land. The "Patient/Deer ID" number MUST BE specific to the field data sheet the employee is using to record data. Specific CWD field data sheets will not be provided, as current field data sheets (i.e. Age/Weight Antler Data Sheets, Hunter Check Station Data Sheets, etc.) will be appropriate in most cases. Field staff may produce their own CWD data sheet if necessary. The field data sheet must contain: Employee Name Sample Number (same as Patient/Deer ID on TVMDL Accession Form Sample Date Deer Age Deer Sex County of Kill Hunter Name Hunting License Number Ranch name or tract name/location ONLY with landowner permission. Should a CWD positive be detected, TAHC will use hunter contact information to conduct CWD investigation under their regulatory authority. Make sure the container containing the brain stem sample is legibly identified with the sample number, deer age and sex, county of kill and date. Although the sample number is all that is needed, additional information will help resolve any problems should batches of samples be combined. Should a landowner retain deer heads for our sampling purposes, remind the landowner to issue the hunters a proof of sex document as provided for in TAHC 65.10 (c). In addition, a Wildlife resource document (PWD 905) must accompany the head until the carcass reaches a final destination and finally processed. Samples MAY NOT be taken from legally harvested deer without the hunter's consent.

ACTIONS SHOULD A CWD POSITIVE BE DETECTED Should sampling detect a CWD positive animal, TAHC and TPWD would activate the Media Response Plan (Appendix F). TAHC and TPWD would immediately begin review of the information at hand and determine the action to be taken within the Response Plan (Appendix C.) The first action should be to inform landowners adjacent to the property containing the CWD positive and hold a meeting with advisory committees and affected landowner to discuss plans for secondary sampling. Planning for secondary sampling, investigating movements of deer into and away from property for further actions would then be the next step. The secondary sampling is critical for determining distribution and prevalence of the disease.

As distribution and prevalence is being determined, information review and discussions with TPWD advisory committees (e.g., Private Lands Advisory Board, Hunting Advisory Committee, White-tailed Deer Advisory Committee etc.) and landowners would take place in order to determine the appropriate management action to be taken.

and the discovery of several CWD positive mule deer in New Mexico, approximately 35 miles north of the Texas border were well out of the known boundaries of the disease.

The disease prevalence appears to be increasing in localized areas, although it is not clear whether this is due to increased incidence, or increased surveillance, reporting, and testing. Information from states with direct experience in managing CWD is being used for developing Texas plans as we learn from their experiences.

TPWD and TAHC are developing stepped up targeted and geographically-focused surveillance plans to monitor free-ranging deer for the presence of the disease and a rapid response plan to guide both TPWD and TAHC should CWD be detected in the State. TPWD and TAHC are also evaluating cervid management laws, rules, and policies for free ranging and scientific breeder permitted cervids under their authority to identify issues and potential weaknesses related to disease management. In these efforts, TPWD and TAHC will work with other agencies and organizations responsible for or are concerned about cervid disease management in an attempt to ensure comprehensive approaches to effective management of CWD risks (see Appendix C: Importation of Susceptible Cervids).

----- Original Message -----

From: Terry S. Singeltary Sr.


Sent: Thursday, December 14, 2006 9:52 PM

Subject: cwd at Texas border and low sampling figures ???

Greetings TAHC,

can someone please explain to me any reasoning at all for the very low sampling for CWD which have been taken where CWD is literally right at the steps of one of Texas borders, but yet across the state elsewhere, the numbers for testing increases ???

i do not understand the low sampling for cwd size where it is at our borders, compared to the highter numbers elsewhere???

see Texas hunter kill sample for CWD to Aug 31, 2005

see map where CWD has been documented at Texas border in free ranging deer and elk

kind regards,


Subject: CWD 3 NEW CASES SOUTHERN NEW MEXICO Date: July 10, 2006 at 8:51 am PST

New Mexico Department of Game and Fish Media contact: Dan Williams, (505) 476-8004 Public contact: (505) 476-8000



SANTA FE – Three deer in southern New Mexico have tested positive for chronic wasting disease, bringing the total number of confirmed CWD-infected deer in the state to 15 since the first infected deer was discovered in 2002.

The Department received test results Wednesday from the state Veterinary Diagnostic Services laboratory in Albuquerque that two wild deer captured near the White Sands Missile Range headquarters east of Las Cruces had tested positive for chronic wasting disease. A third wild deer captured in the small community of Timberon in the southern Sacramento Mountains also tested positive for the disease.

The discoveries of the infected deer were part of the Department's ongoing efforts to monitor the disease, which to date has been confined to the southern Sacramento Mountains southeast of Cloudcroft and areas surrounding the Organ Mountains near Las Cruces. Two wild elk from the southern Sacramento Mountains tested positive for the disease in December 2005.

Chronic wasting disease is a fatal neurological illness that afflicts deer, elk and moose. There is no evidence of CWD being transmitted to humans or livestock. The disease causes animals to become emaciated, display abnormal behavior and lose control of bodily functions. To date, it has been found in captive and wild deer, elk and moose in eight states and two Canadian provinces.

For more information about CWD in New Mexico and how hunters can assist in research and prevention, please visit the New Mexico Department of Game and Fish Web site, . More information about CWD also can be found on the Chronic Wasting Disease Alliance site at .




CWD Sampling Maps

Three Year Summary of Hunter-Kill CWD Sampling (as of August 31, 2005)

CWD Sampling Maps Three Year Summary of Hunter-Kill CWD Sampling (as of August 31, 2005) USDA CWD Maps March 2006 — Current Distribution of CWD TAHC CWD Monitoring Program Information CWD Sample Submission and Costs 2006 Factsheet For Producers Enrolling in the Complete Herd Monitoring Program USDA CWD Maps March 2006 — Current Distribution of CWD TAHC CWD Monitoring Program Information CWD Sample Submission and Costs 2006 Factsheet For Producers Enrolling in the Complete Herd Monitoring Program

----- Original Message -----

From: "Terry S. Singeltary Sr."

To: Sent: Monday, June 27, 2005 6:51 PM


##################### Bovine Spongiform Encephalopathy #####################

From: TSS


Date: June 27, 2005 at 4:43 pm PST

New Mexico Department of Game and Fish

Contact: Dan Williams, (505) 476-8004





SANTA FE – Two mule deer captured in the Organ Mountains as part of an ongoing research project near White

Sands Missile Range have tested positive for chronic wasting disease (CWD), a fatal neurological disease that

attacks the brains of infected deer and elk, the Department of Game and Fish announced.

The number of confirmed CWD cases in New Mexico now stands at 11 since 2002, when the disease was first

confirmed in a deer found near the eastern foothills of the Organ Mountains. All 11 CWD-infected deer were found

in the same general area of southern New Mexico. The origin of the disease in New Mexico remains unknown.

The carcasses of the infected deer will be incinerated, said Kerry Mower, the Department’s lead wildlife disease


Chronic wasting disease causes animals to become emaciated, display abnormal behavior, lose bodily functions

and die. The disease has been found in wild deer and elk, and in captive deer and elk, in eight states and two

Canadian provinces. There currently is no evidence of CWD being transmitted to humans or livestock.

Mower said the most recent CWD-positive deer showed no obvious physical signs of having the disease. They

were captured in April 2005 and tested as part of a 3-year-old research project studying deer population dynamics

in southern New Mexico. More than 140 deer have been captured alive and tested for the study, in which

researchers hope to find the cause of a 10-year decline in the area deer population. Study participants include the

Department of Game and Fish, the U.S. Army at White Sands Missile Range and Fort Bliss, Bureau of Land

Management, U.S. Geological Survey at New Mexico State University, and San Andres National Wildlife Refuge.

Hunters can assist the Department in its CWD research and prevention efforts by bringing their fresh, legally

harvested deer or elk head to an area office, where officers will remove the brain stem for testing. Participants will

be eligible for drawings for an oryx hunt on White Sands Missile Range and a trophy elk hunt on the Valle Vidal.

For more information about the drawing and chronic wasting disease, visit the Department web site at


Greetings list members,

I am deeply concerned with these CWD mad deer so close to the Texas border. WHAT keeps them from crossing the border to Texas ??? IF these illegal aliens can so easily cross our borders, why not these infected deer? maybe we should get these minute men to start watching for mad deer coming in to Texas from New Mexico.

I mentioned my concerns several other times before;

-------- Original Message --------

Subject: Current status of CWD testing in Texas

Date: Tue, 10 May 2005 09:09:47 –0500

From: "kschwaus"

To: Mr. Singeltary,

I was asked to provide you with the following information. If you have any other questions regarding CWD sampling in Texas, please do not hesitate to give me a call. My office number is below.

Below I have included a chart showing CWD samples that have been tested since the fall of 2002 through the present at the eco-region level. The second chart shows the totals on a given year. The unknown location samples come from private individuals sending in samples directly to the Texas Veterinary Medical Diagnostic Lab (TVMDL). Due to the confidentiality laws that the TVMDL operates under, they are unable to provide TPWD with the location of those samples.

snip... end...tss


Subject: CWD testing in Texas

Date: Sun, 25 Aug 2002 19:45:14 –0500

From: Kenneth Waldrup



Dear Dr. Singletary,

In Fiscal Year 2001, seven deer from Texas were tested by the National Veterinary Services Laboratory (NVSL) for CWD (5 fallow deer and 2 white-tailed deer).

In Fiscal Year 2002, seven elk from Texas were tested at NVSL (no deer).

During these two years, an additional six elk and one white-tailed deer were tested at the Texas Veterinary Medical Diagnostic Laboratory (TVMDL).

In Fiscal Year 2002, four white-tailed deer (free-ranging clinical suspects) and at least eight other white-tailed deer have been tested at TVMDL.

One elk has been tested at NVSL.

All of these animals have been found negative for CWD.

Dr. Jerry Cooke of the Texas Parks and Wildlife Department also has records of 601 clinically ill white-tailed deer which were necropsied at Texas A&M during the late 1960's and early 1970's, and no spongiform encepalopathies were noted.

Thank you for your consideration.

Ken Waldrup, DVM, PhD Texas Animal Health Commission



Captive Cervids

There have been no reported CWD infections of captive elk or deer in Texas. There is currently no mandatory surveillance program for susceptible cervids kept on game farms, although, there has been voluntary surveillance since 1999, which requires owners of participating herds to maintain an annual herd inventory and submit samples for all mortalities of animals over 16 months of age.

Free-Ranging (Wild) Cervids

There have been no reported CWD infections of free-ranging susceptible cervids in Texas. Currently targeted surveillance of free-ranging cervids having clinical symptoms is ongoing in Texas with no positives identified. Additionally, sampling of hunter-killed animals was initiated statewide during the 2002-2003 deer hunting season and sampling will be continued for the next three to five years.

Historic Status

Some have speculated that CWD is "spontaneous" and may exist naturally at low levels, even in Texas. The Texas Wildlife Disease Project, a cooperative research project between TPWD and Texas A&M University (circa 1965-1975), was created to address two disease issues; a) low reproduction in Texas pronghorn and b) "circling disease" in white-tailed deer. One of the leading veterinary pathologists on this project was already suspicious that the etiology of "circling disease" was scrapie being transmitted from sheep to deer. During the project's existence, a total of 780 clinically affected animals (601 white-tailed deer, 7 mule deer, 2 elk, and 170 exotic deer and antelope) were collected. Tissues, including brain and lymph nodes, from the collected animals were examined for spongiform histological lesions, and all were found to be negative. Had CWD (a form of TSE, like scrapie) existed in Texas during this time frame, it is probable that these investigations would have detected these classic histological lesions, especially in clinically affected animals. It must be noted, however, that the current laboratory tests used to diagnose CWD were not available during the time the Wildlife Disease Project so it can not be stated with absolute certainty that CWD was not present.


Diseases such as CWD tend to be managed more effectively when efforts are applied before or as the disease emerges, rather than after it becomes established. CWD is an emerging disease. The current number of known infections within private elk and deer breeding facilities varies markedly among states (and Canada) and is increasing steadily with continued and expanding surveillance and investigations. The geographic spread of CWD in free-ranging mule deer, white-tailed deer and elk is a concern. The recent discovery of CWD in free-ranging white-tailed deer in Wisconsin and Illinois, approximately 700 miles east of any previously known infection, and the discovery of several CWD positive mule deer in New Mexico, approximately 35 miles north of the Texas border were well out of the known boundaries of the disease.

The disease prevalence appears to be increasing in localized areas, although it is not clear whether this is due to increased incidence, or increased surveillance, reporting, and testing. Information from states with direct experience in managing CWD is being used for developing Texas plans as we learn from their experiences.

TPWD and TAHC are developing stepped up targeted and geographically-focused surveillance plans to monitor free-ranging deer for the presence of the disease and a rapid response plan to guide both TPWD and TAHC should CWD be detected in the State. TPWD and TAHC are also evaluating cervid management laws, rules, and policies for free ranging and scientific breeder permitted cervids under their authority to identify issues and potential weaknesses related to disease management. In these efforts, TPWD and TAHC will work with other agencies and organizations responsible for or are concerned about cervid disease management in an attempt to ensure comprehensive approaches to effective management of CWD risks (see Appendix C: Importation of Susceptible Cervids).

TAHC and TPWD have split jurisdictions and regulatory responsibilities, which creates challenges for both agencies (i.e., TAHC responsible for elk, TPWD responsible for white-tailed deer and mule deer). Both agencies will cooperate to resolve issues as they arise.


1. Education and information sharing with public, constituents, and other government agency personnel concerning CWD. 2. Ongoing targeted surveillance of clinical deer statewide (i.e., collecting and CWD- testing deer/elk exhibiting symptoms that may be consistent with CWD). 3. Development and implementation of a geographically-focused Monitoring Plan involving the sampling and CWD-testing of hunter-harvested deer. 4. TAHC Rules for Importation of Susceptible Cervids (Appendix C ). 5. Response Plan for CWD should it occur in Texas(Appendix D ). 6. TAHC rules for monitoring for CWD in breeding facilities (Appendix E ). 7. Media Response plan development in the possible event of a positive CWD occurrence (Appendix F ). 8. Advance education of relevant professionals such as resource agency personnel, private wildlife consultants, veterinarians, landowners, wildlife co-ops, taxidermists, and others

snip...see full text ;

Thursday, November 29, 2012


Saturday, February 04, 2012

Wisconsin 16 age limit on testing dead deer Game Farm CWD Testing Protocol Needs To Be Revised

Monday, June 11, 2012

OHIO Captive deer escapees and non-reporting


Thursday, December 06, 2012

Pennsylvania CWD Not Found in Pink 23 PA captive escapee, but where is Purple 4 and the other escapees ?

News for Immediate Release

Wednesday, December 05, 2012

Senator Casey Urges USDA To Take Smart Steps to Implement New Measure That Could Help Combat Chronic Wasting Disease Among Deer

From: Terry S. Singeltary Sr.

Sent: Wednesday, December 05, 2012 11:50 AM

To: Cc: ; Terry S. Singeltary Sr.

Subject: Casey Urges USDA To Take Smart Steps to Implement New Measure That Could Help Combat Chronic Wasting Disease Among Deer

Wednesday, November 14, 2012


Tuesday, November 13, 2012


Wednesday, November 07, 2012

PENNSYLVANIA Second Adams County Deer Tests Positive for Chronic Wasting Disease

Tuesday, November 06, 2012

PA Department of Agriculture investigating possible 2nd case of chronic wasting disease

Thursday, November 01, 2012


Friday, October 26, 2012


Tuesday, October 23, 2012

PA Captive deer from CWD-positive farm roaming free

Wednesday, October 17, 2012, 11:33 PM

Pennsylvania CWD number of deer exposed and farms there from much greater than first thought

Monday, October 15, 2012


Release #124-12

Thursday, October 11, 2012

Pennsylvania Confirms First Case CWD Adams County Captive Deer Tests Positive

Friday, October 26, 2012


Sunday, December 09, 2012

Pennsylvania Sportsmen upset with agriculture’s lack of transparency on CWD

Tuesday, December 11, 2012


Thursday, November 29, 2012

Chronic wasting disease on the Canadian prairies

Monday, December 03, 2012

WISCONSIN Deer from Racine County has tested positive for CWD

Friday, November 16, 2012

Yellowstone elk herds feeding grounds, or future killing grounds from CWD

Thursday, November 29, 2012

Wyoming Elk Hunt Area 10 Added to CWD List

Friday, July 20, 2012

CWD found for first time in Iowa at hunting preserve

Wednesday, September 05, 2012

Additional Facility in Pottawatamie County Iowa Under Quarantine for CWD after 5 deer test positive

Friday, September 21, 2012

Chronic Wasting Disease CWD raises concerns about deer farms in Iowa

Tuesday, November 27, 2012

IOWA DNR to increase number of deer tissue samples as part of Surveillance for Chronic Wasting Disease

Tuesday, November 13, 2012

North Carolina commission sets up task force on deer farming

Tuesday, November 13, 2012


Thursday, November 01, 2012


Friday, December 07, 2012

ALABAMA BIG BUCK PROJECT Conservation Commissioner N. Gunter Guy Jr. signs emergency order Prohibiting practice effective December 7, 2012

Monday, October 08, 2012

VDGIF has discovered four positive cases of CWD in Virginia Updated 9/24/2012

Friday, September 28, 2012

Stray elk renews concerns about deer farm security Minnesota

Friday, October 21, 2011

Chronic Wasting Disease Found in Captive Deer Missouri

Thursday, July 19, 2012


Tuesday, April 24, 2012


Saturday, March 10, 2012

CWD, GAME FARMS, urine, feces, soil, lichens, and banned mad cow protein feed CUSTOM MADE for deer and elk

Monday, November 26, 2012

Aerosol Transmission of Chronic Wasting Disease in White-tailed Deer

Monday, November 26, 2012

Rapid Transepithelial Transport of Prions following Inhalation

Friday, November 09, 2012

*** Chronic Wasting Disease CWD in cervidae and transmission to other species

Saturday, October 6, 2012


Friday, August 24, 2012

Diagnostic accuracy of rectal mucosa biopsy testing for chronic wasting disease within white-tailed deer (Odocoileus virginianus) herds in North America

Friday, August 31, 2012


Tuesday, June 05, 2012

Captive Deer Breeding Legislation Overwhelmingly Defeated During 2012 Legislative Session

Thursday, May 31, 2012

CHRONIC WASTING DISEASE CWD PRION2012 Aerosol, Inhalation transmission, Scrapie, cats, species barrier, burial, and more

Subject: DOCKET-- 03D-0186 -- FDA Issues Draft Guidance on Use of Material From Deer and Elk in Animal Feed; Availability

Date: Fri, 16 May 2003 11:47:37 –0500

From: "Terry S. Singeltary Sr."



Volume 3, Number 8 01 August 2003


Tracking spongiform encephalopathies in North America

Xavier Bosch

My name is Terry S Singeltary Sr, and I live in Bacliff, Texas. I lost my mom to hvCJD (Heidenhain variant CJD) and have been searching for answers ever since. What I have found is that we have not been told the truth. CWD in deer and elk is a small portion of a much bigger problem.

49-year-old Singeltary is one of a number of people who have remained largely unsatisfied after being told that a close relative died from a rapidly progressive dementia compatible with spontaneous Creutzfeldt-Jakob disease (CJD). So he decided to gather hundreds of documents on transmissible spongiform encephalopathies (TSE) and realised that if Britons could get variant CJD from bovine spongiform encephalopathy (BSE), Americans might get a similar disorder from chronic wasting disease (CWD)the relative of mad cow disease seen among deer and elk in the USA. Although his feverish search did not lead him to the smoking gun linking CWD to a similar disease in North American people, it did uncover a largely disappointing situation.

Singeltary was greatly demoralised at the few attempts to monitor the occurrence of CJD and CWD in the USA. Only a few states have made CJD reportable. Human and animal TSEs should be reportable nationwide and internationally, he complained in a letter to the Journal of the American Medical Association (JAMA 2003; 285: 733). I hope that the CDC does not continue to expect us to still believe that the 85% plus of all CJD cases which are sporadic are all spontaneous, without route or source.

Until recently, CWD was thought to be confined to the wild in a small region in Colorado. But since early 2002, it has been reported in other areas, including Wisconsin, South Dakota, and the Canadian province of Saskatchewan. Indeed, the occurrence of CWD in states that were not endemic previously increased concern about a widespread outbreak and possible transmission to people and cattle.

To date, experimental studies have proven that the CWD agent can be transmitted to cattle by intracerebral inoculation and that it can cross the mucous membranes of the digestive tract to initiate infection in lymphoid tissue before invasion of the central nervous system. Yet the plausibility of CWD spreading to people has remained elusive.

Getting data on TSEs in the USA from the government is like pulling teeth, Singeltary argues. You get it when they want you to have it, and only what they want you to have.


Sunday, December 2, 2012

CANADA 19 cases of mad cow disease SCENARIO 4: ‘WE HAD OUR CHANCE AND WE BLEW IT’

Friday, November 23, 2012

sporadic Creutzfeldt-Jakob Disease update As at 5th November 2012 UK, USA, AND CANADA

Tuesday, November 6, 2012

Transmission of New Bovine Prion to Mice, Atypical Scrapie, BSE, and Sporadic CJD, November-December 2012 update

Saturday, October 6, 2012


Tuesday, July 17, 2012

O.I.E. BSE, CWD, SCRAPIE, TSE PRION DISEASE Final Report of the 80th General Session, 20 - 25 May 2012



Terry S. Singeltary Sr. P.O. Box 42 Bacliff, Texas USA 77518


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